scholarly journals Calcium mobilization from mitochondria in synaptic transmitter release

2003 ◽  
Vol 163 (3) ◽  
pp. 441-443 ◽  
Author(s):  
Rosario Rizzuto
Keyword(s):  
Transmitter Release ◽  
Calcium Mobilization ◽  
Synaptic Potentiation ◽  
Cell Stimulation ◽  
Voltage Gated ◽  
Unusual Form ◽  
Synaptic Transmitter Release ◽  
Voltage Gated Channels ◽  
Issue Reports ◽  
Synaptic Transmitter

Mitochondria can rapidly accumulate and release Ca2+ upon cell stimulation. A paper by Yang and coworkers in this issue reports an unusual form of synaptic potentiation, dependent on Ca2+ release from mitochondria through the Na+/Ca2+ exchanger and triggered by Na+ entry through voltage-gated channels (Yang et al., 2003).

Calcium Action in Synaptic Transmitter Release

1987 ◽  
Vol 10 (1) ◽  
pp. 633-693 ◽  
Author(s):  
G J Augustine ◽  
M P Charlton ◽  
S J Smith
Keyword(s):  
Transmitter Release ◽  
Synaptic Transmitter Release ◽  
Synaptic Transmitter

scholarly journals Ionic Requirements of Synaptic Transmitter Release

Nature ◽  
1967 ◽  
Vol 215 (5101) ◽  
pp. 651-651 ◽  
Author(s):  
B. KATZ ◽  
R. MILEDI
Keyword(s):  
Transmitter Release ◽  
Synaptic Transmitter Release ◽  
Synaptic Transmitter

scholarly journals Mobile Calcium Channels Contribute to Variability of Pre-synaptic Transmitter Release

2018 ◽  
Vol 114 (3) ◽  
pp. 152a-153a
Author(s):  
Martin Heine ◽  
Jennifer Heck ◽  
Pierre Parutto ◽  
Anna Ciuraszkiewicz ◽  
Arthur Bikbaev ◽  
...  
Keyword(s):  
Calcium Channels ◽  
Transmitter Release ◽  
Synaptic Transmitter Release ◽  
Synaptic Transmitter

Presynaptic Mechanisms of Endocannabinoid-Mediated Long-Term Depression in the Hippocampus

2009 ◽  
Vol 102 (4) ◽  
pp. 2009-2012 ◽  
Author(s):  
Carlos A. Lafourcade
Keyword(s):  
Transmitter Release ◽  
Cannabinoid Receptors ◽  
Long Term Depression ◽  
Synaptic Transmitter Release ◽  
Presynaptic Mechanisms ◽  
Endogenous Cannabinoids ◽  
Insight Into ◽  
Synaptic Transmitter

Endogenous cannabinoids (eCBs) mobilized from postsynaptic cells activate presynaptic cannabinoid receptors and thereby inhibit synaptic transmitter release. The inhibition can be either brief (seconds) or long-lasting (minutes to hours). Recent studies provide insight into the presynaptic mechanisms responsible for long-lasting, eCB-mediated long-term depression. Among these, the proteins PKA and RIM1α appear to be crucial, although other possibilities are emerging.

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