Recurrent Hyperthyroidism in Consecutive Pregnancies Characterized by Hyperemesis

Thyroid ◽  
1996 ◽  
Vol 6 (5) ◽  
pp. 465-466 ◽  
Author(s):  
SHAHLA NADER ◽  
JOAN MASTROBATTISTA
Keyword(s):  
Recurrent Hyperthyroidism

RECURRENT HYPERTHYROIDISM AFTER IODINE-125 TREATMENT

The Lancet ◽  
1975 ◽  
Vol 306 (7934) ◽  
pp. 564-565 ◽  
Author(s):  
JanD. Wiener
Keyword(s):  
Iodine 125 ◽  
Recurrent Hyperthyroidism

A study of thyroid function after subtotal thyroidectomy for Graves' disease: particularly on TRH tests, T3 suppression tests and antithyroid antibodies in euthyroid patients

1983 ◽  
Vol 103 (1) ◽  
pp. 28-33 ◽  
Author(s):  
Osamu Fukino ◽  
Hajime Tamai ◽  
Shinichi Fujii ◽  
Noriyuki Ohsako ◽  
Sunao Matsubayashi ◽  
...  
Keyword(s):  
Elevated Serum ◽  
Subtotal Thyroidectomy ◽  
Graves Disease ◽  
Trh Test ◽  
Antithyroid Antibodies ◽  
Abnormal Response ◽  
Suppression Test ◽  
Recurrent Hyperthyroidism ◽  
Serum Tsh

Abstract. Of 305 patients who underwent subtotal thyroidectomy for Graves' disease between 1969 and 1975, recurrent hyperthyroidism was found in 31 (10.2%) and hypothyroidism in 18 (5.9%). The remaining 256 patients were clinically euthyroid, but an elevated serum TSH level was found in 104 (34.1%) and an elevated serum T3 level in 19 (6.28%). In 57 of 133 clinically and biochemically euthyroid patients, a TRH test, T3 suppression test and measurement of antithyroid antibodies were performed. Twenty-nine of the 57 patients (50.9%) showed an abnormal response to TRH. Eight of these (14.0%) showed an impaired or absent response. The T3 suppression test showed that 15 of the 57 patients (26.3%) were non-suppressible. Positive antithyroid antibodies, especially antimicrosomal antibodies, were more frequent in non-suppressible and TRH-non-responsive patients than in suppressible and TRH-responsive patients. It is suggested that after operation for Graves' disease: 1) only half of the clinically euthyroid patients were biochemically euthyroid, 2) of the clinically and biochemically euthyroid patients, there were many with abnormalities in TRH responsiveness and T3 suppressibility, and 3) thyroid functional status is unstable and long careful follow-up is important after operation for Graves' disease.

T3 TOXICOSIS IN ADOLESCENCE: PRESENTATION AS RECURRENT HYPERTHYROIDISM

PEDIATRICS ◽  
1973 ◽  
Vol 52 (5) ◽  
pp. 649-652
Author(s):  
T. W. AvRuskin ◽  
S. Tang ◽  
L. Shenkman ◽  
C. S. Hollander
Keyword(s):  
Female Patient ◽  
Intravenous Infusion ◽  
Pediatric Population ◽  
Normal Serum ◽  
Thyrotropin Releasing Hormone ◽  
Graves Disease ◽  
Adolescent Patient ◽  
Serum T3 ◽  
Recurrent Hyperthyroidism ◽  
Serum Tsh

The second adolescent patient with recurrent hyperthyroidism caused by isolated hypersecretion of T3 (T3 toxicosis) has been discovered. In 1969, this 17-year-old female patient initially presented with conventional Graves' disease was treated with propylthiouracil, but discontinued her medication after three months. Two and one-half half years later, she developed clinical and laboratory tory features of recurrent Graves' disease, save for a normal total and free thyroxine (T4). Serum T3 by radioimmunoassay was elevated (210 ng/100 ml) and thyroid binding globulin capacity for T4 was normal. Serum TSH was not detectable and failed to rise after intravenous infusion of 400 mg thyrotropin-releasing hormone. Other parameters of T3 toxicosis, as noted in adults with this syndrome, were confirmed. T3 toxicosis presenting as recurrent hyperthyroidism may be more common than previously recognized in the pediatric population.

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