Impact of Nd : YAG laser radiation (λ = 1.44 μm) on myocardial tissue in the treatment of coronary heart disease by transmyocardial laser revascularisation

2019 ◽  
Vol 49 (10) ◽  
pp. 982-987
Author(s):  
S V Belov ◽  
Yu K Danileiko ◽  
A B Egorov ◽  
L G Shilin ◽  
A M Shulutko
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Laser Radiation ◽  
Myocardial Tissue ◽  
Yag Laser ◽  
Transmyocardial Laser Revascularisation

scholarly journals Anisotropic microfibrous scaffolds enhance the organization and function of cardiomyocytes derived from induced pluripotent stem cells

2017 ◽  
Vol 5 (8) ◽  
pp. 1567-1578 ◽  
Author(s):  
Maureen Wanjare ◽  
Luqia Hou ◽  
Karina H. Nakayama ◽  
Joseph J. Kim ◽  
Nicholas P. Mezak ◽  
...  
Keyword(s):  
Stem Cells ◽  
Coronary Heart Disease ◽  
Heart Disease ◽  
Induced Pluripotent Stem Cells ◽  
Pluripotent Stem Cells ◽  
Myocardial Tissue ◽  
Tissue Constructs ◽  
Induced Pluripotent ◽  
And Function

Engineering of myocardial tissue constructs is a promising approach for treatment of coronary heart disease.

scholarly journals Fuling-Guizhi Herb Pair in Coronary Heart Disease: Integrating Network Pharmacology and In Vivo Pharmacological Evaluation

2020 ◽  
Vol 2020 ◽  
pp. 1-10 ◽  
Author(s):  
Bailu Duan ◽  
Lintao Han ◽  
Shuping Ming ◽  
JingJing Li ◽  
Qiong Wang ◽  
...  
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Experimental Verification ◽  
Inflammatory Responses ◽  
Underlying Mechanism ◽  
Network Pharmacology ◽  
Myocardial Tissue ◽  
Pharmacological Mechanism ◽  
Underlying Mechanisms

The Fuling (Poria cocos)-Guizhi (Cinnamomi ramulus) herb pair (FGHP) is a commonly used traditional Chinese herbal formula with coronary heart disease (CHD) treatment potential. However, the mechanism of FGHP in the treatment of CHD was still unclear. In this study, the action targets and underlying mechanism of FGHP against CHD were successfully achieved by combined network pharmacology prediction with experimental verification. 76 common targets were screened out by overlapping the chemical-protein data of FGHP and CHD-related targets. Then, two key targets were further selected for verification by using western blot analysis after analyzing PPI, GO function, and KEGG pathway. Results indicated FGHP could alleviate CHD syndromes and regulate inflammatory responses in acute myocardial ischemia rats, and the reduction of expression of TNF-α and IL-6 in myocardial tissue would be one of its possible underlying mechanisms. Our work demonstrated that network pharmacology combined with experimental verification provides a credible method to elucidate the pharmacological mechanism of FGHP against CHD.

Support, stress, and recovery from coronary heart disease: A longitudinal causal model.

1989 ◽  
Vol 8 (2) ◽  
pp. 175-193 ◽  
Author(s):  
Alan F. Fontana ◽  
Robert D. Kerns ◽  
Roberta L. Rosenberg ◽  
Kathleen L. Colonese
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Causal Model
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