Vascular amyloidosis impairs the gliovascular unit in a mouse model of Alzheimer’s disease

Ian F. Kimbrough; Stefanie Robel; Erik D. Roberson; Harald Sontheimer

doi:10.1093/brain/awv327

Vascular amyloidosis impairs the gliovascular unit in a mouse model of Alzheimer’s disease

Brain ◽

10.1093/brain/awv327 ◽

2015 ◽

Vol 138 (12) ◽

pp. 3716-3733 ◽

Cited By ~ 61

Author(s):

Ian F. Kimbrough ◽

Stefanie Robel ◽

Erik D. Roberson ◽

Harald Sontheimer

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Model Of Alzheimer’S Disease ◽

Gliovascular Unit

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O3-06-01: Vascular amyloidosis impairs the gliovascular unit in a mouse model of Alzheimer's disease

Alzheimer s & Dementia ◽

10.1016/j.jalz.2015.07.267 ◽

2015 ◽

Vol 11 (7S_Part_5) ◽

pp. P230-P230 ◽

Cited By ~ 1

Author(s):

Ian Kimbrough ◽

Stefanie Robel ◽

Erik Roberson ◽

Harald Sontheimer

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Model Of Alzheimer’S Disease ◽

Gliovascular Unit

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The effects of a neuroligin-1-derived peptide on behaviour in the 5XFAD mouse model of Alzheimer's disease

10.26226/morressier.5785edc6d462b80296c991f6 ◽

2016 ◽

Author(s):

Katrin Sonn

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Model Of Alzheimer’S Disease ◽

5Xfad Mouse

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Effects of Interleukin-6 trans-signalling in a mouse model of Alzheimer’s disease and high fat diet-induced obesity

10.26226/morressier.5b681764b56e9b005965c045 ◽

2018 ◽

Author(s):

Anna Escrig Monfort

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Interleukin 6 ◽

High Fat Diet ◽

High Fat ◽

Diet Induced Obesity ◽

Model Of Alzheimer’S Disease

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Faculty Opinions recommendation of Preserved fronto-striatal plasticity and enhanced procedural learning in a transgenic mouse model of Alzheimer's disease overexpressing mutant hAPPswe.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1020559.235469 ◽

2004 ◽

Author(s):

Karl-Peter Giese

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Transgenic Mouse ◽

Transgenic Mouse Model ◽

Procedural Learning ◽

Model Of Alzheimer’S Disease

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Faculty Opinions recommendation of Rapid appearance and local toxicity of amyloid-beta plaques in a mouse model of Alzheimer's disease.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1099453.555719 ◽

2008 ◽

Author(s):

Massimo Pandolfo

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Amyloid Beta ◽

Model Of Alzheimer’S Disease ◽

Local Toxicity ◽

Rapid Appearance

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Faculty Opinions recommendation of Clusters of hyperactive neurons near amyloid plaques in a mouse model of Alzheimer's disease.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1123163.580293 ◽

2008 ◽

Author(s):

Geneviève Dupont

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Amyloid Plaques ◽

Model Of Alzheimer’S Disease

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Faculty Opinions recommendation of Inhibitors of class 1 histone deacetylases reverse contextual memory deficits in a mouse model of Alzheimer's disease.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.4693956.4576054 ◽

2010 ◽

Author(s):

Francine Benes ◽

Barbara Gisabella

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Histone Deacetylases ◽

Memory Deficits ◽

Contextual Memory ◽

Model Of Alzheimer’S Disease ◽

Class 1

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Faculty Opinions recommendation of Modulation of gamma-secretase reduces beta-amyloid deposition in a transgenic mouse model of Alzheimer's disease.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.5594956.5560054 ◽

2010 ◽

Author(s):

Riqiang Yan

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Transgenic Mouse ◽

Beta Amyloid ◽

Transgenic Mouse Model ◽

Amyloid Deposition ◽

Gamma Secretase ◽

Model Of Alzheimer’S Disease

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Faculty Opinions recommendation of Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer's disease.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.717977209.793470743 ◽

2013 ◽

Author(s):

Matt Kaeberlein

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Amyloid Beta ◽

Cognitive Deficits ◽

Model Of Alzheimer’S Disease

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Faculty Opinions recommendation of Inhibitor of the tyrosine phosphatase STEP reverses cognitive deficits in a mouse model of Alzheimer's disease.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.718520520.793504888 ◽

2015 ◽

Author(s):

Amy Barrios

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Mouse Model ◽

Cognitive Deficits ◽

Tyrosine Phosphatase ◽

Model Of Alzheimer’S Disease

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