scholarly journals Investigations on HONO formation from photolysis of adsorbed HNO3 on quartz glass surfaces

2016 ◽  
Vol 18 (14) ◽  
pp. 9616-9625 ◽  
Author(s):  
Sebastian Laufs ◽  
Jörg Kleffmann

HONO formation by photolysis of HNO3 on clean surfaces is no significant source of HONO and NOx in the atmosphere.

2011 ◽  
Vol 12 ◽  
pp. 452-461 ◽  
Author(s):  
Jörg Hildebrand ◽  
Kerstin Hecht ◽  
Jens Bliedtner ◽  
Hartmut Müller

2018 ◽  
Vol 20 (48) ◽  
pp. 30537-30539 ◽  
Author(s):  
Michael N. Sullivan ◽  
Liang T. Chu ◽  
Lei Zhu

Determination of HNO3 coverage on silica provides insight into different HNO3 surface photolysis rates without and with humidity.


2012 ◽  
Vol 39 ◽  
pp. 277-285 ◽  
Author(s):  
Jörg Hildebrand ◽  
Kerstin Hecht ◽  
Jens Bliedtner ◽  
Hartmut Müller

Fuel ◽  
1995 ◽  
Vol 74 (3) ◽  
pp. 452-455 ◽  
Author(s):  
Andreas Berger ◽  
Gerd Rotzoll

2020 ◽  
Vol 40 (23) ◽  
pp. 2314001
Author(s):  
陈亮 Chen Liang ◽  
刘晓东 Liu Xiaodong ◽  
刘静 Liu Jing ◽  
熊政军 Xiong Zhengjun

1984 ◽  
Vol 51 (03) ◽  
pp. 371-375 ◽  
Author(s):  
Kangathevy Morgan ◽  
Sandra Schiffman ◽  
Donald Feinstein

SummaryTwo patients with hereditary factor XI deficiency developed inhibitors following plasma transfusions. Neither had severe spontaneous bleeding. The patients’ plasmas neutralized both factor XI in plasma, purified factor XI, and purified factor XIa. The inhibitor in both patients’ plasmas adsorbed to Protein A- Sepharose. The inhibitors eluted from Protein A-Sepharose were partially neutralized by kappa and lambda light chain antisera indicating that they were polyclonal IgG antibodies. Both inhibitors markedly decreased adsorption of factor XI to glass surfaces. The cleavage of factor XI by trypsin was unaffected by the inhibitors. The lack of severe spontaneous bleeding in both of these patients strongly suggests that an alternate coagulation mechanism bypassing factor XI must compensate for this severe defect.


Sign in / Sign up

Export Citation Format

Share Document