Reply to comment on “Brownian diffusion of a particle at an air/liquid interface: elastic (not viscous) response of the surface”

2017 ◽  
Vol 19 (33) ◽  
pp. 22594-22595
Author(s):  
Jhoan Toro-Mendoza ◽  
Gieberth Rodriguez-Lopez ◽  
Oscar Paredes-Altuve
Keyword(s):  
Particle Dynamics ◽  
Liquid Interface ◽  
Brownian Diffusion ◽  
Air Liquid Interface

In a Comment on our article “Brownian diffusion of a particle at an air/liquid interface: Elastic (not viscous) response of the surface”, Boniello et al. pointed out their disagreement with our argument that the elastic drag of the surface affects the particle dynamics.

Brownian diffusion of a particle at an air/liquid interface: the elastic (not viscous) response of the surface

2017 ◽  
Vol 19 (13) ◽  
pp. 9092-9095 ◽  
Author(s):  
Jhoan Toro-Mendoza ◽  
Gieberth Rodriguez-Lopez ◽  
Oscar Paredes-Altuve
Keyword(s):  
Diffusion Coefficient ◽  
Brownian Particle ◽  
Translational Diffusion ◽  
Liquid Interface ◽  
Elastic Response ◽  
Brownian Diffusion ◽  
Translational Diffusion Coefficient ◽  
Air Liquid Interface

Here, the effect of the elastic response of the surface on the translational diffusion coefficient of a partly submerged-in-water spherical Brownian particle is considered.

Comment on “Brownian diffusion of a particle at an air/liquid interface: elastic (not viscous) response of the surface”

2017 ◽  
Vol 19 (33) ◽  
pp. 22592-22593 ◽  
Author(s):  
G. Boniello ◽  
A. Stocco ◽  
C. Blanc ◽  
M. Nobili
Keyword(s):  
Recent Article ◽  
Liquid Interface ◽  
Solid Particles ◽  
Elastic Response ◽  
Brownian Diffusion ◽  
Fluid Interfaces ◽  
Air Liquid Interface

In a recent article Toro-Mendoza et al. considered an elastic response of an interface in order to explain the enhanced lateral drag of solid particles straddling fluid interfaces we recently measured.

Rapid, efficient and dose-controlled delivery of liquid aerosol to pulmonary cells cultured at the air-liquid interface

Pneumologie ◽  
2011 ◽  
Vol 65 (12) ◽  
Author(s):  
M Selmansberger ◽  
AG Lenz ◽  
M Schmidmeir ◽  
O Eickelberg ◽  
T Stoeger ◽  
...  
Keyword(s):  
Liquid Interface ◽  
Controlled Delivery ◽  
Pulmonary Cells ◽  
Air Liquid Interface ◽  
Cells Cultured

Zwischen Luft und Wasser – Air-Liquid-Interface-Kulturen als In-vitro-Modell des Respirationstrakts

2020 ◽  
Author(s):  
S. Runft ◽  
L. Burigk ◽  
A. Lehmbecker ◽  
K. Schöne ◽  
D. Waschke ◽  
...  
Keyword(s):  
Liquid Interface ◽  
Air Liquid Interface

scholarly journals Drag on a spherical particle at the air–liquid interface: Interplay between compressibility, Marangoni flow, and surface viscosities

2021 ◽  
Vol 33 (6) ◽  
pp. 062103
Author(s):  
Meisam Pourali ◽  
Martin Kröger ◽  
Jan Vermant ◽  
Patrick D. Anderson ◽  
Nick O. Jaensson
Keyword(s):  
Spherical Particle ◽  
Liquid Interface ◽  
Marangoni Flow ◽  
Air Liquid Interface

Nonclassical Surface Nucleation of 6CB at the Air–Liquid Interface of a 6CB Oil-in-Water Nanoemulsion

Langmuir ◽  
2021 ◽  
Author(s):  
Jin Tae Park ◽  
Govind Paneru ◽  
Masao Iwamatsu ◽  
Bruce M. Law ◽  
Hyuk Kyu Pak
Keyword(s):  
Liquid Interface ◽  
Surface Nucleation ◽  
Oil In Water ◽  
Air Liquid Interface

scholarly journals Effect of Alpha-1 Antitrypsin on CFTR Levels in Primary Human Airway Epithelial Cells Grown at the Air-Liquid-Interface

Molecules ◽  
2021 ◽  
Vol 26 (9) ◽  
pp. 2639
Author(s):  
Frauke Stanke ◽  
Sabina Janciauskiene ◽  
Stephanie Tamm ◽  
Sabine Wrenger ◽  
Ellen Luise Raddatz ◽  
...  
Keyword(s):  
Epithelial Cells ◽  
Human Lung ◽  
Liquid Interface ◽  
Lung Epithelial Cells ◽  
Western Blots ◽  
Mesenchymal Transition ◽  
Protein Levels ◽  
Lung Epithelial ◽  
Cftr Protein ◽  
Air Liquid Interface

The cystic fibrosis transmembrane conductance regulator (CFTR) gene is influenced by the fundamental cellular processes like epithelial differentiation/polarization, regeneration and epithelial–mesenchymal transition. Defects in CFTR protein levels and/or function lead to decreased airway surface liquid layer facilitating microbial colonization and inflammation. The SERPINA1 gene, encoding alpha1-antitrypsin (AAT) protein, is one of the genes implicated in CF, however it remains unknown whether AAT has any influence on CFTR levels. In this study we assessed CFTR protein levels in primary human lung epithelial cells grown at the air-liquid-interface (ALI) alone or pre-incubated with AAT by Western blots and immunohistochemistry. Histological analysis of ALI inserts revealed CFTR- and AAT-positive cells but no AAT-CFTR co-localization. When 0.5 mg/mL of AAT was added to apical or basolateral compartments of pro-inflammatory activated ALI cultures, CFTR levels increased relative to activated ALIs. This finding suggests that AAT is CFTR-modulating protein, albeit its effects may depend on the concentration and the route of administration. Human lung epithelial ALI cultures provide a useful tool for studies in detail how AAT or other pharmaceuticals affect the levels and activity of CFTR.

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