Enhanced kinematics in oriented and Cosserat solids with structural inhomogeneity

2021 ◽  
Author(s):  
Sergey A. Lychev ◽  
Konstantin Koifman ◽  
Anton Petrenko
1982 ◽  
Vol 15 (12) ◽  
pp. 2561-2575 ◽  
Author(s):  
Y Tsuchiya ◽  
S Takeda ◽  
S Tamaki ◽  
Y Waseda ◽  
E F W Seymour

1994 ◽  
Vol 35 (5) ◽  
pp. 585-592
Author(s):  
T. I. Ivanova ◽  
O. V. Frank-Kamenetskaya ◽  
S. V. Moshkin ◽  
M. Yu. Vlasov

1995 ◽  
Vol 65 (6) ◽  
pp. 339-342 ◽  
Author(s):  
V.K. Kothari ◽  
A.K. Sengupta ◽  
J.K. Sensarma

2007 ◽  
Vol 539-543 ◽  
pp. 493-498 ◽  
Author(s):  
Ivan Saxl ◽  
Vàclav Sklenička ◽  
L. Ilucová ◽  
Milan Svoboda ◽  
Petr Král

Considerable structural inhomogeneity and anisotropy were found even after eight ECAP passes in high purity aluminium and the creep loading of ECAP material at 473K, 15MPa resulted in scattered fracture times ~ 20-60 hours. The structure revealed by orientation imaging microscopy with different disclination bounds was analysed by stereological methods. The effect of inhomogeneity and grain orientation on the creep fracture time was assessed.


2018 ◽  
Vol 15 (147) ◽  
pp. 20180492 ◽  
Author(s):  
Xunjie Yu ◽  
Raphaël Turcotte ◽  
Francesca Seta ◽  
Yanhang Zhang

Microstructural deformation of elastic lamellae plays important roles in maintaining arterial tissue homeostasis and regulating vascular smooth muscle cell fate. Our study unravels the underlying microstructural origin that enables elastic lamellar layers to evenly distribute the stresses through the arterial wall caused by intraluminal distending pressure, a fundamental requirement for tissue and cellular function. A new experimental approach was developed to quantify the spatial organization and unfolding of elastic lamellar layers under pressurization in mouse carotid arteries by coupling physiological extension–inflation and multiphoton imaging. Tissue-level circumferential stretch was obtained from analysis of the deformation of a thick-walled cylinder. Our results show that the unfolding and extension of lamellar layers contribute simultaneously to tissue-level deformation. The inner lamellar layers are wavier and unfold more than the outer layers. This waviness gradient compensates the larger tissue circumferential stretch experienced at the inner surface, thus equalizing lamellar layer extension through the arterial wall. Discoveries from this study reveal the importance of structural inhomogeneity in maintaining tissue homeostasis through the arterial wall, and may have profound implications on vascular remodelling in aging and diseases, as well as in tissue engineering of functional blood vessels.


2020 ◽  
pp. 1303-1320
Author(s):  
Michael B. Ross ◽  
Jessie C. Ku ◽  
Martin G. Blaber ◽  
Chad A. Mirkin ◽  
George C. Schatz

2020 ◽  
pp. 1303-1320
Author(s):  
Michael B. Ross ◽  
Jessie C. Ku ◽  
Martin G. Blaber ◽  
Chad A. Mirkin ◽  
George C. Schatz

2008 ◽  
Vol 23 (11) ◽  
pp. 2984-2989 ◽  
Author(s):  
Jin Man Park ◽  
Do Hyang Kim ◽  
Ki Buem Kim ◽  
Eric Fleury ◽  
Min Ha Lee ◽  
...  

The effect of microstructural inhomogeneities with different length scale on the plasticity of (Ti45Zr16Be20Cu10Ni9)100–xTax (x = 0, 5, and 10) bulk glassy alloys has been studied. The formation of specific heterogeneous microstructures with a different type of structural inhomogeneity, i.e., short-/medium-range ordered clusters or micrometer-scale ductile dendrites combined with a glassy matrix, evolved by appropriately tuning the alloy chemistry, improves the room temperature plasticity up to ∼12.5% and ∼15%, respectively. The pronouncedly enhanced plasticity is mainly attributed to the retardation of shear localization and multiplication of shear bands by controlling the plastic and failure instabilities otherwise responsible for premature failure.


Sign in / Sign up

Export Citation Format

Share Document