Calcium and Ca-dependent phospholipid signalling systems are involved in spleen lymphocyte apoptosis

2000 ◽  
Vol 28 (5) ◽  
pp. A378-A378
Author(s):  
P. P. Matyshevska ◽  
S. I. Boryssov ◽  
V. N. Solodushko ◽  
M. E. Kucherenko
Keyword(s):  
Spleen Lymphocyte ◽  
Lymphocyte Apoptosis ◽  
Signalling Systems ◽  
Phospholipid Signalling

Moderate physical training attenuates perinatal low-protein-induced spleen lymphocyte apoptosis in endotoxemic adult offspring rats

2015 ◽  
Vol 55 (3) ◽  
pp. 1113-1122 ◽  
Author(s):  
Sueli Moreno Senna ◽  
Marília Kalinne Torres ◽  
Daíllo Augusto Pereira Lopes ◽  
Maria Claudia Alheiros-Lira ◽  
Diógenis Barbosa de Moura ◽  
...  
Keyword(s):  
Physical Training ◽  
Adult Offspring ◽  
Spleen Lymphocyte ◽  
Lymphocyte Apoptosis ◽  
Low Protein

P-59: Insulin-sensitive phospholipid signalling systems and their relation to glucose transport

2009 ◽  
Vol 104 (S 02) ◽  
pp. 125-126
Author(s):  
Mary L. Standaert ◽  
Purushotham Karnam ◽  
Gautam Bandyopadhyay ◽  
Lamar Galloway ◽  
Robert V. Farese
Keyword(s):  
Glucose Transport ◽  
Signalling Systems ◽  
Phospholipid Signalling

scholarly journals Vocal plasticity in a reptile

2017 ◽  
Vol 284 (1855) ◽  
pp. 20170451 ◽  
Author(s):  
Henrik Brumm ◽  
Sue Anne Zollinger
Keyword(s):  
Signal Detection ◽  
Communication Systems ◽  
Vocal Communication ◽  
Animal Communication ◽  
High Amplitude ◽  
Vocal Plasticity ◽  
Lombard Effect ◽  
Gekko Gecko ◽  
High Degree ◽  
Signalling Systems

Sophisticated vocal communication systems of birds and mammals, including human speech, are characterized by a high degree of plasticity in which signals are individually adjusted in response to changes in the environment. Here, we present, to our knowledge, the first evidence for vocal plasticity in a reptile. Like birds and mammals, tokay geckos ( Gekko gecko ) increased the duration of brief call notes in the presence of broadcast noise compared to quiet conditions, a behaviour that facilitates signal detection by receivers. By contrast, they did not adjust the amplitudes of their call syllables in noise (the Lombard effect), which is in line with the hypothesis that the Lombard effect has evolved independently in birds and mammals. However, the geckos used a different strategy to increase signal-to-noise ratios: instead of increasing the amplitude of a given call type when exposed to noise, the subjects produced more high-amplitude syllable types from their repertoire. Our findings demonstrate that reptile vocalizations are much more flexible than previously thought, including elaborate vocal plasticity that is also important for the complex signalling systems of birds and mammals. We suggest that signal detection constraints are one of the major forces driving the evolution of animal communication systems across different taxa.

scholarly journals Awakening the oocyte: controlling primordial follicle development

Reproduction ◽  
2009 ◽  
Vol 137 (1) ◽  
pp. 1-11 ◽  
Author(s):  
Eileen A McLaughlin ◽  
Skye C McIver
Keyword(s):  
Population Control ◽  
Reproductive Potential ◽  
Animal Husbandry ◽  
Primordial Follicle ◽  
Domestic Animal ◽  
Follicle Growth ◽  
Cellular Mechanisms ◽  
Primordial Follicles ◽  
Control Oocyte ◽  
Signalling Systems

Oocytes are sequestered in primordial follicles before birth and remain quiescent in the ovary, often for decades, until recruited into the growing pool throughout the reproductive years. Therefore, activation of follicle growth is a major biological checkpoint that controls female reproductive potential. However, we are only just beginning to elucidate the cellular mechanisms required for either maintenance of the quiescent primordial follicle pool or initiation of follicle growth. Understanding the intracellular signalling systems that control oocyte maintenance and activation has significant implications for improving female reproductive productivity and longevity in mammals, and has application in domestic animal husbandry, feral animal population control and infertility in women.

scholarly journals Stress hormones collaborate to induce lymphocyte apoptosis after high level spinal cord injury

2009 ◽  
Vol 110 (5) ◽  
pp. 1409-1421 ◽  
Author(s):  
Kurt M. Lucin ◽  
Virginia M. Sanders ◽  
Phillip G. Popovich
Keyword(s):  
Spinal Cord ◽  
Spinal Cord Injury ◽  
Stress Hormones ◽  
Lymphocyte Apoptosis ◽  
Cord Injury ◽  
High Level
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