scholarly journals Parallel Proteomic and Phosphoproteomic Analyses of Successive Stages of Maize Leaf Development

2013 ◽  
Vol 25 (8) ◽  
pp. 2798-2812 ◽  
Author(s):  
Michelle R. Facette ◽  
Zhouxin Shen ◽  
Fjola R. Björnsdóttir ◽  
Steven P. Briggs ◽  
Laurie G. Smith
Keyword(s):  
Leaf Development ◽  
Maize Leaf

scholarly journals Specification of adaxial cell fate during maize leaf development

Development ◽  
2004 ◽  
Vol 131 (18) ◽  
pp. 4533-4544 ◽  
Author(s):  
M. T. Juarez
Keyword(s):  
Cell Fate ◽  
Leaf Development ◽  
Maize Leaf

scholarly journals Maize leaf development under climate change scenarios

2010 ◽  
Vol 45 (11) ◽  
pp. 1227-1236
Author(s):  
Nereu Augusto Streck ◽  
Josana Andréia Langner ◽  
Isabel Lago
Keyword(s):  
Climate Change ◽  
Elevated Temperature ◽  
Air Temperature ◽  
Leaf Development ◽  
Flag Leaf ◽  
Climate Change Scenarios ◽  
Crop Failure ◽  
Maize Leaf ◽  
Late Emergence ◽  
Leaf Appearance

The objective of this work was to simulate maize leaf development in climate change scenarios at Santa Maria, RS, Brazil, considering symmetric and asymmetric increases in air temperature. The model of Wang & Engel for leaf appearance rate (LAR), with genotype-specific coefficients for the maize variety BRS Missões, was used to simulate tip and expanded leaf accumulated number from emergence to flag leaf appearance and expansion, for nine emergence dates from August 15 to April 15. LAR model was run for each emergence date in 100-year climate scenarios: current climate, and +1, +2, +3, +4 and +5°C increase in mean air temperature, with symmetric and asymmetric increase in daily minimum and maximum air temperature. Maize crop failure due to frost decreased in elevated temperature scenarios, in the very early and very late emergence dates, indicating a lengthening in the maize growing season in warmer climates. The leaf development period in maize was shorter in elevated temperature scenarios, with greater shortening in asymmetric temperature increases, indicating that warmer nights accelerate vegetative development in maize.

Genome-wide transcript analysis of early maize leaf development reveals gene cohorts associated with the differentiation of C4Kranz anatomy

2013 ◽  
Vol 75 (4) ◽  
pp. 656-670 ◽  
Author(s):  
Peng Wang ◽  
Steven Kelly ◽  
Jim P. Fouracre ◽  
Jane A. Langdale
Keyword(s):  
Leaf Development ◽  
Transcript Analysis ◽  
Maize Leaf ◽  
Genome Wide

scholarly journals Active Mutator elements suppress the knotted phenotype and increase recombination at the Kn1-O tandem duplication.

Genetics ◽  
1992 ◽  
Vol 132 (3) ◽  
pp. 813-822 ◽  
Author(s):  
B Lowe ◽  
J Mathern ◽  
S Hake
Keyword(s):  
Transposable Elements ◽  
Leaf Development ◽  
High Frequency ◽  
Homologous Chromosome ◽  
Tandem Duplication ◽  
Fold Increase ◽  
Transcription Unit ◽  
Mutant Phenotype ◽  
Maize Leaf ◽  
Specific Allele

Abstract The KNOTTED-1 (KN1) locus is defined by a number of dominant mutations that affect leaf development. The Kn1-O mutation is characterized by outpocketings of tissue along lateral veins of the maize leaf and by displacement of ligule tissue from the junction of the blade and sheath into the blade. Kn1-O results from a tandem duplication of 17 kb; each repeat includes the entire 8-kb KN1 transcription unit. Mutator (Mu) transposable elements inserted at the junction of the two repeats diminish the mutant phenotype. The Mu insertions affect the Kn1-O mutation in several distinctive ways. (1) Two of the three Mu elements, a Mu1 and a Mu8 element, diminish the mutant phenotype only when active as indicated by hypomethylation; when methylated or inactive, the phenotype is comparable to the Kn1-O progenitor. (2) Additional rearrangements have arisen in these derivatives that further reduce the mutant phenotype. (3) A 100-2000-fold increase in the loss of one repeat occurs in the presence of Mu elements as compared to Kn1-O without elements. The high frequency of loss only occurs when the Mu elements are hypomethylated. The frequency is also influenced by the specific allele carried at the same locus on the homologous chromosome. Reciprocal exchange of flanking markers does not accompany the loss events. Various recombination models that address the events occurring at Kn1-O are presented.

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