Reversible Severe Tricuspid Regurgitation with Right Heart Failure Associated with Thyrotoxicosis

Thyroid ◽  
2006 ◽  
Vol 16 (8) ◽  
pp. 813-814 ◽  
Author(s):  
Jae-Hyeong Park ◽  
Minho Shong ◽  
Jae-Hwan Lee ◽  
Si Wan Choi ◽  
Jin Ok Jeong ◽  
...  
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Right Heart Failure ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

scholarly journals Tachycardia-Induced Right Heart Failure and Severe Tricuspid Regurgitation That Improved with Medication

2015 ◽  
Vol 51 (3) ◽  
pp. 135
Author(s):  
Young Ae Yang ◽  
Dong Heon Yang ◽  
Hong Nyun Kim ◽  
Sang Hoon Kwon ◽  
Se Young Jang ◽  
...  
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Right Heart Failure ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

Normal myocardial function in severe right ventricular volume overload hypertrophy

2001 ◽  
Vol 280 (1) ◽  
pp. H11-H16 ◽  
Author(s):  
Yuji Ishibashi ◽  
Judith C. Rembert ◽  
Blase A. Carabello ◽  
Shintaro Nemoto ◽  
Masayoshi Hamawaki ◽  
...  
Keyword(s):  
Heart Failure ◽  
Right Ventricular ◽  
Tricuspid Regurgitation ◽  
Contractile Function ◽  
Right Heart Failure ◽  
Ventricular Volume ◽  
Left Ventricular ◽  
Right Ventricular Volume ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

Severe left ventricular volume overloading causes myocardial and cellular contractile dysfunction. Whether this is also true for severe right ventricular volume overloading was unknown. We therefore created severe tricuspid regurgitation percutaneously in seven dogs and then observed them for 3.5–4.0 yr. All five surviving operated dogs had severe tricuspid regurgitation and right heart failure, including massive ascites, but they did not have left heart failure. Right ventricular cardiocytes were isolated from these and from normal dogs, and sarcomere mechanics were assessed via laser diffraction. Right ventricular cardiocytes from the tricuspid regurgitation dogs were 20% longer than control cells, but neither the extent (0.171 ± 0.005 μm) nor the velocity (2.92 ± 0.12 μm/s) of sarcomere shortening differed from controls (0.179 ± 0.005 μm and 3.09 ± 0.11 μm/s, respectively). Thus, despite massive tricuspid regurgitation causing overt right heart failure, intrinsic right ventricular contractile function was normal. This finding for the severely volume-overloaded right ventricle stands in distinct contrast to our finding for the left ventricle severely volume overloaded by mitral regurgitation, wherein intrinsic contractile function is depressed.

scholarly journals From the Veins to the Heart: A Rare Cause of Varicose Veins

2015 ◽  
Vol 2015 ◽  
pp. 1-3 ◽  
Author(s):  
Michele Dalla Vestra ◽  
Elisabetta Grolla ◽  
Luca Bonanni ◽  
Vittorio Dorrucci ◽  
Fabio Presotto ◽  
...  
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Ultrasound Examination ◽  
Varicose Veins ◽  
Right Heart Failure ◽  
Pulsating Flow ◽  
Lower Limbs ◽  
Bedside Ultrasound ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

The presence of pulsating varicous veins is an uncommon finding, generically attributed to right heart failure. The precise causes of this phenomenon have been poorly defined in the literature. The finding of this infrequent condition is important because it may be a sign of major diseases, often not known. Here we described a 75-year-old woman presented to the Angiology Unit for the presence of bilateral pulsatile swelling in her groin and along both lower limbs. A bedside ultrasound examination showed an arterial like pulsating flow both in the superficial and in the deep veins of the lower limbs due to a severe tricuspid regurgitation not previously known.

Improvement of Severe Tricuspid Regurgitation with Right Heart Failure Associated with Thyrotoxicosis due to Graves' Disease

2009 ◽  
Vol 17 (1) ◽  
pp. 22 ◽  
Author(s):  
Sun Ho Hwang ◽  
Kyung Hee Hong ◽  
Hyung Min Noh ◽  
Chan Young Park ◽  
Jong Beom Kim ◽  
...  
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Right Heart Failure ◽  
Graves Disease ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

scholarly journals Severe tricuspid regurgitation and isolated right heart failure due to thyrotoxicosis

