scholarly journals Growth Inhibitor Activity in Xanthium in Relation to Photoperiodism

1954 ◽  
Vol 29 (6) ◽  
pp. 533-536 ◽  
Author(s):  
A. K. Khudairi ◽  
Erik K. Bonde
Keyword(s):  
Growth Inhibitor ◽  
Inhibitor Activity

Growth-Inhibiting Properties of Xylem Exudate From Vitis vinifera

1995 ◽  
Vol 22 (1) ◽  
pp. 7 ◽  
Author(s):  
JA Campbell ◽  
BR Loveys ◽  
VWK Lee ◽  
S Strother
Keyword(s):  
Vitis Vinifera ◽  
Inhibitory Activity ◽  
Lemna Minor ◽  
Growth Inhibitor ◽  
Inhibitory Effect ◽  
Vitis Vinifera L ◽  
Inhibitory Effects ◽  
Inhibitor Activity ◽  
Xylem Exudate ◽  
Growth Inhibiting

An inhibitory effect on the growth of Lemna minor L. cultures has been demonstrated in xylem exudate from Vitis vinifera L. var. Waltham Cross bled from canes cut near the time of budburst. Most inhibitory activity was detected up to the time of maximal daily exudation, which corresponded closely with budburst. After this time the inhibitory activity rapidly disappeared. A similar pattern occurred in each of the 3 years of the study, 1988-1990. Using ultrafiltration, it was shown that most of the growth inhibitor activity of the crude exudate was located in the 0.5-10 kDa fraction. This fraction exhibited a seasonal variation in its bioactivity similar to that ofthe crude exudate samples. The 0.5-10 kDa fraction was found to contain abscisic acid but not in a sufficient quantity to account for the inhibitory effects. When chromatographically separated fractions corresponding to oligosaccharides were pooled, biological activity equivalent to that of the crude exudate was retained, which provides evidence that the inhibitor is possibly an oligosaccharide.

Insect growth inhibitor activity of arjunolic acid isolated from Cornus capitata

2002 ◽  
Vol 16 (S1) ◽  
pp. 68-70 ◽  
Author(s):  
R. S. Bhakuni ◽  
Y. N. Shukla ◽  
A. K. Tripathi ◽  
Veena Prajapati ◽  
Sushil Kumar
Keyword(s):  
Growth Inhibitor ◽  
Inhibitor Activity ◽  
Insect Growth ◽  
Arjunolic Acid

scholarly journals Seleksi Bakteri Endofit Penghasil Senyawa Metabolit untuk Pengendalian Cendawan Patogen Terbawa Benih Jagung

2017 ◽  
Vol 12 (5) ◽  
pp. 149
Author(s):  
Andini Hanif ◽  
Bonny Poernomo Wahyu Soekarno ◽  
Abdul Munif
Keyword(s):  
Endophytic Bacteria ◽  
High Growth ◽  
Growth Inhibitor ◽  
Antifungal Compound ◽  
Pseudomonas Sp ◽  
Sterile Soil ◽  
Inhibitor Activity ◽  
Tobacco Leaves ◽  
Seedborne Fungi ◽  
Fusarium Sp

Endophytic bacteria have been reported to produce metabolite as antifungal compound. This study was aimed to obtain endophytic bacteria which are able to produce metabolite to control Fusarium sp., a potential seedborne fungi on maize. Endophytic bacteria were screened by hypersensitive test on tobacco leaves and antagonistic test. Endophytic bacteria isolates with high growth inhibitor activity were selected and examined for their metabolite compound. Thre isolates, i.e. Lactobacillus sp. isolate EF14III, Pseudomonas sp. isolate ER1I, dan Aeromonas sp. isolate ER10I has the potential to inhibit Fusarium sp..  Metabolite compound of Pseudomonas sp. isolates ER1I was able to decrease the infection Fusarium sp. by 65.0% in blotter test and decreased infection of Fusarium sp. up to 59.5% and 60.5% in growing on test using water agar and sterile soil, respectively.  Cyclohexanone with concentration of 9.68% produced by Pseudomonas sp. isolat ERI1 may play a role as antifungal compound.

