scholarly journals πΞcorrelations: An interweaving of resonance interaction, channel coupling, and Coulomb effects

2010 ◽  
Vol 81 (3) ◽  
Author(s):  
B. O. Kerbikov ◽  
L. V. Malinina
2012 ◽  
Vol 33 (7) ◽  
pp. 535-542 ◽  
Author(s):  
Wenjun J. Sun ◽  
Mehri Kaviani Mogadam ◽  
Marianne Sommarin ◽  
Henrietta Nittby ◽  
Leif G. Salford ◽  
...  

1996 ◽  
Vol 74 (7-8) ◽  
pp. 505-508 ◽  
Author(s):  
R. M. Finch ◽  
Á. Kövér ◽  
M. Charlton ◽  
G. Laricchia

Differential cross sections for elastic scattering and ionization in positron–argon collisions as a function of energy (40–150 eV) are reported at 60°. Of particular interest is the energy range 55–60 eV, where earlier measurements by the Detroit group found a drop in the elastic-scattering cross section of a factor of 2. This structure has been tentatively attributed to a cross channel-coupling effect with an open inelastic-scattering channel, most likely ionization. Our results indicate that ionization remains an important channel over the same energy range and only begins to decrease at an energy above 60 eV.


2016 ◽  
Vol 113 ◽  
pp. 06007
Author(s):  
Shin Watanabe ◽  
Takuma Matsumoto ◽  
Kosho Minomo ◽  
Kazuyuki Ogata ◽  
Masanobu Yahiro

2006 ◽  
Vol 913 ◽  
Author(s):  
Pei W. Ding ◽  
Kristel Fobelets ◽  
Jesus E Velazquez-Perez

AbstractA novel field effect transistor (FET) that uses 3-dimensional (3-D) embedded gate fingers – the Screen-Grid Field Effect Transistor (SGFET) – is proposed. The gating action of the SGFET is based on the design of multiple gating cylinders into the channel region, perpendicular to the current flow. Such configuration allows a full 3-D gate control of the current which improves the device characteristics by increasing the gate to channel coupling. Initial investigations of the SGFET using 3-D TCAD TaurusTM simulation software are presented in this paper. The results indicate that the proposed SGFET offers the possibility of downscaling without degrading the output characteristics. A comparison between the SGFET and both bulk and SOI MOSFETs shows the superior characteristics of the SGFET for low power operation.


Author(s):  
Cheng Liu ◽  
Yanxian Lai ◽  
Jingxian Pei ◽  
Huiling Huang ◽  
Junfang Zhan ◽  
...  

Abstract Context Lower serum concentration of apolipoprotein A-I (ApoA-I) is causally associated with heart failure (HF) risk. ATP-sensitive potassium channels (KATP), as a gating channel coupling vascular reactivity and metabolism with ischemic protection, become a new potential target of management for HF. The KATP gene sequence is highly polymorphic and high degree of genetic heterogeneity. Objective To determine whether ATP-sensitive potassium channels (KATP) variants predict the risks of decreased ApoA-I concentration and its related HF. Design, Patients, Settings A total of 634 subjects, including 317 subjects with decreased ApoA-I concentration (< 120 mg/dL) and 317 counterpart subjects (≥ 120 mg/dL), were retrospectively selected. Methods 5 KATP variants were genotyped through MassARRAY platform. The exosome-derived microRNAs (exo-miRs) expression profiles were identified by next-generation sequencing, and the top 10 DE exo-miRs were verified using qPCR in a validation cohort of 240 subjects with decreased ApoA-I concentration. Results KATP rs141294036 was related to increased risk of lower ApoA-I levels (adjusted OR=1.95, P=0.002) and HF incidence (adjusted OR=2.38, P=0.009), especially HFpEF (adjusted OR=2.13, P=0.015). After median 48.6-months follow-up, participants carrying CC genotype of rs141294036 was associated with elevated HF re-hospitalization risk (adjusted HR=1.91, P=0.005). 36 exo-miRs were significantly differentially expressed between different genotypes of rs141294036 in subjects with lower ApoA-I levels, but only 5 exo-miRs (miR-31-5p, miR-126-5p, miR-106a-5p, miR-378i and miR-181c-5p) were further confirmed. Conclusions The KATP rs141294036 was associated with increased risks of lower ApoA-I levels, HF incidence (especially HFpEF) and HF re-hospitalization, involving in those 5 confirmed exo-miRs and its related metabolic pathways.


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