scholarly journals Studies on 3-Indoleacetic Acid Metabolism. VI. 3-Indoleacetic Acid Uptake and Metabolism by Pea Roots and Epicotyls

1960 ◽  
Vol 35 (2) ◽  
pp. 225-232 ◽  
Author(s):  
W. A. Andreae ◽  
M. W. Van Ysselstein
Keyword(s):  
Indoleacetic Acid ◽  
Acid Metabolism ◽  
Acid Uptake ◽  
Pea Roots ◽  
Uptake And Metabolism

scholarly journals Studies on 3-indoleacetic acid metabolism. VII. Metabolism of radioactive 3-indoleacetic acid by pea roots

1961 ◽  
Vol 36 (6) ◽  
pp. 783-787 ◽  
Author(s):  
W. A. Andreae ◽  
J. R. Robinson ◽  
M. W. H. Van Ysselstein
Keyword(s):  
Indoleacetic Acid ◽  
Acid Metabolism ◽  
Pea Roots

scholarly journals Rapid Effects of Indoleacetic Acid and Ethylene on the Growth of Intact Pea Roots

1975 ◽  
Vol 55 (3) ◽  
pp. 443-447 ◽  
Author(s):  
Wilfried E. Rauser ◽  
Roger F. Horton
Keyword(s):  
Indoleacetic Acid ◽  
Pea Roots

Fetal hepatic and umbilical uptakes of glucogenic substrates during a glucagon-somatostatin infusion

2002 ◽  
Vol 282 (3) ◽  
pp. E542-E550 ◽  
Author(s):  
Cecilia Teng ◽  
Frederick C. Battaglia ◽  
Giacomo Meschia ◽  
Michael R. Narkewicz ◽  
Randall B. Wilkening
Keyword(s):  
Amino Acid ◽  
Acid Metabolism ◽  
Fetal Liver ◽  
Amino Acid Uptake ◽  
Hepatic Uptake ◽  
High Rate ◽  
Acid Uptake ◽  
Glucose Carbon ◽  
Ovine Fetal ◽  
Equivalent Reduction

To test the hypothesis that fetal hepatic glutamate output diverts the products of hepatic amino acid metabolism from hepatic gluconeogenesis, ovine fetal hepatic and umbilical uptakes of glucose and glucogenic substrates were measured before and during fetal glucagon-somatostatin (GS) infusion and during the combined infusion of GS, alanine, glutamine, and arginine. Before the infusions, hepatic uptake of lactate, alanine, glutamine, arginine, and other substrates was accompanied by hepatic output of pyruvate, aspartate, serine, glutamate, and ornithine. The GS infusion induced hepatic output of 1.00 ± 0.07 mol glucose carbon/mol O2 uptake, an equivalent reduction in hepatic output of pyruvate and glutamate carbon, a decrease in umbilical glucose uptake and placental uptake of fetal glutamate, an increase in hepatic alanine and arginine clearances, and a decrease in umbilical alanine, glutamine, and arginine uptakes. The latter result suggests that glucagon inhibits umbilical amino acid uptake. We conclude that fetal hepatic pyruvate and glutamate output is part of an adaptation to placental function that requires the fetal liver to maintain both a high rate of catabolism of glucogenic substrates and a low rate of gluconeogenesis.

Essential fatty acid uptake and metabolism in the developing rodent brain

Lipids ◽  
1996 ◽  
Vol 31 (1) ◽  
pp. S103-S107 ◽  
Author(s):  
Robert J. Pawlosky ◽  
Glenn Ward ◽  
Norman Salem
Keyword(s):  
Fatty Acid ◽  
Essential Fatty Acid ◽  
Fatty Acid Uptake ◽  
Acid Uptake ◽  
Rodent Brain ◽  
Uptake And Metabolism

scholarly journals pH Affects 1H-indole-3-butyric Acid Uptake but not Metabolism during the Initiation Phase of Adventitious Root Induction in Apple Microcuttings

