scholarly journals Orthostatic blood pressure changes and arterial baroreflex sensitivity in elderly subjects

1999 ◽  
Vol 28 (6) ◽  
pp. 522-530 ◽  
Author(s):  
M. James
Keyword(s):  
Blood Pressure ◽  
Baroreflex Sensitivity ◽  
Elderly Subjects ◽  
Arterial Baroreflex ◽  
Orthostatic Blood Pressure ◽  
Pressure Changes ◽  
Arterial Baroreflex Sensitivity

Arterial baroreflex sensitivity and blood pressure variabilities before and after carotid surgery

1991 ◽  
Vol 69 (16) ◽  
pp. 763-768 ◽  
Author(s):  
M. Hirschl ◽  
M. M. Hirschl ◽  
D. Magometschnigg ◽  
B. Liebisch ◽  
O. Wagner ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Baroreflex Sensitivity ◽  
Carotid Surgery ◽  
Arterial Baroreflex ◽  
Before And After ◽  
Arterial Baroreflex Sensitivity

scholarly journals Slow Breathing Improves Arterial Baroreflex Sensitivity and Decreases Blood Pressure in Essential Hypertension

Hypertension ◽  
2005 ◽  
Vol 46 (4) ◽  
pp. 714-718 ◽  
Author(s):  
Chacko N. Joseph ◽  
Cesare Porta ◽  
Gaia Casucci ◽  
Nadia Casiraghi ◽  
Mara Maffeis ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Essential Hypertension ◽  
Baroreflex Sensitivity ◽  
Arterial Baroreflex ◽  
Slow Breathing ◽  
Arterial Baroreflex Sensitivity

scholarly journals Arterial baroreflex function in older adults with neurocardiogenic syncope

2009 ◽  
Vol 32 (3) ◽  
pp. 191 ◽  
Author(s):  
Kenneth M Madden ◽  
Chris Lockhart
Keyword(s):  
Blood Pressure ◽  
Older Adults ◽  
Baroreflex Sensitivity ◽  
Arterial Baroreflex ◽  
Neurocardiogenic Syncope ◽  
Baroreflex Function ◽  
Large Numbers ◽  
Falls In Older Adults ◽  
Spontaneous Baroreflex ◽  
Arterial Baroreflex Sensitivity

Purpose: Neurocardiogenic syncope (formerly vasovagal) accounts for large numbers of falls in older adults and the mechanisms are poorly understood. This study examined the differences in baseline arterial baroreflex function in older adults with and without a neruocardiovascular response to orthostatic stress. Methods: Subjects were divided into two groups based on the presence (TT+ group) or absence (TT- group) of a neurocardiovascular response to upright tilting (70 degree head-up tilt for 10 minutes after 400 micrograms of sublingual nitroglycerin). A neurocardiovascular response was defined as presyncopal symptoms (lightheadedness) in association with at least a 30 mm Hg decrease in blood pressure. Before being divided into groups, baroreflex function was assessed using the spontaneous baroreflex method (baroreflex sensitivity, BRS). This method involves the analysis of “spontaneous” swings in blood pressure and heart rate that are mediated by the arterial baroreflexes. Results: 42 older adults (mean age 70.3±0.7 yr) were recruited, of which 18 were in the TT+ and 24 were in the TT- group. At baseline, the TT+ group demonstrated increased arterial baroreflex sensitivity in response to negative blood pressure sequences only (BRSdown, 11.2±1.9 vs. 7.3±1.0 ms/mm Hg, P=0.011). During tilt, the TT+ group demonstrated a much larger decrease in overall arterial baroreflex sensitivity than the TT- group (-6.8±1.2 vs. –3.2±0.9 ms/mm Hg, P=0.012). There was a negative correlation between BRSdown and length of tilt table test (r=-0.329, P=0.041) in the TT+ subjects. Conclusion: Older adults with neurocardiogenic syncope have exaggerated arterial baroreflex sensitivity at baseline.

Relationship between arterial baroreflex, cardiopulmonary vagal reflex and renal natriuretic response to saline in conscious rabbits

1998 ◽  
Vol 95 (6) ◽  
pp. 693-699 ◽  
Author(s):  
Nadine MAYBAUM ◽  
Elena GORODETSKY ◽  
Marta WEINSTOCK
Keyword(s):  
Blood Pressure ◽  
Blood Flow ◽  
Renal Blood Flow ◽  
Sodium Excretion ◽  
Baroreflex Sensitivity ◽  
Saline Infusion ◽  
Vagal Afferents ◽  
Arterial Baroreflex ◽  
Cardiopulmonary Baroreceptors ◽  
Arterial Baroreflex Sensitivity

1. We have previously shown that normotensive rabbits with a genetic impairment in arterial baroreflex sensitivity showed a delayed sodium excretion and failed to increase their renal blood flow in response to a saline infusion that did not alter blood pressure. These renal haemodynamic and excretory abnormalities were abolished by renal denervation. The present study determined the sensitivity of the cardiopulmonary baroreceptors and the renal response to a mild saline infusion in normotensive rabbits varying widely in their arterial baroreflex sensitivity. 2. Sensitivity of cardiopulmonary baroreceptors was assessed from the slope of the relationship of the change in both blood pressure and heart rate and the dose of phenylbiguanide, a stimulator of vagal afferents. 3. The change in renal blood flow and lithium and sodium excretion was measured in response to saline, infused at a rate of 0.11 ml·min-1·kg-1 into the ear vein. Urine was collected via a urethral catheter and renal blood flow was measured by para-aminohippurate clearance. 4. A significant correlation was found between the magnitude of the gain of the cardiac arterial baroreflex and the sensitivity of the cardiopulmonary baroreceptor response to phenylbiguanide. The latter was significantly correlated to renal blood flow and lithium clearance 60–90 min after the start of the saline infusion. 5. It was also found that in some normotensive rabbits there was a blunting of cardiovascular regulation as indicated by a reduced sensitivity of cardiopulmonary and arterial baroreceptors. This may explain their abnormal haemodynamic and natriuretic response to salt.

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