The determination of carbonyl compounds by semicarbazide and hydroxylamine. With special reference to fatty-acid oxidation products

The Analyst ◽  
1953 ◽  
Vol 78 (924) ◽  
pp. 135 ◽  
Author(s):  
A. J. Feuell ◽  
J. H. Skellon
Lipids ◽  
2020 ◽  
Vol 55 (6) ◽  
pp. 693-706
Author(s):  
Ho Hang Leung ◽  
Kin Sum Leung ◽  
Thierry Durand ◽  
Jean‐Marie Galano ◽  
Jetty Chung‐Yung Lee

1991 ◽  
Vol 279 (1) ◽  
pp. 147-150 ◽  
Author(s):  
R Rognstad

The pathways of peroxisomal and mitochondrial fatty acid oxidation were monitored with the use of substrates which produce NAD3H. I used as marker substrates: D-[3-3H]3-hydroxybutyrate for mitochondrial NAD3H production, [2-3H]glycerol for cytosolic NAD3H production, and [2-3H]acetate to measure carbon-bound 3H which was also generated by the metabolism of the commercial 9,10-3H-labelled fatty acids. The assumption that peroxisomal NAD3H can be considered to be equivalent to cytosolic NAD3H was supported using a specific inhibitor of mitochondrial fatty acid oxidation. The approach involves determination of the specific yields, and the relative distribution on carbons 4 and 6, of 3H in glucose from the marker substrates and the labelled fatty acids. In hepatocytes from clofibrate-treated rats, the amount of palmitate or oleate oxidation which starts in the peroxisomes is comparable with that which starts in the mitochondria.


1998 ◽  
Vol 265 (1409) ◽  
pp. 2027-2032 ◽  
Author(s):  
M. Regert ◽  
H. A. Bland ◽  
S. N. Dudd ◽  
P. F. V. Bergen ◽  
R. P. Evershed

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