Research on stimulus pattern for inhibition of detrusor hyperreflexia

2010 ◽  
Author(s):  
Qian Dong ◽  
Xu Zhang ◽  
Chen Sun ◽  
GuoQing Chen ◽  
Nan Cui
Keyword(s):  
Stimulus Pattern ◽  
Detrusor Hyperreflexia

Intravesical Capsaicin in Neurologic Impaired Patients With Detrusor Hyperreflexia

1996 ◽  
Vol 19 (3) ◽  
pp. 190-193 ◽  
Author(s):  
Akhil Das ◽  
Michael B. Chancellor ◽  
Toyohiko Watanabe ◽  
John Sedor ◽  
David A. Rivas
Keyword(s):  
Detrusor Hyperreflexia

Anal Sphincter Maximum Functional Electrical Stimulation in Detrusor Hyperreflexia

1994 ◽  
Vol 152 (5 Part 1) ◽  
pp. 1460-1462 ◽  
Author(s):  
T. Petersen ◽  
J.E. Just-Christensen ◽  
P. Kousgaard ◽  
B. Holmboe ◽  
B. Klemar
Keyword(s):  
Electrical Stimulation ◽  
Anal Sphincter ◽  
Functional Electrical Stimulation ◽  
Detrusor Hyperreflexia

Caudolateral areas of medulla-mediating release of ACTH in cats

1986 ◽  
Vol 251 (5) ◽  
pp. R934-R940
Author(s):  
D. A. Bereiter ◽  
D. S. Gann
Keyword(s):  
Brain Stem ◽  
Stimulus Pattern ◽  
Reticular Nucleus ◽  
Lateral Reticular Nucleus ◽  
Plasma Acth ◽  
Acth Stimulation ◽  
Afferent Information ◽  
Burst Pattern ◽  
Stimulation Of ◽  
Acth Release

The effect of electrical stimulation of the caudolateral brain stem on plasma adrenocorticotropin (ACTH) was assessed in cats anesthetized with alpha-chloralose-urethan. To examine the influence of stimulus pattern on ACTH release, an equal number of pulses was presented in a continuous pattern and in a burst pattern at each electrode site. Stimulation of the magnocellular portion (layers 4-6) of trigeminal nucleus caudalis evoked a significant (P less than 0.01) and equal peak change in plasma ACTH after continuous pattern (+121 +/- 32 pg/ml) and after burst pattern stimuli (+126 +/- 30 pg/ml, n = 21). In contrast, stimulation of more ventromedial portions (layers 7-8) of nucleus caudalis had no significant effect on plasma ACTH. Stimulation of the trigeminal lateral cervical region the caudal extent of the A1 noradrenergic cell group, or the lateral reticular nucleus evoked significant peak increases in plasma ACTH regardless of stimulus pattern. Transient changes in arterial pressure accompanied brain stem stimulation and were not correlated with the changes in ACTH. The results indicate that stimulation of trigeminal subnucleus caudalis, a brain stem region that processes nociceptor afferent information, evokes a prompt increase in plasma ACTH. Stimulation of brain stem regions that process autonomic and cardiovascular afferent information (A1 region, lateral reticular nucleus) also facilitate ACTH release. No significant influence of stimulus pattern on brain stem-evoked ACTH release was seen. The results support the hypothesis that the influence of the central nervous system on ACTH release may be processed by parallel pathways at the caudal brain stem level.

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