Substituent control of potassium and rubidium uptake by asymmetric calix[4]-thiacalix[4]tubes

2006 ◽  
Vol 4 (8) ◽  
pp. 1555 ◽  
Author(s):  
Elena Khomich ◽  
Marat Kashapov ◽  
Ivan Vatsouro ◽  
Elvira Shokova ◽  
Vladimir Kovalev
Keyword(s):  
1973 ◽  
Vol 24 (6) ◽  
pp. 1046-1052 ◽  
Author(s):  
G. A. ROBERTSON ◽  
B. C. LOUGHMAN

1962 ◽  
Vol 203 (6) ◽  
pp. 1191-1192 ◽  
Author(s):  
Jerold M. Lowenstein

The avidity of red cells for Rb86, a tracer of potassium in biological systems, was measured before, during, and after pregnancy. There was a gradual rise in rubidium uptake throughout pregnancy. The average uptake at 8 months' gestation was 50% higher than control levels; 6 weeks postpartum the uptake had decreased, but not to the control value.


2005 ◽  
Vol 28 (7) ◽  
pp. 850-862 ◽  
Author(s):  
AUGUSTO J. VALLEJO ◽  
MARIA LUISA PERALTA ◽  
GUILLERMO E. SANTA-MARIA

1985 ◽  
Vol 130 (3) ◽  
pp. 1066-1071 ◽  
Author(s):  
Natividad Hernando ◽  
Carlos Caramelo ◽  
Alberto Tejedor ◽  
Arturo Fernandez-Cruz ◽  
JoséM. López-Novoa

1979 ◽  
Vol 45 (1) ◽  
pp. 83-87 ◽  
Author(s):  
SUNE PETTERSSON ◽  
PAUL JENSEN
Keyword(s):  

1985 ◽  
Vol 69 (6) ◽  
pp. 737-743 ◽  
Author(s):  
Richard B. Wanless ◽  
Mark I. M. Noble ◽  
Angela J. D. Drake-Holland

1. The mechanism by which noradrenaline stimulates ouabain-sensitive rubidium uptake in guinea-pig myocardial tissue has been studied. 2. The stimulatory action of low concentrations of noradrenaline was reversed by high doses of propranolol (10−5 mol/l) in atrial tissue, but was not reversed by α- or β-, or combined α- and β-adrenoceptor antagonists in ventricular tissue. 3. Rubidium uptake was also found to increase with increasing extracellular potassium concentration ([K]o). The percentage values of stimulation by noradrenaline decreased with increasing [K]o. 4. Noradrenaline had no effect on the rate of ATP splitting by an isolated membrane preparation of Na,K-ATPase. 5. It is proposed that noradrenaline stimulates active cation transport by either (a) an effect secondary to increased passive efflux of K ions, or (b) an action at a novel adrenergic receptor, distinct from the ATPase enzyme itself.


Sign in / Sign up

Export Citation Format

Share Document