scholarly journals Do Lifestyle Factors Including Smoking, Alcohol, and Exercise Impact Your Risk of Developing Kidney Stone Disease? Outcomes of a Systematic Review

2020 ◽  
Author(s):  
Patrick Jones ◽  
Sulaiman Sadaf Karim ◽  
Kithmini N. Gamage ◽  
Theodoras Tokas ◽  
Enakshee Jamnadass ◽  
...  
Author(s):  
Antonio Carbone ◽  
Yazan Al Salhi ◽  
Andrea Tasca ◽  
Giovanni Palleschi ◽  
Andrea Fuschi ◽  
...  

2016 ◽  
Vol 30 (3) ◽  
pp. 246-253 ◽  
Author(s):  
Yee Wong ◽  
Paul Cook ◽  
Paul Roderick ◽  
Bhaskar K. Somani

2017 ◽  
Vol 31 (8) ◽  
pp. 729-735 ◽  
Author(s):  
Robert M. Geraghty ◽  
Silvia Proietti ◽  
Olivier Traxer ◽  
Matthew Archer ◽  
Bhaskar K. Somani

2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Chien-Heng Chen ◽  
Jia-In Lee ◽  
Jhen-Hao Jhan ◽  
Yung-Chin Lee ◽  
Jiun-Hung Geng ◽  
...  

AbstractResearch indicates smoking increases the risk of various kidney diseases, although the risk of developing kidney stone disease in non-smokers exposed to secondhand smoke is unknown. This study analyzed a total of 19,430 never-smokers with no history of kidney stone disease who participated in the Taiwan Biobank from 2008 to 2019. They were divided into two groups by secondhand smoke exposure; no exposure and exposure groups; the mean age of participants was 51 years, and 81% were women. Incident kidney stone development was observed in 352 (2.0%) and 50 (3.3%) participants in the no exposure and exposure groups during a mean follow-up of 47 months. The odds ratio (OR) of incident kidney stone was significantly higher in the exposure group than the no exposure group [OR, 1.64; 95% confidence interval (95% CI) 1.21 to 2.23]. Participants with > 1.2 h per week exposure were associated with almost twofold risk of developing kidney stones compared with no exposure (OR, 1.92; 95% CI 1.29 to 2.86). Our study suggests that secondhand smoke is a risk factor for development of kidney stones and supports the need for a prospective evaluation of this finding.


2020 ◽  
Vol 46 (Supp1) ◽  
pp. S92-S103
Author(s):  
Kithmini Nadeeshani Gamage ◽  
◽  
Enakshee Jamnadass ◽  
Sadaf Karim Sulaiman ◽  
Amelia Pietropaolo ◽  
...  

2021 ◽  
Vol 127 (4) ◽  
pp. 38-43
Author(s):  
Ilia Kordubailo ◽  
Oleg Nikitin ◽  
Olga Nishkumay ◽  
Pavlo Samchuk

the prevalence of kidney stone disease (KSD) and osteoporosis (OP) increases every year. In the prevention of osteoporosis, it is important to consume a sufficient amount of calcium-rich foods in the daily diet, as well as the use of calcium. One of the important reasons for the insufficient use of calcium-containing products and medicines is the anxiety not only of patients, but, very importantly, of doctors as much as possible. This has serious justification, as nephrolithiasis occurs in approximately 5% of the population, and the risk of developing kidney stones during life is 8-10%. It is believed that secondary hyperparathyroidism, which is caused by hypocalcemia due to insufficient consumption of calcium-containing products and impaired renal function, leads to increased bone resorption, formation of kidney stone disease. It is important to consider that against the background of hypertensive, atherosclerotic kidney disease, tubulo-interstitial lesions of the kidneys with decreasing glomerular filtration rate decreases the synthesis of 1α-hydroxylase - an enzyme by which 25-hydroxycholecalciferol (25 (OH) active D3, calcium) form of vitamin D3–1.25 dihydroxycholecalciferol (1.25 (OH) 2D3, calcitriol - D-hormone) and secondary hyperparathyroidism develops. In this case, the purpose of correction along with the treatment of urolithiasis (spa treatment, given the attendance of the presence of KSD, to carry out the distance lithotripsy), intake of active metabolites of vitamin D (should be started with low doses, independent of the initial PTH concentration, and then titrated based on the PTH response) conducting X-ray densitometry.


Nutrients ◽  
2021 ◽  
Vol 13 (12) ◽  
pp. 4270
Author(s):  
Yazeed Barghouthy ◽  
Mariela Corrales ◽  
Bhaskar Somani

Objectives: Kidney stone disease (KSD) has a strong association with diet metabolic syndrome. This review aims at exploring the lithogenic risk posed by the current most popular diets. Our approach was to search for the effect of each diet type on the major urinary risk factors, to try to draw conclusions regarding the association of a specific diet type and KSD. Methods: This systematic review searched for the available literature exploring the association between the existing popular fad diets and KSD. Articles in English, French and Spanish were included, without restriction of the search period with the final search done in August 2021. Results: Total number of studies and studies for each diet type was as follows: 22 articles for the low carbohydrate diet, 20 articles for high protein diets, 26 articles for vegetarian and vegan diets. There exists a substantial variability in different low carbohydrate and high protein diets, and considerable overlap between modern popular fad diets. High carbohydrate intake might increase urine uric acid, calcium and oxalate levels. High protein diets increase urine calcium and uric acid and lower urine pH and citrate. Consumption of fruits and vegetables increases the urinary volume and urinary citrate. In vegan diets, sufficient daily calcium intake is important to avoid possible secondary hyperoxaluria. Conclusions: Few studies evaluated the direct relationship between modern fad diets and KSD. In general, the reduction of carbohydrate in the diet, and counterbalancing protein rich diets with sufficient intake of fruits and vegetables, seem to play a protective role against KSD formation. Maintaining sufficient calcium intake in vegan and vegetarian diets is important. Additional research is needed to directly evaluate the link between KSD and each diet type.


2021 ◽  
Vol 22 (6) ◽  
Author(s):  
Y. Premakumar ◽  
N. Gadiyar ◽  
B. M. Zeeshan Hameed ◽  
D. Veneziano ◽  
B. K. Somani

Abstract Purpose of review We aim to provide an up-to-date literature review to further characterise the association of kidney stone disease (KSD) with gastrointestinal (GI) surgery. As KSD is associated with significant morbidity, it is important to quantify and qualify this association to provide better care and management for the patient subgroup. Objective To perform a systematic review of the existing literature to evaluate the association of KSD following GI surgery. Methods A literature search was performed of the following databases: MEDLINE, EMBASE, Scopus, Google Scholar, Key Urology, Uptodate and Cochrane Trials from January 2000 to June 2020. Recent Findings A total of 106 articles were identified, and after screening for titles, abstracts and full articles, 12 full papers were included. This involved a total of 9299 patients who underwent primary GI surgery. Over a mean follow-up period of 5.4 years (range: 1–14.4 years), 819 (8.8%) developed KSD, varying from 1.2 to 83% across studies. The mean time to stone formation was approximately 3 years (range: 0.5–9 years). In the 4 studies that reported on the management of KSD (n = 427), 38.6% went on to have urological intervention. Summary There is a high incidence of KSD following primary GI surgery, and after a mean follow-up of 3 years, around 9% of patients developed KSD. While the GI surgery was done for obesity, inflammatory bowel disease or cancer, the risk of KSD should be kept in mind during follow-up, and prompt urology involvement with metabolic assessment, medical and or surgical management offered as applicable.


Sign in / Sign up

Export Citation Format

Share Document