scholarly journals Do Hormones, Telomere Lengths, and Oxidative Stress form an Integrated Phenotype? A Case Study in Free-Living Tree Swallows

2016 ◽  
Vol 56 (2) ◽  
pp. 138-145 ◽  
Author(s):  
J. Q. Ouyang ◽  
Á. Z. Lendvai ◽  
I. T. Moore ◽  
F. Bonier ◽  
M. F. Haussmann
Ecotoxicology ◽  
2009 ◽  
Vol 18 (5) ◽  
pp. 499-503 ◽  
Author(s):  
Dana M. Hawley ◽  
Kelly K. Hallinger ◽  
Daniel A. Cristol

Ecology ◽  
2011 ◽  
Vol 92 (4) ◽  
pp. 952-966 ◽  
Author(s):  
Maria G. Palacios ◽  
David W. Winkler ◽  
Kirk C. Klasing ◽  
Dennis Hasselquist ◽  
Carol M. Vleck

Ecotoxicology ◽  
2009 ◽  
Vol 18 (5) ◽  
pp. 514-521 ◽  
Author(s):  
Melinda D. Franceschini ◽  
Oksana P. Lane ◽  
David C. Evers ◽  
J. Michael Reed ◽  
Bart Hoskins ◽  
...  

2006 ◽  
Vol 189 (5) ◽  
pp. 2101-2109 ◽  
Author(s):  
Bryan W. Davies ◽  
Graham C. Walker

ABSTRACT During the initial stages of symbiosis with the host plant Medicago sativa, Sinorhizobium meliloti must overcome an oxidative burst produced by the plant in order for proper symbiotic development to continue. While identifying mutants defective in symbiosis and oxidative stress defense, we isolated a mutant with a transposon insertion mutation of sitA, which encodes the periplasmic binding protein of the putative iron/manganese ABC transporter SitABCD. Disruption of sitA causes elevated sensitivity to the reactive oxygen species hydrogen peroxide and superoxide. Disruption of sitA leads to elevated catalase activity and a severe decrease in superoxide dismutase B (SodB) activity and protein level. The decrease in SodB level strongly correlates with the superoxide sensitivity of the sitA mutant. We demonstrate that all free-living phenotypes of the sitA mutant can be rescued by the addition of exogenous manganese but not iron, a result that strongly implies that SitABCD plays an important role in manganese uptake in S. meliloti.


Ecotoxicology ◽  
2010 ◽  
Vol 20 (1) ◽  
pp. 39-46 ◽  
Author(s):  
Kelly K. Hallinger ◽  
Kerri L. Cornell ◽  
Rebecka L. Brasso ◽  
Daniel A. Cristol

2009 ◽  
Vol 33 (4) ◽  
pp. 456-463 ◽  
Author(s):  
Maria G. Palacios ◽  
Joan E. Cunnick ◽  
David Vleck ◽  
Carol M. Vleck

Author(s):  
Hasan Haci Yeter ◽  
Berfu Korucu ◽  
Elif Burcu Bali ◽  
Ulver Derici

Abstract. Background: The pathophysiological basis of chronic kidney disease and its complications, including cardiovascular disease, are associated with chronic inflammation and oxidative stress. We investigated the effects of active vitamin D (calcitriol) and synthetic vitamin D analog (paricalcitol) on oxidative stress in hemodialysis patients. Methods: This cross-sectional study was composed of 83 patients with a minimum hemodialysis vintage of one year. Patients with a history of any infection, malignancy, and chronic inflammatory disease were excluded. Oxidative markers (total oxidant and antioxidant status) and inflammation markers (C-reactive protein and interleukin-6) were analyzed. Results: A total of 47% (39/83) patients were using active or analog vitamin D. Total antioxidant status was significantly higher in patients with using active or analog vitamin D than those who did not use (p = 0.006). Whereas, total oxidant status and oxidative stress index were significantly higher in patients with not using vitamin D when compared with the patients who were using vitamin D preparation (p = 0.005 and p = 0.004, respectively). On the other hand, total antioxidant status, total oxidant status, and oxidative stress index were similar between patients who used active vitamin D or vitamin D analog (p = 0.6; p = 0.4 and p = 0.7, respectively). Conclusion: The use of active or selective vitamin D analog in these patients decreases total oxidant status and increases total antioxidant status. Also, paricalcitol is as effective as calcitriol in decreasing total oxidant status and increasing total antioxidant status in patients with chronic kidney disease.


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