scholarly journals Electrical stimulation enhances cell migration and integrative repair in the meniscus

2014 ◽  
Vol 4 (1) ◽  
Author(s):  
Xiaoning Yuan ◽  
Derya E. Arkonac ◽  
Pen-hsiu Grace Chao ◽  
Gordana Vunjak-Novakovic
Keyword(s):  
Cell Migration ◽  
Electrical Stimulation ◽  
Integrative Repair

Effect of Transcutaneous Electrical Stimulation on Rabbit Corneal Epithelial Cell Migration

Cornea ◽  
2012 ◽  
Vol 31 (5) ◽  
pp. 559-563 ◽  
Author(s):  
Alireza Ghaffarieh ◽  
Fariborz Ghaffarpasand ◽  
Maryam Dehghankhalili ◽  
Nazafarin Honarpisheh ◽  
Samira Nirumandi ◽  
...  
Keyword(s):  
Cell Migration ◽  
Electrical Stimulation ◽  
Epithelial Cell ◽  
Corneal Epithelial Cell ◽  
Transcutaneous Electrical Stimulation ◽  
Corneal Epithelial ◽  
Epithelial Cell Migration

Electrical stimulation of the motor cortex enhances progenitor cell migration in the adult rat brain

2013 ◽  
Vol 231 (2) ◽  
pp. 165-177 ◽  
Author(s):  
Ali Jahanshahi ◽  
Lisa Schonfeld ◽  
Marcus L. F. Janssen ◽  
Sarah Hescham ◽  
Ersoy Kocabicak ◽  
...  
Keyword(s):  
Cell Migration ◽  
Electrical Stimulation ◽  
Motor Cortex ◽  
Rat Brain ◽  
Progenitor Cell ◽  
Adult Rat ◽  
Adult Rat Brain ◽  
Stimulation Of

scholarly journals Finite Element Model for Cardiac Cell mechano-electrical stimulation

2020 ◽  
Vol 8 ◽  
Author(s):  
Pau Urdeitx Diaz ◽  
Mohamed H. Doweidar
Keyword(s):  
Finite Element ◽  
Cell Migration ◽  
Electrical Stimulation ◽  
Mechanical Stimulation ◽  
Collective Behavior ◽  
Element Model ◽  
Cardiac Cell ◽  
Alternating Electric Field ◽  
Low Stiffness ◽  
Electrical Stimuli

Cardiomyocyte behavior is highly dependent on the mechanical and electrical stimuli. We present a computational model, based on the FEM, to evaluate their behavior under electro-mechanical stimulation. Cell migration, adhesion, and collective behavior have been considered. Low stiffness and high alternating electric field have shown to be the best combination.

Activity-dependent neuronal cell migration induced by electrical stimulation

2008 ◽  
Vol 47 (1) ◽  
pp. 93-99 ◽  
Author(s):  
Se Hoon Jeong ◽  
Sang Beom Jun ◽  
Jong Keun Song ◽  
Sung June Kim
Keyword(s):  
Cell Migration ◽  
Electrical Stimulation ◽  
Neuronal Cell ◽  
Activity Dependent

scholarly journals Charge-Balanced Electrical Stimulation Can Modulate Neural Precursor Cell Migration in the Presence of Endogenous Electric Fields in Mouse Brains

eNeuro ◽  
2019 ◽  
Vol 6 (6) ◽  
pp. ENEURO.0382-19.2019 ◽  
Author(s):  
Stephanie N. Iwasa ◽  
Abdolazim Rashidi ◽  
Elana Sefton ◽  
Nancy X. Liu ◽  
Milos R. Popovic ◽  
...  
Keyword(s):  
Cell Migration ◽  
Electrical Stimulation ◽  
Electric Fields ◽  
Precursor Cell ◽  
Neural Precursor ◽  
Neural Precursor Cell

Neutrophil migration through in vitro interstitial matrix

1992 ◽  
Vol 50 (1) ◽  
pp. 894-895
Author(s):  
J. Roemer ◽  
S.R. Simon
Keyword(s):  
Extracellular Matrix ◽  
Smooth Muscle ◽  
Cell Migration ◽  
Smooth Muscle Cells ◽  
Inflammatory Cell ◽  
Neutrophil Migration ◽  
Culture Conditions ◽  
Aortic Smooth Muscle ◽  
Specific Culture

We are developing an in vitro interstitial extracellular matrix (ECM) system for study of inflammatory cell migration. Falcon brand Cyclopore membrane inserts of various pore sizes are used as a support substrate for production of ECM by R22 rat aortic smooth muscle cells. Under specific culture conditions these cells produce a highly insoluble matrix consisting of typical interstitial ECM components, i.e.: types I and III collagen, elastin, proteoglycans and fibronectin.

The “KIMWIPE” Effect in Ultra-Thin Cryosections

1985 ◽  
Vol 43 ◽  
pp. 458-459
Author(s):  
I. Taylor ◽  
P. Ingram ◽  
J.R. Sommer
Keyword(s):  
Skeletal Muscle ◽  
Electrical Stimulation ◽  
Muscle Fibers ◽  
Ice Crystals ◽  
Crystal Formation ◽  
Ice Crystal ◽  
Thin Sections ◽  
Skeletal Muscle Fibers ◽  
Freeze Dried ◽  
Ice Crystal Formation

In studying quick-frozen single intact skeletal muscle fibers for structural and microchemical alterations that occur milliseconds, and fractions thereof, after electrical stimulation, we have developed a method to compare, directly, ice crystal formation in freeze-substituted thin sections adjacent to all, and beneath the last, freeze-dried cryosections. We have observed images in the cryosections that to our knowledge have not been published heretofore (Figs.1-4). The main features are that isolated, sometimes large regions of the sections appear hazy and have much less contrast than adjacent regions. Sometimes within the hazy regions there are smaller areas that appear crinkled and have much more contrast. We have also observed that while the hazy areas remain still, the regions of higher contrast visibly contract in the beam, often causing tears in the sections that are clearly not caused by ice crystals (Fig.3, arrows).

The role of DUBs in the post-translational control of cell migration

2019 ◽  
Vol 63 (5) ◽  
pp. 579-594 ◽  
Author(s):  
Guillem Lambies ◽  
Antonio García de Herreros ◽  
Víctor M. Díaz
Keyword(s):  
Cell Migration ◽  
Translational Control ◽  
Epithelial To Mesenchymal Transition ◽  
Extracellular Matrix Remodeling ◽  
Matrix Remodeling ◽  
Mesenchymal Transition ◽  
Tgfβ Receptors ◽  
Fundamental Process ◽  
Multistep Process

Abstract Cell migration is a multifactorial/multistep process that requires the concerted action of growth and transcriptional factors, motor proteins, extracellular matrix remodeling and proteases. In this review, we focus on the role of transcription factors modulating Epithelial-to-Mesenchymal Transition (EMT-TFs), a fundamental process supporting both physiological and pathological cell migration. These EMT-TFs (Snail1/2, Twist1/2 and Zeb1/2) are labile proteins which should be stabilized to initiate EMT and provide full migratory and invasive properties. We present here a family of enzymes, the deubiquitinases (DUBs) which have a crucial role in counteracting polyubiquitination and proteasomal degradation of EMT-TFs after their induction by TGFβ, inflammatory cytokines and hypoxia. We also describe the DUBs promoting the stabilization of Smads, TGFβ receptors and other key proteins involved in transduction pathways controlling EMT.

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