AChlamydomonasAlgal Bioassay for Detecting Growth Inhibitor Herbicides

Weed Science ◽  
1980 ◽  
Vol 28 (5) ◽  
pp. 515-520 ◽  
Author(s):  
F. D. Hess
Keyword(s):  
Aqueous Medium ◽  
Cell Population ◽  
Green Alga ◽  
Growth Inhibitor ◽  
Inhibitor Activity ◽  
Technical Grade ◽  
Chlamydomonas Eugametos ◽  
Synchronous Cell

The single-celled green algaChlamydomonas eugametoswas found to be well suited for detecting growth inhibitor activity of chemicals. Tests consisted of solubilizing technical grade compounds in 20-ml aliquots of a 1 × 105cells/ml zoospore culture. Commercially available growth inhibitor herbicides significantly inhibited synchronous cell population increases within 48 h at concentrations ranging from 1 × 10−4to 1 × 10−7M. Examples of compounds that inhibitedChlamydomonasmore than 50% were butylate (S-ethyl diisobutylthiocarbamate) and diallate [S-(2,3-dichloroallyl)diisopropylthiocarbamate] at 1 × 10−4M, CDEC [2 chloroallyl diethyldithiocarbamate] and propham (isopropyl carbanilate) at 1 × 10−5M, alachlor [2-chloro-2′,6′-diethyl-N-(methoxymethyl)acetanilide] and trifluralin [α,α,α-trifluoro-2,6-dinitro-N,N-dipropyl-p-toluidine] at 1 × 10−6M, and bifenox [methyl 5-(2,4-dichlorophenoxy)-2-nitrobenzoate] at 1 × 10−7M. Ethanol, dimethylsulfoxide (DMSO), or acetone can be used to solubilize herbicides in the aqueous medium. DMSO and ethanol are not detrimental to the cells if the concentration is kept at 1% v/v or less.

Calmodulin-inhibitor activity of tajixanthone analogues from the fungus Emericella sp strain 25379

Planta Medica ◽  
2008 ◽  
Vol 74 (09) ◽  
Author(s):  
M Figueroa ◽  
M González ◽  
R Rodríguez-Sotres ◽  
R Mata
Keyword(s):  
Inhibitor Activity ◽  
Calmodulin Inhibitor

Immunological Relationship Between Plasminogen Activator Inhibitors from Different Sources

1987 ◽  
Vol 57 (01) ◽  
pp. 029-034 ◽  
Author(s):  
Göran Urdén ◽  
Joanna Chmielewska ◽  
Tomas Carlsson ◽  
Björn Wiman
Keyword(s):  
Plasminogen Activator ◽  
Polyclonal Antibodies ◽  
Active Form ◽  
Polyclonal Antiserum ◽  
The Other ◽  
Hep G2 ◽  
Inhibitor Activity ◽  
Tissue Plasminogen ◽  
Immunological Relationship ◽  
Different Sources

SummaryPolyclonal antibodies have been raised against the inhibitor moiety in the purified complex between tissue plasminogen activator and its fast inhibitor (PA-inhibitor) in human plasma/ serum. A radioimmunoassay for quantitation of PA-inhibitor antigen was developed. The polyclonal antiserum and a previously described monoclonal antibody against the PA-inhibitor (14) have been used to study the immunological relationship between PA-inhibitors from plasma, serum, platelets, placenta extract and conditioned media from Hep G2 and HT 1080 cells. It was demonstrated that the ratio between PA-inhibitor activity and antigen varied considerably between the different sources. In the plasma samples studied, similar activity and antigen concentrations were found, suggesting that the PA-inhibitor in these samples mainly was in an active form. On the other hand the other sources seemed to contain variable amounts of inactive PA-inhibitor forms. Immunoadsorption experiments revealed that the PA-inhibitor (activity and antigen) from all the sources were specifically bound to the insolubilized antibodies (polyclonal and monoclonal). In no case, however, could active PA-inhibitor be eluted from the immunoadsorption columns. Also the competitive radioimmunoassays suggested that the PA-inhibitors from the different sources studied, were closely immunologically related.

A Simple and Rapid Method to Determine GPIIb/IIIa-Receptor Inhibitor Activity in Whole Blood

1999 ◽  
Vol 19 (03) ◽  
pp. 134-138
Author(s):  
Gitta Kühnel ◽  
A. C. Matzdorff
Keyword(s):  
Flow Cytometry ◽  
Platelet Count ◽  
Blood Sample ◽  
Platelet Activation ◽  
Whole Blood ◽  
Receptor Occupancy ◽  
Aggregate Formation ◽  
Inhibitor Activity ◽  
Receptor Inhibitor

SummaryWe studied the effect of GPIIb/IIIa-inhibitors on platelet activation with flow cytometry in vitro. Citrated whole blood was incubated with increasing concentrations of three different GPIIb/IIIa-inhibitors (c7E3, DMP728, XJ757), then thrombin or ADP were added and after 1 min the sample was fixed. Samples without c7E3 but with 0.1 U/ml thrombin had a decrease in platelet count. Samples with increasing concentrations of c7E3 had a lesser or no decrease in platelet count. The two other inhibitors (DMP 725, XJ757) gave similar results. GPIIb/IIIa-inhibitors prevent aggregate formation and more single platelets remain in the blood sample. The agonist-induced decrease in platelet count correlates closely with the concentration of the GPIIb/IIIa inhibitor and receptor occupancy. This correlation may be used as a simple measure for inhibitor activity in whole blood.

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