Transterm: a database of mRNAs and translational control elements

G. H. Jacobs

doi:10.1093/nar/30.1.310

Evidence for Translational Control Elements Within the 5′-Untranslated Region of GLUT1 Glucose Transporter mRNA

Journal of Neurochemistry ◽

10.1046/j.1471-4159.1996.67041335.x ◽

2002 ◽

Vol 67 (4) ◽

pp. 1335-1343 ◽

Cited By ~ 24

Author(s):

Ruben J. Boado ◽

Haruhisa Tsukamoto ◽

William M. Pardridge

Keyword(s):

Translational Control ◽

Untranslated Region ◽

Glucose Transporter ◽

Translational Control Elements ◽

Glucose Transporter Mrna ◽

Glut1 Glucose Transporter

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Modification of translational control elements as a new approach to design of attenuated picornavirus strains

Journal of Biotechnology ◽

10.1016/0168-1656(95)00088-7 ◽

1996 ◽

Vol 44 (1-3) ◽

pp. 119-128 ◽

Cited By ~ 21

Author(s):

Vadim I. Agol ◽

Evgeny V. Pilipenko ◽

Olga R. Slobodskaya

Keyword(s):

Translational Control ◽

New Approach ◽

Translational Control Elements

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Analyses of murine Postsynaptic Density-95 identify novel isoforms and potential translational control elements

Molecular Brain Research ◽

10.1016/j.molbrainres.2004.09.024 ◽

2005 ◽

Vol 133 (1) ◽

pp. 143-152 ◽

Cited By ~ 10

Author(s):

Matthew Bence ◽

Margaret I. Arbuckle ◽

Kirsten S. Dickson ◽

Seth G.N. Grant

Keyword(s):

Translational Control ◽

Postsynaptic Density ◽

Translational Control Elements ◽

Novel Isoforms

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5′ end-dependent translation initiation of hepatitis C viral RNA and the presence of putative positive and negative translational control elements within the 5′ untranslated region

Virology ◽

10.1016/0042-6822(92)90264-p ◽

1992 ◽

Vol 191 (2) ◽

pp. 889-899 ◽

Cited By ~ 48

Author(s):

Byoung J. Yoo ◽

Richard R. Spaete ◽

Adam P. Geballe ◽

Mark Selby ◽

Michael Houghton ◽

...

Keyword(s):

Hepatitis C ◽

Translation Initiation ◽

Translational Control ◽

Untranslated Region ◽

Viral Rna ◽

Translational Control Elements

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Translational Control Elements in the Major Human Transforming Growth Factor-β1 mRNA

Growth Factors ◽

10.3109/08977199809002120 ◽

1998 ◽

Vol 16 (2) ◽

pp. 89-100 ◽

Cited By ~ 17

Author(s):

Robin S.H. Allison ◽

Michelle L. Mumy ◽

Lalage M. Wakefield

Keyword(s):

Growth Factor ◽

Translational Control ◽

Transforming Growth Factor ◽

Transforming Growth Factor Β1 ◽

Translational Control Elements

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Mechanism of post-segregational killing by hok-homologue pnd of plasmid R483: Two translational control elements in the pnd mRNA

Journal of Molecular Biology ◽

10.1006/jmbi.1995.0296 ◽

1995 ◽

Vol 249 (2) ◽

pp. 270-282 ◽

Cited By ~ 10

Author(s):

Allan K. Nielsen ◽

Kenn Gerdes

Keyword(s):

Translational Control ◽

Translational Control Elements

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The role of DUBs in the post-translational control of cell migration

Essays in Biochemistry ◽

10.1042/ebc20190022 ◽

2019 ◽

Vol 63 (5) ◽

pp. 579-594 ◽

Cited By ~ 2

Author(s):

Guillem Lambies ◽

Antonio García de Herreros ◽

Víctor M. Díaz

Keyword(s):

Cell Migration ◽

Translational Control ◽

Epithelial To Mesenchymal Transition ◽

Extracellular Matrix Remodeling ◽

Matrix Remodeling ◽

Mesenchymal Transition ◽

Tgfβ Receptors ◽

Fundamental Process ◽

Multistep Process

Abstract Cell migration is a multifactorial/multistep process that requires the concerted action of growth and transcriptional factors, motor proteins, extracellular matrix remodeling and proteases. In this review, we focus on the role of transcription factors modulating Epithelial-to-Mesenchymal Transition (EMT-TFs), a fundamental process supporting both physiological and pathological cell migration. These EMT-TFs (Snail1/2, Twist1/2 and Zeb1/2) are labile proteins which should be stabilized to initiate EMT and provide full migratory and invasive properties. We present here a family of enzymes, the deubiquitinases (DUBs) which have a crucial role in counteracting polyubiquitination and proteasomal degradation of EMT-TFs after their induction by TGFβ, inflammatory cytokines and hypoxia. We also describe the DUBs promoting the stabilization of Smads, TGFβ receptors and other key proteins involved in transduction pathways controlling EMT.

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