The Effect of Caloric Intake on Nitrogen Balance in Chronic Renal Failure

1972 ◽  
Vol 43 (5) ◽  
pp. 679-688 ◽  
Author(s):  
B. E. B. Hyne ◽  
Edna Fowell ◽  
H. A. Lee
Keyword(s):  
Renal Failure ◽  
Body Weight ◽  
Chronic Renal Failure ◽  
Nitrogen Balance ◽  
Caloric Intake

1. Nitrogen balance in uraemic patients on similar nitrogen intakes improves with increasing caloric intake in the range of 36–55 cal/kg body weight. 2. The degree of improvement in nitrogen balance is compatible with increased utilization of endogenous nitrogen probably as a result of increased dietary calories.

scholarly journals Nitrogen Balance in Patients with Chronic Renal Failure on Diets Containing Varying Quantities of Protein

BMJ ◽  
1969 ◽  
Vol 1 (5646) ◽  
pp. 735-740 ◽  
Author(s):  
J. Ford ◽  
M. E. Phillips ◽  
F. E. Toye ◽  
V. A. Luck ◽  
H. E. de Wardener
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Nitrogen Balance

UTILIZATION OF PARENTERALLY ADMINISTERED NITROGEN BY INFANTS RECEIVING SUBOPTIMAL CALORIES

PEDIATRICS ◽  
1959 ◽  
Vol 23 (4) ◽  
pp. 690-698
Author(s):  
Philip L. Calcagno ◽  
Mitchell I. Rubin ◽  
P. K. Mukherji
Keyword(s):  
Amino Acids ◽  
Body Weight ◽  
Amino Acid ◽  
Nitrogen Balance ◽  
Protein Hydrolysate ◽  
Caloric Intake ◽  
Parenteral Administration ◽  
Amino Acid Nitrogen ◽  
Negative Balance ◽  
Protein Starvation

The parenteral administration of 100 mg of amino-acid nitrogen/kg of body weight/ day given within 4 hours to infants who are underfed (40 cal/kg of body weight/day) and protein-starved can change the nitrogen balance from negative to positive or greatly reduce the degree of negative balance. The retained nitrogen is not degraded to urea or excreted as amino acids in the urine in the after-period. It is postulated that the retained amino-acid nitrogen is utilized for synthesis of tissue. Even when the total caloric intake is suboptimal, the administration of a protein hydrolysate during prolonged protein starvation in infants who are unable to ingest protein seems desirable.

The Nephroprotective Effects of the Herbal Medicine Preparation, WH30+, on the Chemical-Induced Acute and Chronic Renal Failure in Rats

2005 ◽  
Vol 33 (03) ◽  
pp. 491-500 ◽  
Author(s):  
Heidi H. Y. Ngai ◽  
Wai-Hung Sit ◽  
Jennifer M. F. Wan
Keyword(s):  
Renal Failure ◽  
Body Weight ◽  
Acute Renal Failure ◽  
Chronic Renal Failure ◽  
Serum Creatinine ◽  
Salvia Miltiorrhiza ◽  
Chinese Herb ◽  
The Body ◽  
Cordyceps Sinensis ◽  
Urea Nitrogen

In this study, we evaluated the renal protective effects of a Chinese herbal preparation WH30+in male Wistar rats with glycerol-induced acute renal failure and adenine-induced chronic renal failure. WH30+is a Chinese herb preparation composed of Rheum Palmatum, Salvia Miltiorrhiza, Cordyceps Sinensis, Leonurus Sibiricus, Epihedium Macranthum, Radix Astragali, and Radix Codonopsis Pilosulae, which has been used to treat kidney deficiency in human. An acute renal failure and chronic renal failure rat model were introduced by glycerol injection (i.m.) and fed with adenine-excessive diet, respectively. WH30+was administered to rats at the dose of 50 mg/kg/day from 10 days before the diseases were induced until the rats were sacrificed. A reduction in body weight ( p < 0.01) was observed in rats with chronic renal failure, but there was no difference between treatment groups. However, the body weight of rats with acute renal failure without treatment was significantly lower than those treated with WH30+( p < 0.05). Overall, serum creatinine and urea nitrogen were elevated significantly ( p < 0.01) in renal failure rats compared to control. Treatment with WH30+improved both serum creatinine and urea nitrogen slightly in both models. The WH30+-treated rats with acute renal failure had significantly ( p < 0.05) greater creatinine clearance than those without treatment. The results of the study show that WH30+is more effective in the prevention of acute renal failure than chronic renal failure.

