Method of assessing index profile data

1978 ◽  
Vol 14 (14) ◽  
pp. 416 ◽  
Author(s):  
J.P. Hazan ◽  
J.P. Cabanie ◽  
J.J. Bernard
Keyword(s):  
Profile Data ◽  
Index Profile

Archive of digital chirp subbottom profile data collected during USGS cruise 10BIM04 offshore Cat Island, Mississippi, September 2010

Data Series ◽  
10.3133/ds724 ◽  
2012 ◽  
Author(s):  
Arnell S. Forde ◽  
Shawn V. Dadisman ◽  
Jack G. Kindinger ◽  
Jennifer L. Miselis ◽  
Dana S. Wiese ◽  
...  
Keyword(s):  
Profile Data ◽  
Subbottom Profile

Archive of digital Chirp subbottom profile data collected during USGS cruises 10CCT01, 10CCT02, and 10CCT03, Mississippi and Alabama Gulf Islands, March and April 2010

Data Series ◽  
10.3133/ds611 ◽  
2011 ◽  
Author(s):  
Arnell S. Forde ◽  
Shawn V. Dadisman ◽  
James G. Flocks ◽  
Dana S. Wiese ◽  
Nancy T. DeWitt ◽  
...  
Keyword(s):  
Profile Data ◽  
Subbottom Profile ◽  
Gulf Islands

scholarly journals Neuroimmune Response Mediated by Cytokines in Natural Scrapie after Chronic Dexamethasone Treatment

Biomolecules ◽  
2021 ◽  
Vol 11 (2) ◽  
pp. 204
Author(s):  
Isabel M. Guijarro ◽  
Moisés Garcés ◽  
Pol Andrés-Benito ◽  
Belén Marín ◽  
Alicia Otero ◽  
...  
Keyword(s):  
Prion Diseases ◽  
Global Approach ◽  
Profile Data ◽  
Dexamethasone Treatment ◽  
New Approach ◽  
Complex Interactions ◽  
Natural Scrapie ◽  
Anti Inflammatory

The actual role of prion protein-induced glial activation and subsequent cytokine secretion during prion diseases is still incompletely understood. The overall aim of this study is to assess the effect of an anti-inflammatory treatment with dexamethasone on different cytokines released by neuroglial cells that are potentially related to neuroinflammation in natural scrapie. This study emphasizes the complex interactions existent among several pleiotropic neuromodulator peptides and provides a global approach to clarify neuroinflammatory processes in prion diseases. Additionally, an impairment of communication between microglial and astroglial populations mediated by cytokines, mainly IL-1, is suggested. The main novelty of this study is that it is the first one assessing in situ neuroinflammatory activity in relation to chronic anti-inflammatory therapy, gaining relevance because it is based on a natural model. The cytokine profile data would suggest the activation of some neurotoxicity-associated route. Consequently, targeting such a pathway might be a new approach to modify the damaging effects of neuroinflammation.

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