2012 ◽  
Vol 64 (6) ◽  
pp. 600-602 ◽  
Author(s):  
Maria Bonou ◽  
Konstantinos M. Lampropoulos ◽  
Maria Andriopoulou ◽  
Dimitrios Kotsas ◽  
John Lakoumentas ◽  
...  
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Right Heart Failure ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

Effects of Tolvaptan on Congestion and Neurohumoral Factors: A Case of Right Heart Failure Caused by Severe Tricuspid Regurgitation

2014 ◽  
Vol 20 (10) ◽  
pp. S208-S209
Author(s):  
Yodo Tamaki ◽  
Yukiko Hayama ◽  
Naoaki Onishi ◽  
Soichiro Enomoto ◽  
Makoto Miyake ◽  
...  
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Right Heart Failure ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

scholarly journals Right heart failure secondary to traumatic tricuspid flail with severe tricuspid regurgitation

2020 ◽  
Vol 6 (5) ◽  
Author(s):  
Rodrigo Escalante-Armenta ◽  
Miguel Tapia-Sansores ◽  
María Camila Aguirre-Orozco ◽  
Luis Javier Castellanos-Vizcaíno ◽  
Nilda Espinola-Zavaleta
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Right Heart Failure ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation

scholarly journals Hypertrophic Cardiomyopathy and Tricuspid Regurgitation with Right Heart Failure Mimicking Constrictive Pericarditis: A Rare Case Report

2016 ◽  
Vol 11 (1) ◽  
pp. 45-47
Author(s):  
SMG Saklayen ◽  
Rakibul Hasan ◽  
Redoy Ranjan ◽  
Mostafizur Rahman ◽  
Rezwanul Hoque ◽  
...  
Keyword(s):  
Heart Failure ◽  
Tricuspid Valve ◽  
Tricuspid Regurgitation ◽  
Constrictive Pericarditis ◽  
Right Heart Failure ◽  
Right Atrial ◽  
Atrial Wall ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation ◽  
Rare Case Report

Cardiomyopathy is the measurable deterioration of the function of the myocardium for any reason, usually leading to heart failure. Tricuspid regurgitation may result from structural alterations of any one or all of the components of the tricuspid valve apparatus which include the leaflets, chordae tendinae, annulus, and papillary muscles or adjacent right ventricular muscle. We are reporting a case of Cardiomyopathy with tricuspid regurgitation with right heart failure in a 38 years male. Preoperatively he was diagnosed as a case of constrictive pericarditis. Diagnosis of Tricuspid regurgitation with cardiomegaly was confirmed peroperatively. During operatrion tricuspid valve anatomy dimunited and severe tricuspid regurgitation was identified. Grossly dilated RA, RV identified and other anatomy of heart was normal. De-Vega Tricuspid anuloplasty done with pledgeted stich 2-0 polyster. Part of right atrial wall (2X2.5 inch) excised and resected portion sent for histopathology. Postoperative course was uneventful with marked improvement of symptoms.University Heart Journal Vol. 11, No. 1, January 2015; 45-47

scholarly journals Feasibility of Transcatheter Caval Valve Implantation to Improve Sleep-Disordered Breathing in Patients With Severe Tricuspid Regurgitation—A Pilot Study

2021 ◽  
Vol 8 ◽  
Author(s):  
Youmeng Wang ◽  
Roberto Fernandes Branco ◽  
Andrea Fietzeck ◽  
Thomas Penzel ◽  
Christoph Schöbel
Keyword(s):  
Heart Failure ◽  
Tricuspid Regurgitation ◽  
Sleep Disordered Breathing ◽  
Right Heart Failure ◽  
Right Heart ◽  
Severe Tricuspid Regurgitation ◽  
Laboratory Results ◽  
Echocardiographic Parameters ◽  
Valve Implantation

Transcatheter caval valve implantation (CAVI) has been evaluated as a treatment option for inoperable patients with severe symptomatic tricuspid regurgitation (TR). We studied the effect of CAVI on sleep disorder breathing (SDB) in patients with right heart failure and TR. Twenty right heart failure patients with severe symptomatic TR who underwent portable monitoring of SDB (ApneaLink), echocardiography, cardiopulmonary exercise (CPET), and laboratory testing were enrolled. This was a single-center, nonblinded study. There were no significant changes in sleep variables, echocardiographic parameters, laboratory results, lung function, and CPET after CAVI. In conclusion, these data suggest that CAVI may not have an effect on SDB; however, additional follow-up fully powered studies with appropriate statistical analyses are needed.

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