Visualization of the interaction between Gβγ and tubulin during light-induced cell elongation of Blepharisma japonicum

2010 ◽  
Vol 9 (8) ◽  
pp. 1101 ◽  
Author(s):  
Katarzyna Sobierajska ◽  
Joanna Głos ◽  
Joanna Dąborowska ◽  
Joanna Kucharska ◽  
Cezary Bregier ◽  
...  
Keyword(s):  
Cell Elongation ◽  
Blepharisma Japonicum

Studies on the mechanism of cell elongation in Blepharisma japonicum

1991 ◽  
Vol 26 (3-4) ◽  
pp. 365-369 ◽  
Author(s):  
Masaki Ishida ◽  
Yoshinobu Shigenaka ◽  
Toshinobu Suzaki ◽  
Koji Taneda
Keyword(s):  
Cell Elongation ◽  
Blepharisma Japonicum

Studies on the mechanism of cell elongation in Blepharisma japonicum

1991 ◽  
Vol 27 (1) ◽  
pp. 46-54 ◽  
Author(s):  
Masaki Ishida ◽  
Toshinobu Suzaki ◽  
Yoshinobu Shigenaka
Keyword(s):  
Cell Elongation ◽  
Blepharisma Japonicum

scholarly journals Slm9, a Novel Nuclear Protein Involved in Mitotic Control in Fission Yeast

Genetics ◽  
2000 ◽  
Vol 155 (2) ◽  
pp. 623-631
Author(s):  
Junko Kanoh ◽  
Paul Russell
Keyword(s):  
Cell Cycle ◽  
Fission Yeast ◽  
Cell Elongation ◽  
Nuclear Protein ◽  
G1 Arrest ◽  
Permissive Temperature ◽  
Cdc2 Kinase ◽  
Synthetic Lethal ◽  
Cycle Arrest ◽  
Novel Protein

Abstract In the fission yeast Schizosaccharomyces pombe, as in other eukaryotic cells, Cdc2/cyclin B complex is the key regulator of mitosis. Perhaps the most important regulation of Cdc2 is the inhibitory phosphorylation of tyrosine-15 that is catalyzed by Wee1 and Mik1. Cdc25 and Pyp3 phosphatases dephosphorylate tyrosine-15 and activate Cdc2. To isolate novel activators of Cdc2 kinase, we screened synthetic lethal mutants in a cdc25-22 background at the permissive temperature (25°). One of the genes, slm9, encodes a novel protein of 807 amino acids. Slm9 is most similar to Hir2, the histone gene regulator in budding yeast. Slm9 protein level is constant and Slm9 is localized to the nucleus throughout the cell cycle. The slm9 disruptant is delayed at the G2-M transition as indicated by cell elongation and analysis of DNA content. Inactivation of Wee1 fully suppressed the cell elongation phenotype caused by the slm9 mutation. The slm9 mutant is defective in recovery from G1 arrest after nitrogen starvation. The slm9 mutant is also UV sensitive, showing a defect in recovery from the cell cycle arrest after UV irradiation.

Sign in / Sign up

Export Citation Format

Share Document