scholarly journals Carbocatalytic ozonation toward advanced water purification

2021 ◽  
Author(s):  
Ya Liu ◽  
Chunmao Chen ◽  
Xiaoguang Duan ◽  
Shaobin Wang ◽  
Yuxian Wang
Keyword(s):  
Reactive Oxygen Species ◽  
Water Purification ◽  
State Of The Art ◽  
Reactive Oxygen ◽  
Catalytic Ozonation ◽  
Oxygen Species ◽  
Wastewater Remediation ◽  
Ozone Oxidation ◽  
High Oxidation ◽  
Carbon Based

Carbon-based catalytic ozonation is state-of-the-art technology with high oxidation capabilities for wastewater remediation, taking advantage of the synergies of direct ozone oxidation and the generated reactive oxygen species (ROS). Replacing...

Toxicological Mechanisms of Carbon-Based Nanomaterials

2011 ◽  
Vol 345 ◽  
pp. 12-17
Author(s):  
Han Jiang ◽  
Xue Feng Wang ◽  
Zi Hao Wang ◽  
Jing Ding ◽  
Wei Bin Shi
Keyword(s):  
Reactive Oxygen Species ◽  
Materials Science ◽  
Quantum Effect ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Nano Materials ◽  
Carbon Based Nanomaterials ◽  
The Impact ◽  
Carbon Based

Abstact:With the development of materials science and combination with medicine, more and more nano-materials are used in clinical medicine.There are much advantage in nano-material .The reason that the nano-materials are utilized widely is its quantum effect and huge superficalarea.But with further research ,more and more disadvantages are found. Many research on the environmental and human health risks of engineered nano-materials focus on their acute toxicity. However,the long-term chronic effects of nanomaterials on living systems is neglected.The potential of nanomaterials to promote the formation of reactive oxygen species is one of the primary reason in their genotoxic. DNA would be damaged by the reactive oxygen species. If the DNA cannot be renew ,it could lead to gene mutation , canceration. This review focuses on the impact of carbon-based nano-materials on DNA .

scholarly journals Prodrug strategies for targeted therapy triggered by reactive oxygen species

MedChemComm ◽  
2019 ◽  
Vol 10 (9) ◽  
pp. 1531-1549 ◽  
Author(s):  
Jorge Peiró Cadahía ◽  
Viola Previtali ◽  
Nikolaj S. Troelsen ◽  
Mads H. Clausen
Keyword(s):  
Reactive Oxygen Species ◽  
Targeted Therapy ◽  
State Of The Art ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Future Perspectives ◽  
Comprehensive Review

A comprehensive review of ROS-activated produg strategies for targeted therapy, including state-of-the-art and future perspectives.

Mitochondria as a source of reactive oxygen species

2009 ◽  
pp. c3 ◽  
Author(s):  
Helena M. Cochemé ◽  
Michael P. Murphy
Keyword(s):  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Oxygen Species

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

Fas ligand expression in rat kupffer cells in response to lipopolysaccharide is mediated by reactive oxygen species

2001 ◽  
Vol 120 (5) ◽  
pp. A361-A361
Author(s):  
K UCHIKURA ◽  
T WADA ◽  
Z SUN ◽  
S HOSHINO ◽  
G BULKLEY ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Kupffer Cells ◽  
Fas Ligand ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Ligand Expression
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