scholarly journals Purine Nucleotide Metabolism of Germinating Soybean Embryonic Axes

1979 ◽  
Vol 63 (1) ◽  
pp. 100-104 ◽  
Author(s):  
James D. Anderson
Keyword(s):  
Nucleotide Metabolism ◽  
Purine Nucleotide ◽  
Embryonic Axes ◽  
Purine Nucleotide Metabolism

Some Aspects of Purine Nucleotide Metabolism in Lymphocytes of B-CLL

1991 ◽  
Vol 77 (2) ◽  
pp. 112-117 ◽  
Author(s):  
Antonella Tabucchi ◽  
Roberto Leoncini ◽  
Roberto Pagani ◽  
Maria Pizzichini ◽  
Lucia Terzuoli ◽  
...  
Keyword(s):  
Nucleotide Metabolism ◽  
Purine Nucleotide ◽  
Purine Nucleotide Metabolism

Effect of Testosterone on Purine Nucleotide Metabolism in Rat Liver

2004 ◽  
Vol 36 (9) ◽  
pp. 614-619 ◽  
Author(s):  
E. Marinello ◽  
R. Leoncini ◽  
L. Terzuoli ◽  
D. Vannoni ◽  
B. Porcelli ◽  
...  
Keyword(s):  
Rat Liver ◽  
Nucleotide Metabolism ◽  
Purine Nucleotide ◽  
Purine Nucleotide Metabolism

scholarly journals Multiple Sclerosis: Evaluation of Purine Nucleotide Metabolism in Central Nervous System in Association with Serum Levels of Selected Fat-Soluble Antioxidants

2014 ◽  
Vol 2014 ◽  
pp. 1-9 ◽  
Author(s):  
Ľubomír Kuračka ◽  
Terézia Kalnovičová ◽  
Jarmila Kucharská ◽  
Peter Turčáni
Keyword(s):  
Multiple Sclerosis ◽  
Negative Impact ◽  
Coenzyme Q ◽  
Serum Levels ◽  
Nucleotide Metabolism ◽  
Demyelinating Diseases ◽  
Control Group ◽  
Purine Nucleotide ◽  
Physiological Values ◽  
Purine Nucleotide Metabolism

In the pathogenesis of demyelinating diseases including multiple sclerosis (MS) an important role is played by oxidative stress. Increased energy requirements during remyelination of axons and mitochondria failure is one of the causes of axonal degeneration and disability in MS. In this context, we analyzed to what extent the increase in purine catabolism is associated with selected blood lipophilic antioxidants and if there is any association with alterations in serum levels of coenzyme Q10. Blood serum and cerebrospinal fluid (CSF) samples from 42 patients with diagnosed MS and 34 noninflammatory neurologic patients (control group) were analyzed. Compared to control group, MS patients had significantly elevated values of all purine nucleotide metabolites, except adenosine. Serum lipophilic antioxidantsγ-tocopherol,β-carotene, and coenzyme Q10for the vast majority of MS patients were deficient or moved within the border of lower physiological values. Serum levels of TBARS, marker of lipid peroxidation, were increased by 81% in the MS patients. The results indicate that the deficit of lipophilic antioxidants in blood of MS patients may have a negative impact on bioenergetics of reparative remyelinating processes and promote neurodegeneration.

Developmental changes in purine nucleotide metabolism in cultured rat astroglia

1995 ◽  
Vol 13 (8) ◽  
pp. 887-896 ◽  
Author(s):  
E. Zoref-Shani ◽  
Y. Bromberg ◽  
G. Lilling ◽  
I. Gozes ◽  
S. Brosh ◽  
...  
Keyword(s):  
Nucleotide Metabolism ◽  
Developmental Changes ◽  
Purine Nucleotide ◽  
Purine Nucleotide Metabolism
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