DNA introduces an independent temperature responsiveness to thermosensitive microgels and enables switchable plasmon coupling as well as controlled uptake and release

Nanoscale ◽  
2021 ◽  
Author(s):  
Sabine Eisold ◽  
Laura Hoppe Alvarez ◽  
Ke Ran ◽  
Rebecca Hengsbach ◽  
Gerhard Fink ◽  
...  

DNA-microgel hybrid systems with dual thermal responsiveness are suited for programmed and reversible uptake and release of molecular and nanoparticulate guest compounds in biological environments.

2016 ◽  
Vol 2016 ◽  
pp. 1-21 ◽  
Author(s):  
Mohamed El Kabbash ◽  
Alireza Rahimi Rashed ◽  
Kandammathe Valiyaveedu Sreekanth ◽  
Antonio De Luca ◽  
Melissa Infusino ◽  
...  

The presence of an excitonic element in close proximity of a plasmonic nanostructure, under certain conditions, may lead to a nonradiative resonant energy transfer known as Exciton Plasmon Resonant Energy Transfer (EPRET) process. The exciton-plasmon coupling and dynamics have been intensely studied in the last decade; still many relevant aspects need more in-depth studies. Understanding such phenomenon is not only important from fundamental viewpoint, but also essential to unlock many promising applications. In this review we investigate the plasmon-exciton resonant energy transfer in different hybrid systems at the nano- and mesoscales, in order to gain further understanding of such processes across scales and pave the way towards active plasmonic devices.


1980 ◽  
Vol 44 (03) ◽  
pp. 143-145 ◽  
Author(s):  
J Dalsgaard-Nielsen ◽  
J Gormsen

SummaryHuman platelets in platelet rich plasma (PRP) incubated at 37° C with 0.3–2% halothane for 5–10 min lost the ability to aggregate with ADP, epinephrine and collagen.At the same time uptake and release of 14C-serotonin was inhibited. When halothane supply was removed, platelet functions rapidly returned to normal. However, after high concentrations of halothane, the inhibition of platelet aggregation was irreversible or only partially reversible.The results suggest that halothane anaesthesia produces a transient impairment of platelet function.


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