1998 ◽  
Vol 123 (1) ◽  
pp. 6-10 ◽  
Author(s):  
James F. Harbage ◽  
Dennis P. Stimart ◽  
Carol Auer
Keyword(s):  
Butyric Acid ◽  
High Performance ◽  
Root Formation ◽  
Root Induction ◽  
Acid Uptake ◽  
Root Initiation ◽  
Medium Ph ◽  
Initiation Phase ◽  
Maximum Root ◽  
Uptake And Metabolism

The influence of root initiation medium pH on root formation was investigated in relation to uptake and metabolism of applied IBA in microcuttings of Malus ×domestica Borkh. `Gala' and `Triple Red Delicious'. Root formation and uptake of H3-IBA were related inversely to root initiation medium pH. Maximum root count (10.3 roots) and IBA uptake were observed at pH 4.0. Regardless of pH, overall root count of `Gala' was higher (13.5 roots) than `Triple Red Delicious' (4 roots). Uptake of IBA was highest at pH 4.0 for `Gala' (1.7% uptake) and at pH 4 and 5 for `Triple Red Delicious' (0.75% uptake). Metabolism of IBA was the same regardless of root initiation medium pH or cultivar examined. One-half of the IBA taken up was converted to a compound that coeluted with IBAsp during high-performance liquid chromatography. Apparently, pH regulates root formation by affecting IBA uptake but not metabolism. The level of auxin in tissue appeared unrelated to root formation between genotypes. Chemical names used: 1H-indole-3-butyric acid (IBA); 5-H3-indole-3-butyric acid (H3-IBA); indole-3-butrylaspartic acid (IBAsp).

scholarly journals Correlation between increased atrial expression of genes related to fatty acid metabolism and autophagy in patients with chronic atrial fibrillation

2019 ◽  
Author(s):  
Yasushige Shingu ◽  
Shingo Takada ◽  
Takashi Yokota ◽  
Ryosuke Shirakawa ◽  
Akira Yamada ◽  
...  
Keyword(s):  
Gene Expression ◽  
Atrial Fibrillation ◽  
Fatty Acid ◽  
Fatty Acid Metabolism ◽  
Acid Metabolism ◽  
Right Atrial ◽  
Fatty Acid Transport ◽  
Acid Uptake ◽  
Fatty Acid Binding ◽  
Expression Of Genes

AbstractAtrial metabolic disturbance contributes to the onset and development of atrial fibrillation (AF). Autophagy plays a role in maintaining the cellular energy balance. We examined whether the altered atrial expression of genes related to fatty acid metabolism is linked to that related to autophagy in chronic AF. Right atrial tissue was obtained during heart surgery from 51 patients with sinus rhythm (SR, n=38) or chronic AF (n=13). Preoperative fasting serum free-fatty-acid levels were significantly higher in the AF patients. The atrial gene expression of fatty acid binding protein 3 (FABP3), which is involved in the cells’ fatty acid uptake and intracellular fatty acid transport, was significantly increased in AF patients compared to SR patients; in the SR patients it was positively correlated with the right atrial diameter and intra-atrial EMD, parameters of structural and electrical atrial remodeling that was evaluated by an echocardiography. In contrast, the two groups’ atrial contents of diacylglycerol (DAG), a toxic fatty acid metabolite, were comparable. Importantly, the atrial gene expression of microtubule-associated protein light chain 3 (LC3) was significantly increased in the AF patients, and autophagy-related genes including LC3 were positively correlated with the atrial expression of FABP3. In conclusion, in chronic AF patients, the atrial expression of FABP3 was upregulated in association with autophagy-related genes without altered atrial DAG content. Our findings may support the hypothesis that dysregulated cardiac fatty acid metabolism contributes to the progression of AF and induction of autophagy has a cardioprotective effect against cardiac lipotoxicity in chronic AF.

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