Flow resistance and electric oscillations in the flux‐flow state of Nb‐25 at.% Zr wires

1974 ◽  
Vol 45 (12) ◽  
pp. 5422-5431 ◽  
Author(s):  
Sakuro Matsunaga
Keyword(s):  
Flow Resistance ◽  
Flow State ◽  
Flux Flow

scholarly journals Layer-Current Waveform of Coaxial Multi-layer HTS Cable Considering the Flux Flow State.

2003 ◽  
Vol 38 (2) ◽  
pp. 54-61 ◽  
Author(s):  
Youngseok KIM ◽  
Sanghyun KIM ◽  
Toshiya HARANO ◽  
Makoto TSUDA ◽  
Naoyuki HARADA ◽  
...  
Keyword(s):  
Flow State ◽  
Current Waveform ◽  
Flux Flow

The behaviour of the flux flow resistance in YBCO/(Ag2O)x

1994 ◽  
Vol 7 (3) ◽  
pp. 99-102 ◽  
Author(s):  
M M Abdelhadi ◽  
K A Ziq
Keyword(s):  
Flow Resistance ◽  
Flux Flow

Flux Flow Resistance in Bi2223 Generated by Pulse Currents

2005 ◽  
Vol 15 (2) ◽  
pp. 2003-2006 ◽  
Author(s):  
K. Mutsuura ◽  
H. Shimizu ◽  
Y. Yokomizu ◽  
T. Matsumura
Keyword(s):  
Flow Resistance ◽  
Flux Flow

Hemodynamic functions and blood viscosity in surface hypothermia

1978 ◽  
Vol 235 (2) ◽  
pp. H136-H143 ◽  
Author(s):  
R. Y. Chen ◽  
S. Chien
Keyword(s):  
Body Temperature ◽  
Blood Viscosity ◽  
Flow Resistance ◽  
Plasma Viscosity ◽  
Low Flow ◽  
Flow State ◽  
Surface Cooling ◽  
Plasma Loss ◽  
Pulmonary Flow ◽  
Anesthetized Dogs

Hemodynamic functions and blood viscosity changes in hypothermia (core approximately 25 degrees C) were studied in 14 pentobarbital-anesthetized dogs subjected to surface cooling. The viscosity of blood (eta B) increased progressively to 173% of that at 37 degrees C when body temperature was lowered to 25 degrees C. The increase in blood viscosity was caused by: a) the direct effect of low temperature on plasma viscosity, b) hemoconcentration as a result of plasma loss, and c) the low-flow (low-shear) state induced by hypothermia. A larger portion of the increased viscosity was caused by the low-flow state in hypothermia. The systemic flow resistance (SFR) increased to 271% of control, and this was attributable about equally to the increases in blood viscosity and systemic vascular hindrance (SFR/eta B). Similarly, the viscosity of blood contributed significantly to raising the pulmonary flow resistance. The relative constancy of mixed venous O2 saturation suggests that the cardiac output at low body temperature is generally adequate to meet the metabolic needs

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