Corticosteroid osteoporosis

2001 ◽  
Vol 15 (3) ◽  
pp. 401-413 ◽  
Author(s):  
Philip Sambrook ◽  
Nancy E. Lane
1993 ◽  
Vol 328 (24) ◽  
pp. 1747-1752 ◽  
Author(s):  
Philip Sambrook ◽  
Joan Birmingham ◽  
Paul Kelly ◽  
Susan Kempler ◽  
Tuan Nguyen ◽  
...  

Rheumatology ◽  
1995 ◽  
Vol 34 (1) ◽  
pp. 8-12 ◽  
Author(s):  
P. N. SAMBROOK ◽  
G. JONES

1973 ◽  
Vol 2 (2) ◽  
pp. 355-368 ◽  
Author(s):  
J.C. Gallagher ◽  
J. Aaron ◽  
A. Horsman ◽  
R. Wilkinson ◽  
B.E.C. Nordin

1993 ◽  
Vol 7 (3) ◽  
pp. 573-587 ◽  
Author(s):  
Ian R. Reid ◽  
Andrew B. Grey

2002 ◽  
Vol 36 (3) ◽  
pp. 512-516 ◽  
Author(s):  
John A Dougherty

OBJECTIVE: To evaluate the role of risedronate in corticosteroid-induced osteoporosis. DATA SOURCES: Clinical literature was accessed through MEDLINE (1966–February 2001). Key search terms included risedronate, corticosteroid, osteoporosis, and bisphosphonate. DATA SYNTHESIS: Corticosteroid-induced osteoporosis (CIO) is clinically challenging and can lead to fractures. Risedronate, an oral bisphosphonate, has been studied for use in CIO. Trials focusing on the use of risedronate in these patients were reviewed. CONCLUSIONS: Risedronate 5 mg/d increased bone mineral density at lumbar, femoral neck, and trochanter skeletal sites in patients recently initiated on or receiving long-term corticosteroid therapy. Further investigation is needed to determine risedronate's effects on fracture prevention. The drug was well tolerated.


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