Pathophysiology of adrenal hypertension

2002 ◽  
Vol 22 (1) ◽  
pp. 44-53 ◽  
Author(s):  
Miroslava H. Moneva ◽  
Celso E. Gomez-Sanchez
Keyword(s):  
Adrenal Hypertension

Adrenal Hypertension

2017 ◽  
Author(s):  
Naomi D.L. Fisher ◽  
Gail K Adler
Keyword(s):  
Myocardial Infarction ◽  
Adrenal Glands ◽  
Cushing Syndrome ◽  
Rare Tumor ◽  
Cushing Disease ◽  
Increased Risk ◽  
Aldosterone Production ◽  
Artery Disease ◽  
Secondary Causes ◽  
Adrenal Hypertension

The secondary causes of hypertension are associated with the excess of the principal hormones produced by the adrenal glands: cortisol, epinephrine, and aldosterone. Excess aldosterone production is recognized as primary hyperaldosteronism, or primary aldosteronism (PA). Individuals with PA are at increased risk for a variety of disorders, including atrial fibrillation, coronary artery disease, myocardial infarction, and stroke. Pheochromocytoma is a very rare tumor (accounting for fewer than one in 10,000 hypertension cases) and is marked by high secretions of catecholamines, mostly epinephrine as well as norepinephrine. Cushing disease and Cushing syndrome are addressed in a separate review. This review contains 5 highly rendered figures, 4 tables, and 39 references.

Adrenalectomy in Adrenal Hypertension

Cardiology ◽  
1985 ◽  
Vol 72 (1) ◽  
pp. 167-173 ◽  
Author(s):  
J. Hauss ◽  
A. Moeller ◽  
H. Bünte
Keyword(s):  
Adrenal Hypertension

Molecular Mechanism of Na+,K+-ATPase Malfunction in Mutations Characteristic of Adrenal Hypertension

Biochemistry ◽  
2014 ◽  
Vol 53 (4) ◽  
pp. 746-754 ◽  
Author(s):  
Wojciech Kopec ◽  
Bastien Loubet ◽  
Hanne Poulsen ◽  
Himanshu Khandelia
Keyword(s):  
Molecular Mechanism ◽  
Adrenal Hypertension

scholarly journals Morphology of focal adrenal lesions in patients with adrenal hypertension

2018 ◽  
Vol 24 (4) ◽  
pp. 441-447
Author(s):  
V. G. Chobitko ◽  
O. V. Maksimova ◽  
M. M. Orlova ◽  
A. V. Frolova ◽  
A. M. Napsheva
Keyword(s):  
Blood Pressure ◽  
Essential Hypertension ◽  
Morphological Study ◽  
Adrenal Glands ◽  
Exclusion Criterion ◽  
Adrenal Tumors ◽  
Blood Pressure Elevation ◽  
Asymptomatic Pheochromocytoma ◽  
Design And Methods ◽  
Adrenal Hypertension

Objective. To evaluate the morphologу of focal formations of the adrenal glands after adrenalectomy.Design and methods. We analyzed the adrenal glands of 44 patients who underwent the surgery in the Research Institute of Fundamental and Clinical Urinephrology of Saratov in 2004–2016. Renovascular hypertension was the exclusion criterion.Results. The majority of the examined patients (72,7 %) had adrenal hypertension, caused by hormonally active adrenal tumors: pheochromocytoma, corticosteroma and aldosterome. Hormonally inactive tumors were found in 12,5 % patients with adrenal hypertension. Therefore, essential hypertension was diagnosed in these cases. In total 27,3 % patients underwent resection of the incidentalomas, among them 16,7 % were diagnosed as “silent” chromaffin tumors which do not lead to the blood pressure elevation.Conclusions. In the majority of cases (87,5 %), patients who undergo surgery due to adrenal focal formation demonstrate adrenal hypertension related to a hormonally active tumor. Adrenal hypertension is more often diagnosed in middle-aged women, who show higher blood pressure, smaller adrenal glands compared to those who have hormonally inactive adenomas. Based on the morphological study, asymptomatic pheochromocytoma was diagnosed in 16,7 % cases.

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