scholarly journals Increase in Muscle Activation during Physiotherapy with Electromyography Biofeedback for Patients with Acute Cervical Spinal Cord Injuries*

2019 ◽  
Author(s):  
Guijin Li ◽  
Caitlin Doman ◽  
Nina L. Suresh
Keyword(s):  
Spinal Cord ◽  
Muscle Activation ◽  
Spinal Cord Injuries ◽  
Cervical Spinal Cord

Binding the Abdomen Can Improve Speech in Men With Phrenic Nerve Pacers

2002 ◽  
Vol 11 (1) ◽  
pp. 71-76 ◽  
Author(s):  
Jeannette D. Hoit ◽  
Robert B. Banzett ◽  
Robert Brown
Keyword(s):  
Spinal Cord ◽  
Phrenic Nerve ◽  
Muscle Activation ◽  
Spinal Cord Injuries ◽  
Cervical Spinal Cord ◽  
Neck Muscle ◽  
Behavioral Strategies ◽  
Tracheal Pressure ◽  
The Subject ◽  
High Cervical

Two men with high cervical spinal cord injuries and phrenic nerve pacers were studied with and without an elastic binder around the abdomen. Speech improved with the binder, as determined by listener preference ratings provided by 10 judges and by the subjects themselves. Improvement was substantial in one subject and slight in the other. The subject with the greater improvement exhibited higher peak tracheal pressure, higher sound pressure level, and longer utterance duration with the binder in place. Speech improvement was attributed primarily to augmentation of tidal volume associated with the use of the binder. An abdominal binder can be an effective intervention to improve speech in certain individuals with spinal cord injuries and phrenic nerve pacers. Speech may be further improved by using behavioral strategies, such as neck muscle activation, glossopharyngeal breathing, and pharyngeal or buccal speech production.

Metrizamide Myelography for Cervical Spinal Cord Injuries

Radiology ◽  
1978 ◽  
Vol 129 (3) ◽  
pp. 707-711 ◽  
Author(s):  
Jin S. Leo ◽  
R. Thomas Bergeron ◽  
Irvin I. Kricheff ◽  
M. Vallo Benjamin
Keyword(s):  
Spinal Cord ◽  
Spinal Cord Injuries ◽  
Cervical Spinal Cord

Pneumonia as a modifiable risk factor of mechanical ventilation in cervical spinal cord injuries

2018 ◽  
Vol 2 (4) ◽  
pp. 213-217
Author(s):  
Han Ting Wang ◽  
David Williamson ◽  
Mary Francispillai ◽  
Andréanne Richard-Denis ◽  
Martin Albert
Keyword(s):  
Spinal Cord ◽  
Mechanical Ventilation ◽  
Risk Factor ◽  
Spinal Cord Injuries ◽  
Cervical Spinal Cord ◽  
Modifiable Risk Factor

Smart-device environmental control systems: experiences of people with cervical spinal cord injuries

2017 ◽  
Vol 13 (8) ◽  
pp. 724-730
Author(s):  
Bethany Hooper ◽  
Michele Verdonck ◽  
Delena Amsters ◽  
Michelle Myburg ◽  
Emily Allan
Keyword(s):  
Spinal Cord ◽  
Control Systems ◽  
Spinal Cord Injuries ◽  
Environmental Control ◽  
Cervical Spinal Cord ◽  
Smart Device

scholarly journals Disorder of Serum Electrolyte Concentrations in acute Cervical Spinal Cord Injuries

1985 ◽  
Vol 34 (1) ◽  
pp. 284-288
Author(s):  
Motofumi Komori ◽  
Kunio Sasaki ◽  
Keiichiro Shiba ◽  
Koichiro Yamano ◽  
Takayoshi Ueta ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Spinal Cord Injuries ◽  
Cervical Spinal Cord ◽  
Serum Electrolyte

Effect of Heparin Following Cervical Spinal Cord Injuries in Rats

Neurosurgery ◽  
2011 ◽  
Vol 69 (4) ◽  
pp. 930-941 ◽  
Author(s):  
Yiyan Zheng ◽  
Yi Ping Zhang ◽  
Lisa B E Shields ◽  
Yongjie Zhang ◽  
Marcus W Siu ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Spinal Cord Injuries ◽  
Cervical Spinal Cord

scholarly journals High-frequency epidural stimulation across the respiratory cycle evokes phrenic short-term potentiation after incomplete cervical spinal cord injury

2017 ◽  
Vol 118 (4) ◽  
pp. 2344-2357 ◽  
Author(s):  
Elisa J. Gonzalez-Rothi ◽  
Kristi A. Streeter ◽  
Marie H. Hanna ◽  
Anna C. Stamas ◽  
Paul J. Reier ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Spinal Cord Injury ◽  
High Frequency ◽  
Muscle Activation ◽  
Cervical Spinal Cord ◽  
Cervical Spinal Cord Injury ◽  
Burst Frequency ◽  
Epidural Stimulation ◽  
Term Potentiation ◽  
Cord Injury

C2 spinal hemilesion (C2Hx) paralyzes the ipsilateral diaphragm, but recovery is possible through activation of “crossed spinal” synaptic inputs to ipsilateral phrenic motoneurons. We tested the hypothesis that high-frequency epidural stimulation (HF-ES) would potentiate ipsilateral phrenic output after subacute and chronic C2Hx. HF-ES (300 Hz) was applied to the ventrolateral C4 or T2 spinal cord ipsilateral to C2Hx in anesthetized and mechanically ventilated adult rats. Stimulus duration was 60 s, and currents ranged from 100 to 1,000 µA. Bilateral phrenic nerve activity and ipsilateral hypoglossal (XII) nerve activity were recorded before and after HF-ES. Higher T2 stimulus currents potentiated ipsilateral phasic inspiratory activity at both 2 and 12 wk post-C2Hx, whereas higher stimulus currents delivered at C4 potentiated ipsilateral phasic phrenic activity only at 12 wk ( P = 0.028). Meanwhile, tonic output in the ipsilateral phrenic nerve reached 500% of baseline values at the high currents with no difference between 2 and 12 wk. HF-ES did not trigger inspiratory burst-frequency changes. Similar responses occurred following T2 HF-ES. Increases in contralateral phrenic and XII nerve output were induced by C4 and T2 HF-ES at higher currents, but the relative magnitude of these changes was small compared with the ipsilateral phrenic response. We conclude that following incomplete cervical spinal cord injury, HF-ES of the ventrolateral midcervical or thoracic spinal cord can potentiate efferent phrenic motor output with little impact on inspiratory burst frequency. However, the substantial increases in tonic output indicate that the uninterrupted 60-s stimulation paradigm used is unlikely to be useful for respiratory muscle activation after spinal injury. NEW & NOTEWORTHY Previous studies reported that high-frequency epidural stimulation (HF-ES) activates the diaphragm following acute spinal transection. This study examined HF-ES and phrenic motor output following subacute and chronic incomplete cervical spinal cord injury. Short-term potentiation of phrenic bursting following HF-ES illustrates the potential for spinal stimulation to induce respiratory neuroplasticity. Increased tonic phrenic output indicates that alternatives to the continuous stimulation paradigm used in this study will be required for respiratory muscle activation after spinal cord injury.

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