Natriuresis, Natriuretic Hormone Response to Water Immersion in Normotensives - Relation to Arterial Blood Pressure Level

1987 ◽  
Vol 72 (s16) ◽  
pp. 41P-41P
Author(s):  
Alice V. Stanton ◽  
Martin J. O'Hara ◽  
John B. Moynihan
2014 ◽  
Vol 95 (3) ◽  
pp. 472-474
Author(s):  
P I Petrov

Aim. To determine the association between the systolic arterial blood pressure level and the inter-arm asymmetry measurements in healthy individuals. Methods. 580 healthy volunteers aged 20 to 60 years (401 females, 179 males) were included in the study. Arterial blood pressure was measured in both left and right wrists using an automatic blood pressure sensor with the detector of hand position in a quiet, comfortable environment. Depending on the systolic arterial blood pressure level on the left arm, subjects were distributed to 5 groups: the 1st - blood pressure of 99 mm Hg and lower; 2nd - 100-119; 3rd - 120-129; 4th - 130-139; 5th -140 and over. Each group were further subdivided to 3 subgroups depending on the inter-arm blood pressure level asymmetry (difference of blood pressure levels on both arms): 1st - symmetry, 2nd - acceptable asymmetry (not exceeding 15 mm Hg), 3rd - abnormal asymmetry (16 mm Hg and over). Results. The major part of the examined subjects had systolic arterial blood pressure level between 100 and 119 mm Hg, systolic blood pressure below 99 mm Hg was registered more rarely. Among 580 examined subjects, abnormal asymmetry was more frequently seen in patients with blood pressure below 99 mm Hg and exceeding 140 mm Hg, the lowest frewuency was in the group with blood pressure between 120 and 129 mm Hg. The right arm was the dominating arm for the asymmetry in subjects with blood pressure below 119 mm Hg, in other cases the dominating arm was not determined. Conclusion. The study demonstrates the presence of vascular inter-arm asymmetry in systolic blood pressure in individuals who perceive themselves as healthy. Inter-arm asymmetry depended on the level of systolic arterial blood pressure.


1984 ◽  
Vol 6 (1) ◽  
pp. 33-39 ◽  
Author(s):  
G. Vachtsevanos ◽  
C. Kalaitzakis ◽  
N. Papamarkos ◽  
G. Ziakas ◽  
K. Economou ◽  
...  

1998 ◽  
Vol 275 (1) ◽  
pp. H139-H144 ◽  
Author(s):  
Olivier Régrigny ◽  
Philippe Delagrange ◽  
Elizabeth Scalbert ◽  
Jeffrey Atkinson ◽  
Isabelle Lartaud-Idjouadiene

Because melatonin is a cerebral vasoconstrictor agent, we tested whether it could shift the lower limit of cerebral blood flow autoregulation to a lower pressure level, by improving the cerebrovascular dilatory reserve, and thus widen the security margin. Cerebral blood flow and cerebrovascular resistance were measured by hydrogen clearance in the frontal cortex of adult male Wistar rats. The cerebrovasodilatory reserve was evaluated from the increase in the cerebral blood flow under hypercapnia. The lower limit of cerebral blood flow autoregulation was evaluated from the fall in cerebral blood flow following hypotensive hemorrhage. Rats received melatonin infusions of 60, 600, or 60,000 ng ⋅ kg−1 ⋅ h−1, a vehicle infusion, or no infusion ( n= 9 rats per group). Melatonin induced concentration-dependent cerebral vasoconstriction (up to 25% of the value for cerebrovascular resistance of the vehicle group). The increase in vasoconstrictor tone was accompanied by an improvement in the vasodilatory response to hypercapnia (+50 to +100% vs. vehicle) and by a shift in the lower limit of cerebral blood flow autoregulation to a lower mean arterial blood pressure level (from 90 to 50 mmHg). Because melatonin had no effect on baseline mean arterial blood pressure, the decrease in the lower limit of cerebral blood flow autoregulation led to an improvement in the cerebrovascular security margin (from 17% in vehicle to 30, 55, and 55% in the low-, medium-, and high-dose melatonin groups, respectively). This improvement in the security margin suggests that melatonin could play an important role in the regulation of cerebral blood flow and may diminish the risk of hypoperfusion-induced cerebral ischemia.


1978 ◽  
Vol 55 (s4) ◽  
pp. 61s-63s ◽  
Author(s):  
T. Philipp ◽  
A. Distler ◽  
U. Cordes ◽  
H. P. Wolff

1. An inverse relationship was found between plasma noradrenaline and reactivity to exogenous noradrenaline in normotensive subjects. 2. The relationship between plasma noradrenaline and reactivity was disturbed in age-matched patients with essential hypertension. 3. A multiple-regression analysis showed a highly significant correlation between adrenergic activity and reactivity to noradrenaline and the mean arterial blood pressure level (r = 0·91). The results suggest that adrenergic activity and pressor response to noradrenaline combined are important determinants of arterial blood pressure. 4. An inverse relationship could also be demonstrated between plasma renin activity and reactivity to exogenous angiotensin II. No difference was observed between normotensive and hypertensive subjects.


1979 ◽  
Vol 237 (2) ◽  
pp. H213-H217
Author(s):  
G. Baccelli ◽  
G. Mancia ◽  
A. Del Bo ◽  
R. Albertini ◽  
A. Zanchetti

The hemodynamic changes occurring during spontaneous micturition were recorded in conscious cats. Arterial blood pressure was continuously measured by chronically implanted arterial catheter, heart rate (HR) by a cardiotachometer, and cardiac output (CO), superior mesenteric (MF), renal (RF), and external iliac blood flows (IF) by chronically implanted electromagnetic flow probes. Spontaneous micturition was accompanied by little change in mean arterial pressure (-9.7 +/- 0.7%), but by a marked decrease in HR (-49.0 +/- 1.2%) and CO (-28.6 +/- 2.5%), and therefore by a marked decrease in total peripheral conductance (-21.0 +/- 3.5%). Visceral and hindlimb blood flows were markedly reduced during micturition (MF, -34.7 +/- 2.1%; RF, -22.6 +/- 1.5%; and IF, -48.7 +/- 1.5%, respectively) due to a marked reduction in regional conductances in both these areas. The vasomotor changes in the regional circulations were prevented by local sympathectomy. Thus spontaneous micturition is associated with marked changes in cardiac function and systemic circulation. Cardiac output is decreased, but diffuse nervous systemic vasoconstriction compensates for this and provides maintenance of arterial blood pressure level.


1973 ◽  
Vol 45 (s1) ◽  
pp. 145s-149s ◽  
Author(s):  
R. Sannerstedt ◽  
H. Wasir ◽  
R. Henning ◽  
L. Werkö

1. Five men with borderline, latent arterial hypertension of the hyperkinetic type were studied haemodynamically at rest and during dynamic exercise before and after a 6-week period of supervised physical training. 2. Tendencies to lower heart rate, cardiac output and arterial blood pressure, both at rest and during a standardized work-load, were observed after the training period, with significant differences between paired observations for the heart rate and mean arterial blood pressure during exercise. The systemic vascular resistance being unchanged, there was a certain trend to an increased widening of the arteriovenous oxygen difference, both at rest and during exercise. 3. The present findings from a small number of subjects indicate that physical conditioning of patients with latent hypertension of the hyperkinetic type contributes to a normalization of their circulation toward a normokinetic one, thereby also normalizing their blood pressure level and achieving a more economic energy expenditure in the cardiovascular system.


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