A Site-Specific Comparison of Simplified Procedures for Evaluating Cyclic Resistance of Non-Plastic Silt

Author(s):  
A. S. Bradshaw ◽  
C. D. P. Baxter ◽  
R. A. Green
2008 ◽  
Vol 197 (6) ◽  
pp. 787-794 ◽  
Author(s):  
B. Y. Hernandez ◽  
L. R. Wilkens ◽  
X. Zhu ◽  
K. McDuffie ◽  
P. Thompson ◽  
...  

1994 ◽  
Vol 31 (1) ◽  
pp. 53-60 ◽  
Author(s):  
V.S. Pillai ◽  
P.M. Byrne

The effect of overburden pressure on liquefaction resistance of sand is studied and results of a site-specific investigation are presented. When estimating liquefaction resistance of sand from the indirect approach using the chart suggested by Seed et al. (1984) a correction factor Kσ is applied to account for vertical effective overburden stresses larger than 1 tsf. Published data indicate a decrease in Kσ with increased confining stress but with a wide range of Kσ values for the same confining stresses, predicting significantly differing liquefaction resistance. The effect of confining stresses on liquefaction resistance was investigated as part of a comprehensive seismic assessment of Duncan Dam in British Columbia. The results indicate that Kσ is dependent on confining stresses and the relative density of the soil, and values are generally significantly higher than much of the previously published data. Key words : liquefaction, sand, confining stress, density, cyclic resistance ratio.


1989 ◽  
Vol 9 (4) ◽  
pp. 1507-1512 ◽  
Author(s):  
H Zhu ◽  
H Conrad-Webb ◽  
X S Liao ◽  
P S Perlman ◽  
R A Butow

All mRNAs of yeast mitochondria are processed at their 3' ends within a conserved dodecamer sequence, 5'-AAUAAUAUUCUU-3'. A dominant nuclear suppressor, SUV3-I, was previously isolated because it suppresses a dodecamer deletion at the 3' end of the var1 gene. We have tested the effects of SUV3-1 on a mutant containing two adjacent transversions within a dodecamer at the 3' end of fit1, a gene located within the 1,143-base-pair intron of the 21S rRNA gene, whose product is a site-specific endonuclease required in crosses for the quantitative transmission of that intron to 21S alleles that lack it. The fit1 dodecamer mutations blocked both intron transmission and dodecamer cleavage, neither of which was suppressed by SUV3-1 when present in heterozygous or homozygous configurations. Unexpectedly, we found that SUV3-1 completely blocked cleavage of the wild-type fit1 dodecamer and, in SUV3-1 homozygous crosses, intron conversion. In addition, SUV3-1 resulted in at least a 40-fold increase in the amount of excised intron accumulated. Genetic analysis showed that these phenotypes resulted from the same mutation. We conclude that cleavage of a wild-type dodecamer sequence at the 3' end of the fit1 gene is essential for fit1 expression.


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