scholarly journals Apoptosis: A Functional Paradigm for Programmed Plant Cell Death Induced by a Host-Selective Phytotoxin and Invoked during Development.

1996 ◽  
pp. 375-391 ◽  
Author(s):  
H. Wang ◽  
J. Li ◽  
R. M. Bostock ◽  
D. G. Gilchrist
Keyword(s):  
Cell Death ◽  
Plant Cell

Programmed Plant Cell Death: A Review

2011 ◽  
Vol 23 (1) ◽  
Author(s):  
Mohamed Al-Whaibi
Keyword(s):  
Cell Death ◽  
Plant Cell

Elevated Levels of Phosphorylated Sphingobases Do Not Antagonize Sphingobase- or Fumonisin B1-Induced Plant Cell Death

2019 ◽  
Vol 60 (5) ◽  
pp. 1109-1119 ◽  
Author(s):  
Ren� Glenz ◽  
Dorette Schmalhaus ◽  
Markus Krischke ◽  
Martin J Mueller ◽  
Frank Waller
Keyword(s):  
Cell Death ◽  
Plant Cell ◽  
Fumonisin B1

scholarly journals Hydrogen peroxide as a signal controlling plant programmed cell death

2005 ◽  
Vol 168 (1) ◽  
pp. 17-20 ◽  
Author(s):  
Tsanko S. Gechev ◽  
Jacques Hille
Keyword(s):  
Hydrogen Peroxide ◽  
Cell Death ◽  
Programmed Cell Death ◽  
Signaling Pathways ◽  
Microarray Analysis ◽  
Plant Cell ◽  
Full Genome ◽  
Genome Coverage ◽  
Regulatory Mutants

Hydrogen peroxide (H2O2) has established itself as a key player in stress and programmed cell death responses, but little is known about the signaling pathways leading from H2O2 to programmed cell death in plants. Recently, identification of key regulatory mutants and near-full genome coverage microarray analysis of H2O2-induced cell death have begun to unravel the complexity of the H2O2 network. This review also describes a novel link between H2O2 and sphingolipids, two signals that can interplay and regulate plant cell death.

Why are the anionic porphyrins so efficient to induce plant cell death? A structure-activity relationship study to solve the puzzle

2019 ◽  
Vol 368 ◽  
pp. 276-289 ◽  
Author(s):  
Stéphanie Leroy-Lhez ◽  
Olivier Rezazgui ◽  
Mohammad Issawi ◽  
Mourad Elhabiri ◽  
Claude Alain Calliste ◽  
...  
Keyword(s):  
Cell Death ◽  
Plant Cell ◽  
Structure Activity Relationship ◽  
Activity Relationship ◽  
Structure Activity ◽  
Relationship Study

scholarly journals Identification and characterization of suppressors of plant cell death (SPD) effectors from Magnaporthe oryzae

2016 ◽  
Vol 18 (6) ◽  
pp. 850-863 ◽  
Author(s):  
William Sharpee ◽  
Yeonyee Oh ◽  
Mihwa Yi ◽  
William Franck ◽  
Alex Eyre ◽  
...  
Keyword(s):  
Cell Death ◽  
Plant Cell ◽  
Magnaporthe Oryzae ◽  
Identification And Characterization

scholarly journals Participation of the mitochondrial permeability transition pore in nitric oxide-induced plant cell death

FEBS Letters ◽  
2001 ◽  
Vol 510 (3) ◽  
pp. 136-140 ◽  
Author(s):  
Elzira E. Saviani ◽  
Cintia H. Orsi ◽  
Jusceley F.P. Oliveira ◽  
Cecı́lia A.F. Pinto-Maglio ◽  
Ione Salgado
Keyword(s):  
Nitric Oxide ◽  
Cell Death ◽  
Plant Cell ◽  
Mitochondrial Permeability Transition Pore ◽  
Mitochondrial Permeability Transition ◽  
Permeability Transition Pore ◽  
Permeability Transition ◽  
Mitochondrial Permeability

Overexpression of Xanthomonas campestris pv. vesicatoria effector AvrBsT in Arabidopsis triggers plant cell death, disease and defense responses

Planta ◽  
2012 ◽  
Vol 236 (4) ◽  
pp. 1191-1204 ◽  
Author(s):  
In Sun Hwang ◽  
Nak Hyun Kim ◽  
Du Seok Choi ◽  
Byung Kook Hwang
Keyword(s):  
Cell Death ◽  
Plant Cell ◽  
Xanthomonas Campestris ◽  
Defense Responses

scholarly journals Pectobacterium carotovorum Elicits Plant Cell Death with DspE/F but the P. carotovorum DspE Does Not Suppress Callose or Induce Expression of Plant Genes Early in Plant–Microbe Interactions

2011 ◽  
Vol 24 (7) ◽  
pp. 773-786 ◽  
Author(s):  
Hye-Sook Kim ◽  
Phanit Thammarat ◽  
Steven A. Lommel ◽  
Clifford S. Hogan ◽  
Amy O. Charkowski
Keyword(s):  
Gene Expression ◽  
Cell Death ◽  
Pseudomonas Syringae ◽  
Plant Cell ◽  
Expression Profiles ◽  
Gene Expression Profiles ◽  
Secretion System ◽  
Broad Host Range ◽  
Pectobacterium Carotovorum ◽  
Callose Deposition

The broad-host-range bacterial soft rot pathogen Pectobacterium carotovorum causes a DspE/F-dependent plant cell death on Nicotiana benthamiana within 24 h postinoculation (hpi) followed by leaf maceration within 48 hpi. P. carotovorum strains with mutations in type III secretion system (T3SS) regulatory and structural genes, including the dspE/F operon, did not cause hypersensitive response (HR)-like cell death and or leaf maceration. A strain with a mutation in the type II secretion system caused HR-like plant cell death but no maceration. P. carotovorum was unable to impede callose deposition in N. benthamiana leaves, suggesting that P. carotovorum does not suppress this basal immunity function. Within 24 hpi, there was callose deposition along leaf veins and examination showed that the pathogen cells were localized along the veins. To further examine HR-like plant cell death induced by P. carotovorum, gene expression profiles in N. benthamiana leaves inoculated with wild-type and mutant P. carotovorum and Pseudomonas syringae strains were compared. The N. benthamiana gene expression profile of leaves infiltrated with Pectobacterium carotovorum was similar to leaves infiltrated with a Pseudomonas syringae T3SS mutant. These data support a model where Pectobacterium carotovorum uses the T3SS to induce plant cell death in order to promote leaf maceration rather than to suppress plant immunity.

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