Degradation studies of 1, 6-diisocyanatohexane-extended poly (1, 4-butylene succinate) - bioactive glass scaffolds for bone tissue repair applications

2016 ◽  
Author(s):  
Kulwinder Kaur ◽  
K. J. Singh ◽  
Vikas Anand
2017 ◽  
Vol 37 (4) ◽  
pp. 1723-1733 ◽  
Author(s):  
Devis Bellucci ◽  
Luca Desogus ◽  
Selena Montinaro ◽  
Roberto Orrù ◽  
Giacomo Cao ◽  
...  

2017 ◽  
Vol 79 ◽  
pp. 286-295 ◽  
Author(s):  
Devis Bellucci ◽  
Alexandre Anesi ◽  
Roberta Salvatori ◽  
Luigi Chiarini ◽  
Valeria Cannillo

2019 ◽  
Vol 21 (8) ◽  
pp. 1900287 ◽  
Author(s):  
Melek Erol‐Taygun ◽  
Irem Unalan ◽  
Maizlinda Izwana Binti Idris ◽  
João F. Mano ◽  
Aldo R. Boccaccini

2012 ◽  
Vol 2012 ◽  
pp. 1-6 ◽  
Author(s):  
E. De Luna-Bertos ◽  
J. Ramos-Torrecillas ◽  
O. García-Martínez ◽  
L. Díaz-Rodríguez ◽  
C. Ruiz

Nonsteroidal anti-inflammatory drugs (NSAIDs) are commonly used in bone tissue repair treatment for their pharmacological action. The objective of this study was to determine the effect of aspirin, on osteoblast growth, using MG63 cell line as osteoblast model. MTT spectrophotometry results showed that 20, 100, and 1000 μM aspirin doses have an inhibitory effect on growth. Cell cycle analysis revealed that aspirin doses of 100 and 1000 μM arrest the cell cycle in phase GO/G1. Parallel apoptosis/necrosis studies showed no changes in comparison to control cells after treatment with 1 or 10 μM aspirin but a significantly increased percentage of cells in apoptosis at doses of 20, 100, and 1000 μM. We highlight that treatment of osteoblast-like cells with 1000 μM aspirin increased not only the percentage of cells in apoptosis but also the percentage of necrotic cells, which was not observed in aspirin treatments at lower doses.


2018 ◽  
Vol 61 (12) ◽  
pp. 1553-1567 ◽  
Author(s):  
Lin Zhong ◽  
Ying Qu ◽  
Kun Shi ◽  
Bingyang Chu ◽  
Minyi Lei ◽  
...  

2019 ◽  
Vol 207 ◽  
pp. 480-491 ◽  
Author(s):  
Aline E. Aguiar ◽  
Mariana de O. Silva ◽  
Andrea C.D. Rodas ◽  
Celso A. Bertran

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