scholarly journals The Autism Genetic Resource Exchange: A Resource for the Study of Autism and Related Neuropsychiatric Conditions

2001 ◽  
Vol 69 (2) ◽  
pp. 463-466 ◽  
Author(s):  
Daniel H. Geschwind ◽  
Janice Sowinski ◽  
Catherine Lord ◽  
Portia Iversen ◽  
Jonathan Shestack ◽  
...  
Keyword(s):  
Genetic Resource ◽  
Resource Exchange ◽  
Autism Genetic Resource Exchange ◽  
Neuropsychiatric Conditions

Search for autism loci by combined analysis of Autism Genetic Resource Exchange and Finnish families

2005 ◽  
Vol 59 (1) ◽  
pp. 145-155 ◽  
Author(s):  
Tero Ylisaukko-oja ◽  
Maricela Alarcón ◽  
Rita M. Cantor ◽  
Mari Auranen ◽  
Raija Vanhala ◽  
...  
Keyword(s):  
Genetic Resource ◽  
Resource Exchange ◽  
Combined Analysis ◽  
Autism Genetic Resource Exchange

scholarly journals BBGRE: brain and body genetic resource exchange

Database ◽  
2013 ◽  
Vol 2013 ◽  
Author(s):  
Joo Wook Ahn ◽  
Abhishek Dixit ◽  
Caroline Johnston ◽  
Caroline M. Ogilvie ◽  
David A. Collier ◽  
...  
Keyword(s):  
Genetic Resource ◽  
Resource Exchange

Neurotropic enteroviruses co-opt “fair-weather-friend” commensal gut microbiota to drive host infection and central nervous system disturbances

2019 ◽  
Vol 42 ◽  
Author(s):  
Kevin B. Clark
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Gut Bacteria ◽  
Infection Cycle ◽  
Fair Weather ◽  
Host Health ◽  
Microbe Interactions ◽  
Animal Hosts ◽  
Host Infection ◽  
Neuropsychiatric Conditions

Abstract Some neurotropic enteroviruses hijack Trojan horse/raft commensal gut bacteria to render devastating biomimicking cryptic attacks on human/animal hosts. Such virus-microbe interactions manipulate hosts’ gut-brain axes with accompanying infection-cycle-optimizing central nervous system (CNS) disturbances, including severe neurodevelopmental, neuromotor, and neuropsychiatric conditions. Co-opted bacteria thus indirectly influence host health, development, behavior, and mind as possible “fair-weather-friend” symbionts, switching from commensal to context-dependent pathogen-like strategies benefiting gut-bacteria fitness.

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