scholarly journals Remifentanil and nitrous oxide reduce changes in cerebral blood flow velocity in the middle cerebral artery caused by pain

2003 ◽  
Vol 90 (3) ◽  
pp. 296-299 ◽  
Author(s):  
I.H. Lorenz ◽  
C. Kolbitsch ◽  
M. Hinteregger ◽  
P. Bauer ◽  
M. Spiegel ◽  
...  
Keyword(s):  
Blood Flow ◽  
Nitrous Oxide ◽  
Cerebral Blood Flow ◽  
Middle Cerebral Artery ◽  
Flow Velocity ◽  
Cerebral Artery ◽  
Blood Flow Velocity ◽  
Cerebral Blood Flow Velocity

Slow oscillations in middle cerebral artery cerebral blood flow velocity and aging

2007 ◽  
Vol 29 (3) ◽  
pp. 260-263 ◽  
Author(s):  
Philip M. Lewis ◽  
Piotr Smielewski ◽  
John D. Pickard ◽  
Marek Czosnyka
Keyword(s):  
Blood Flow ◽  
Cerebral Blood Flow ◽  
Middle Cerebral Artery ◽  
Flow Velocity ◽  
Cerebral Artery ◽  
Blood Flow Velocity ◽  
Cerebral Blood Flow Velocity ◽  
Slow Oscillations

scholarly journals Blood Flow Velocity Changes in the Middle Cerebral Artery Induced by Driving Fatigue

2011 ◽  
Vol 2-3 ◽  
pp. 219-222
Author(s):  
Hong Wang ◽  
Xiao Mei Chi ◽  
Ya Jing Yan ◽  
Ning Ning Zhang
Keyword(s):  
Blood Flow ◽  
Cerebral Blood Flow ◽  
Middle Cerebral Artery ◽  
Flow Velocity ◽  
Cerebral Artery ◽  
Blood Flow Velocity ◽  
Cerebral Blood Flow Velocity ◽  
Driving Fatigue ◽  
Velocity Changes ◽  
Fatigue Driving

The cerebral blood flow velocity (CBFV) of middle cerebral artery (MCA) was detected during the fatigue driving using Transcranial Doppler. The CBFV was also analyzed after the fatigue driving by different means of relaxation to alleviate brain fatigue. The results show that the CBFV in MCA is reduced by driving fatigue.

The effect of NeuroAiD™ (MLC601) on cerebral blood flow velocity in subjects' post brain infarct in the middle cerebral artery territory

2011 ◽  
Vol 22 (5) ◽  
pp. 509-513 ◽  
Author(s):  
Reza Bavarsad Shahripour ◽  
Gholamreza Shamsaei ◽  
Hosein Pakdaman ◽  
Nastaran Majdinasab ◽  
Ehsan Mohammadiani Nejad ◽  
...  
Keyword(s):  
Blood Flow ◽  
Cerebral Blood Flow ◽  
Middle Cerebral Artery ◽  
Flow Velocity ◽  
Cerebral Artery ◽  
Blood Flow Velocity ◽  
Cerebral Blood Flow Velocity ◽  
Middle Cerebral Artery Territory ◽  
Brain Infarct

Direct Cerebral Vasodilatory Effects of Sevoflurane and Isoflurane 

1999 ◽  
Vol 91 (3) ◽  
pp. 677-677 ◽  
Author(s):  
Basil F. Matta ◽  
Karen J. Heath ◽  
Kate Tipping ◽  
Andrew C. Summors
Keyword(s):  
Blood Pressure ◽  
Blood Flow ◽  
Cerebral Blood Flow ◽  
Mean Arterial Pressure ◽  
Arterial Pressure ◽  
Middle Cerebral Artery ◽  
Flow Velocity ◽  
Cerebral Artery ◽  
Blood Flow Velocity ◽  
End Tidal

Background The effect of volatile anesthetics on cerebral blood flow depends on the balance between the indirect vasoconstrictive action secondary to flow-metabolism coupling and the agent's intrinsic vasodilatory action. This study compared the direct cerebral vasodilatory actions of 0.5 and 1.5 minimum alveolar concentration (MAC) sevoflurane and isoflurane during an propofol-induced isoelectric electroencephalogram. Methods Twenty patients aged 20-62 yr with American Society of Anesthesiologists physical status I or II requiring general anesthesia for routine spinal surgery were recruited. In addition to routine monitoring, a transcranial Doppler ultrasound was used to measure blood flow velocity in the middle cerebral artery, and an electroencephalograph to measure brain electrical activity. Anesthesia was induced with propofol 2.5 mg/kg, fentanyl 2 micro/g/kg, and atracurium 0.5 mg/kg, and a propofol infusion was used to achieve electroencephalographic isoelectricity. End-tidal carbon dioxide, blood pressure, and temperature were maintained constant throughout the study period. Cerebral blood flow velocity, mean blood pressure, and heart rate were recorded after 20 min of isoelectric encephalogram. Patients were then assigned to receive either age-adjusted 0.5 MAC (0.8-1%) or 1.5 MAC (2.4-3%) end-tidal sevoflurane; or age-adjusted 0.5 MAC (0.5-0.7%) or 1.5 MAC (1.5-2%) end-tidal isoflurane. After 15 min of unchanged end-tidal concentration, the variables were measured again. The concentration of the inhalational agent was increased or decreased as appropriate, and all measurements were repeated again. All measurements were performed before the start of surgery. An infusion of 0.01% phenylephrine was used as necessary to maintain mean arterial pressure at baseline levels. Results Although both agents increased blood flow velocity in the middle cerebral artery at 0.5 and 1.5 MAC, this increase was significantly less during sevoflurane anesthesia (4+/-3 and 17+/-3% at 0.5 and 1.5 MAC sevoflurane; 19+/-3 and 72+/-9% at 0.5 and 1.5 MAC isoflurane [mean +/- SD]; P<0.05). All patients required phenylephrine (100-300 microg) to maintain mean arterial pressure within 20% of baseline during 1.5 MAC anesthesia. Conclusions In common with other volatile anesthetic agents, sevoflurane has an intrinsic dose-dependent cerebral vasodilatory effect. However, this effect is less than that of isoflurane.

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