scholarly journals Cytogenetic investigation of subjects professionally exposed to radiofrequency radiation

Mutagenesis ◽  
2006 ◽  
Vol 21 (2) ◽  
pp. 139-142 ◽  
Author(s):  
Annemarie Maes ◽  
Urbain Van Gorp ◽  
Luc Verschaeve
Keyword(s):  
Radiofrequency Radiation ◽  
Cytogenetic Investigation

Public Exposure from Indoor Radiofrequency Radiation in the City of Hebron, West Bank—Palestine

2015 ◽  
Vol 109 (2) ◽  
pp. 117-121 ◽  
Author(s):  
Adnan Lahham ◽  
Afefeh Sharabati ◽  
Hussien ALMasri
Keyword(s):  
West Bank ◽  
Radiofrequency Radiation ◽  
Public Exposure ◽  
The City

scholarly journals Effects of radiofrequency radiation on colorectal cancer cell proliferation and inflammation

2021 ◽  
Vol 0 (0) ◽  
Author(s):  
Elcin Ozgur ◽  
Handan Kayhan ◽  
Gorkem Kismali ◽  
Fatih Senturk ◽  
Merve Sensoz ◽  
...  
Keyword(s):  
Colorectal Cancer ◽  
Cancer Cell ◽  
Colorectal Cancer Cell ◽  
Radiofrequency Radiation ◽  
Protein Levels ◽  
Intermittent Exposure ◽  
Colorectal Cancer Cells ◽  
Colorectal Cancer Cell Lines ◽  
Hct 116 ◽  
Hct 116 Cells

Abstract Objectives The aim of this study is to investigate the effects of radiofrequency radiation (RFR) on apoptosis, proliferation, stress response, and inflammation markers in colorectal cancer cells. Methods We tested the effects of intermittent exposure to RFR at different frequencies on two different colorectal cancer cell lines; HCT-116 and DLD-1. Protein levels were subsequently analyzed by ELISA. Results RFR led to a decrease in P53, p-P53, p-P38, and p-IkB levels in HCT-116 cells, while leading to an increase in BAD, p-BAD, p-STAT3,NF-κB levels. Two thousand one hundred Megahertz of RFR altered the P53, BAD, and NF-ΚB expression in HCT-116 cells. P53, p-P53, BAD, p-BAD, NF-κB, p-NF-κB, p-P38, p-SAPK/JNK, p-STAT3, and p-IkB levels increased after exposure to RFR at 900 and 2,100 MHz in DLD-1 cells. Unlike HCT-116 cells, 1,800 MHz of RFR was reported to have no effect on DLD1 cells. Conclusions RFR increased apoptosis and inflammatory response in HCT116 cells, while lowering the active P38 and active P53 levels, which are indicators of poor prognosis in several cancers. Genetic differences, such as P53 mutation (DLD-1), are critical to the cell response to RFR, which explains the reason why scientific studies on the effects of RFR yield contradictory results.

Cytogenetic Investigation of the Rh(null) Phenotype

Vox Sanguinis ◽  
1974 ◽  
Vol 27 (2) ◽  
pp. 190-192
Author(s):  
W.L. Marsh ◽  
R.S.K. Chaganti ◽  
James German ◽  
S. Seidl ◽  
W. Spielmann
Keyword(s):  
Cytogenetic Investigation ◽  
Null Phenotype

Micronucleus frequency in erythrocytes of mice after long-term exposure to radiofrequency radiation

2007 ◽  
Vol 83 (4) ◽  
pp. 213-220 ◽  
Author(s):  
Jukka Juutilainen ◽  
Päivi Heikkinen ◽  
Harri Soikkeli ◽  
Jorma Mäki-Paakkanen
Keyword(s):  
Radiofrequency Radiation ◽  
Micronucleus Frequency

Cytogenetic Investigation of the RhnullPhenotype

Vox Sanguinis ◽  
1974 ◽  
Vol 27 (2) ◽  
pp. 190-192
Author(s):  
W. L. Marsh ◽  
R. S. K. Chaganti ◽  
James German ◽  
S. Seidl ◽  
W. Spielmann
Keyword(s):  
Cytogenetic Investigation
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