scholarly journals Rodent and Human Mast Cells Produce Functionally Significant Intracellular Reactive Oxygen Species but Not Nitric Oxide

2004 ◽  
Vol 279 (47) ◽  
pp. 48751-48759 ◽  
Author(s):  
Emily J. Swindle ◽  
Dean D. Metcalfe ◽  
John W. Coleman
Keyword(s):  
Nitric Oxide ◽  
Reactive Oxygen Species ◽  
Mast Cells ◽  
Reactive Oxygen ◽  
Intracellular Reactive Oxygen Species ◽  
Oxygen Species ◽  
Human Mast Cells

scholarly journals Fusigen Reduces Intracellular Reactive Oxygen Species and Nitric Oxide Levels

In Vivo ◽  
2019 ◽  
Vol 33 (2) ◽  
pp. 425-432
Author(s):  
VORAWAT KITIYANANT ◽  
PONGTHARIN LOTRAKUL ◽  
CHOMPOONIK KANCHANABANCA ◽  
PANUWAT PADUNGROS ◽  
HUNSA PUNNAPAYAK ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Intracellular Reactive Oxygen Species ◽  
Oxygen Species

Hybrid vesicles as intracellular reactive oxygen species and nitric oxide generators

Nanoscale ◽  
2019 ◽  
Vol 11 (24) ◽  
pp. 11530-11541 ◽  
Author(s):  
Yan Zhang ◽  
Noga Gal ◽  
Fabian Itel ◽  
Isabella N. Westensee ◽  
Edit Brodszkij ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Intracellular Reactive Oxygen Species ◽  
Host Cells ◽  
Oxygen Species

Artificial organelles are envisioned as nanosized assemblies with intracellular biocatalytic activity to provide the host cells with non-native or missing/lost function.

The Role of Reactive Oxygen Species, Kinases, Hydrogen Sulfide, and Nitric Oxide in the Regulation of Autophagy and Their Impact on Ischemia and Reperfusion Injury in the Heart

2020 ◽  
Vol 16 ◽  
Author(s):  
Andrey Krylatov ◽  
Leonid Maslov ◽  
Sergey Y. Tsibulnikov ◽  
Nikita Voronkov ◽  
Alla Boshchenko ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Reactive Oxygen Species ◽  
Hydrogen Sulfide ◽  
Ischemia Reperfusion ◽  
Reactive Oxygen ◽  
Ischemia And Reperfusion ◽  
Oxygen Species ◽  
Ischemia And Reperfusion Injury ◽  
Adaptive Process

: There is considerable evidence in the heart that autophagy in cardiomyocytes is activated by hypoxia/reoxygenation (H/R) or in hearts by ischemia/reperfusion (I/R). Depending upon the experimental model and duration of ischemia, increases in autophagy in this setting maybe beneficial (cardioprotective) or deleterious (exacerbate I/R injury). Aside from the conundrum as to whether or not autophagy is an adaptive process, it is clearly regulated by a number of diverse molecules including reactive oxygen species (ROS), various kinases, hydrogen sulfide (H2S) and nitric oxide (NO). The purpose this review is to address briefly the controversy regarding the role of autophagy in this setting and to examine a variety of disparate molecules that are involved in its regulation.

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