scholarly journals Sex Difference in Heritability of BMI in South Korean Adolescent Twins**

Obesity ◽  
2007 ◽  
Vol 15 (12) ◽  
pp. 2908-2911 ◽  
Author(s):  
Yoon-Mi Hur
2019 ◽  
Vol 22 (2) ◽  
pp. 114-119 ◽  
Author(s):  
Yoon-Mi Hur ◽  
Hee-Jeong Jin ◽  
Siwoo Lee ◽  
Jong-Woo Kim

AbstractSomatization is known to be more prevalent in Asian than in Western populations. Using a South Korean adolescent and young adult twin sample (N = 1754; 367 monozygotic male, 173 dizygotic male, 681 monozygotic female, 274 dizygotic female and 259 opposite-sex dizygotic twins), the present study aimed to estimate heritability of somatization and to determine common genetic and environmental influences on somatization and hwabyung (HB: anger syndrome). Twins completed self-report questionnaires of the HB symptoms scale and the somatization scale via a telephone interview. The results of the general sex-limitation model showed that 43% (95% CI [36, 50]) of the total variance of somatization was attributable to additive genetic factors, with the remaining variance, 57% (95% CI [50, 64]), being due to individual-specific environmental influences, including measurement error. These estimates were not significantly different between the two sexes. The phenotypic correlation between HB and somatization was .53 (p < .001). The bivariate model-fitting analyses revealed that the genetic correlation between the two symptoms was .68 (95% CI [.59, .77]), while the individual-specific environmental correlation, including correlated measurement error, was .41 (95% CI [.34, .48]). Of the additive genetic factors of 43% that influence somatization, approximately half (20%) were associated with those related to HB, with the remainder being due to genes unique to somatization. A substantial part (48%) of individual environmental variance in somatization was unrelated to HB; only 9% of the environmental variance was shared with HB. Our findings suggest that HB and somatization have shared genetic etiology, but environmental factors that precipitate the development of HB and somatization may be largely independent from each other.


2013 ◽  
Vol 34 (6) ◽  
pp. 268-276 ◽  
Author(s):  
Jong-Hyo Park ◽  
Robert D. Enright ◽  
Marilyn J. Essex ◽  
Carolyn Zahn-Waxler ◽  
John S. Klatt

2006 ◽  
Vol 9 (5) ◽  
pp. 637-641 ◽  
Author(s):  
Yoon-Mi Hur

AbstractHostility has been shown to be a vulnerability marker for various health problems. The present study examined genetic and environmental contributions to individual differences in hostility in South Korean adolescent and young adult twins. Seven hundred and nineteen same- and opposite-sex twin pairs aged from 13 to 23 years completed a hostility scale. The scalar sex-limitation model was applied to the data. The best fitting model indicated that 34% of the total variation of hostility was attributable to genetic factors operating in a nonadditive manner. The remaining 66% of the variance was associated with nonshared environmental influences and measurement error. These findings were largely consistent with results from previous twin studies of personality based on Caucasian twins, rendering support for the pervasive influence of genetic non-additivity on human personality traits and the generalization of the heritability of personality across human populations.


2016 ◽  
Vol 48 (12) ◽  
pp. 2454-2461 ◽  
Author(s):  
EUN-YOUNG LEE ◽  
KIYONG AN ◽  
JUSTIN Y. JEON ◽  
WENDY M. RODGERS ◽  
VICKI J. HARBER ◽  
...  

2008 ◽  
Vol 11 (3) ◽  
pp. 306-313 ◽  
Author(s):  
Yoon-Mi Hur

AbstractIt has been reported that prevalence estimates, symptom presentation, and sociocultural risk and protective factors for depression differ between Caucasian and East Asian populations. But, nonetheless, as the vast majority of twin studies of depression symptoms have been carried out using Caucasians, genetic and environmental influences on depression symptoms in East Asians remain poorly understood. In the present study, the Center for Epidemiologic Studies — Depression Scale (CES-D) was administered to 490 pairs of South Korean adolescent and young adult twins (ages: 13–23 years) by telephone interview. In males, monozygotic (MZ) and dizygotic (DZ) twin correlations were similar (.44 vs. .41), suggesting the importance of shared environmental factors in depression symptoms. In females, however, MZ twin correlation was much greater than DZ twin correlation (.40 vs. .23), indicating the importance of genetic influences. The total phenotypic variance for the CES-D was greater in females than in males. Variance components model confirmed sex differences in the magnitude of genetic and environmental influences on depression symptoms: Additive genetic, shared environmental, and individual specific environmental effects in the full model were, res pectively, 12% (95% CI: 0–54%), 32% (95% CI: 0–53%), and 56% (95% CI: 44–70%) in males, and 41% (95% CI: 0–52%), 0% (95% CI: 0–36%), and 59% (95% CI: 48–72%) in females. Similar results were observed when ‘culturally biased’ items of the CES-D were separately analyzed. These variance components estimates for depression symptoms in East Asians overlap those observed in Caucasians.


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