scholarly journals Korelasi Penambahan Berat Badan Diantara Dua Waktu Dialisis denganKualitas Hidup Pasien Menjalani Hemodialisa

2016 ◽  
Vol 2 (2) ◽  
pp. 51
Author(s):  
Wahyuni Wahyuni ◽  
Winda Irwanti ◽  
Sofyan Indrayana
Keyword(s):  
Quality Of Life ◽  
Renal Failure ◽  
Body Weight ◽  
Chronic Renal Failure ◽  
Sampling Technique ◽  
Correlation Study ◽  
P Value ◽  
Cross Sectional ◽  
Cross Sectional Design

<p>Hemodialysis is a static treatment to substitute kidney function. Without a substitute therapy for kidney, a death as a results of metabolic abnormality can occur rapidly. Common issues on clients undergoing hemodialysis are gaining body weight in between two times dialysis which can affect the quality of life of patients if it is not get the good treatment. This research aimed to identify corelations weight gain in between the two time of dialysis to the quality of life of patients with chronic renal failure who underwent hemodialysis in RSUD Panembahan Senopati Bantul. This was a descriptive correlation study with cross-sectional design, samples were taken with accidental sampling technique consisted of 61 people. Data were analized by Kendal Tau Test(τ). The results of the statistics kendal tau (τ) indicated its value pearson kendal tau (-0,009) with p value 0,938, the fi gures were larger than standard significance α: 0.05, thats mean the hypothesis of this research was rejected. Conclusion, there was no a signifi cant corelation weight gaining in between two time of dialysis to the quality of life of patients with chronic renal failure who underwent hemodialysis in RSUD Panembahan Senopati Bantul.</p>

Creatinine Metabolism in Chronic Renal Failure

1980 ◽  
Vol 58 (4) ◽  
pp. 327-335 ◽  
Author(s):  
W. E. Mitch ◽  
V. U. Collier ◽  
M. Walser
Keyword(s):  
Renal Failure ◽  
Body Weight ◽  
Chronic Renal Failure ◽  
Serum Creatinine ◽  
Renal Clearance ◽  
Average Rate ◽  
Specific Radioactivity ◽  
Normal Subjects ◽  
Volume Of Distribution ◽  
Metabolism Rate

1. Creatinine metabolism was studied in nine patients with severe chronic renal failure who were nevertheless in a nearly steady state with respect to their creatinine pool. Labelled creatinine was injected intravenously and the specific radioactivity of creatinine in urine was measured during the ensuing 5–7 days. 2. In each patient, the decline in specific radioactivity with time was a single exponential function after 12 h. The volume of distribution of creatinine averaged 49.1 ± 2.8% body weight. The average rate of creatinine production was 148 μmol day−1 kg−1, which is similar to predicted values for normal subjects of the same age, weight and sex. Creatinine metabolism rate/kg body weight, estimated as the difference between production rate/kg body weight, determined radioisotopically, and creatinine appearance rate (excretion plus accumulation), averaged 42 μmol day−1 kg−1. 3. Total creatinine metabolism rate/kg body weight was correlated with serum creatinine. Thus, as serum creatinine rises, an increasing fraction of the produced creatinine was metabolized rather than excreted. This relationship could account for the diminished creatinine excretion commonly seen in patients with chronic renal failure. 4. Extrarenal clearance (metabolism/serum creatinine) of this magnitude (approximately 31% of renal clearance in these patients) would be an undetectably small fraction of normal renal clearance. This could explain the absence of demonstrable creatinine metabolism in normal subjects. 5. Two pathways of metabolism were identified: a recycling of creatinine to creatine and an irreversible degradation of creatinine to products other than creatine.

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