scholarly journals Ban on sale may cause Indian blood shortage

1998 ◽  
Vol 4 (2) ◽  
pp. 139-139
Author(s):  
K.S. Jayaraman
Keyword(s):  
Blood Shortage ◽  
Indian Blood

Racing Indian Language, Languaging an Indian Race

2020 ◽  
pp. 367-397
Author(s):  
Barbra A. Meek
Keyword(s):  
Native American ◽  
The Other ◽  
Indian Languages ◽  
Indian Language ◽  
Popular Narrative ◽  
American Heritage ◽  
The One ◽  
Indian Blood

This chapter is an exploration of how race and language become entangled in representations and ideas about what it means to be seen and recognized as Native American. Most conceptions of Indianness derive from scholarly European-derived representations and evaluations and from popular narrative media, the one often bootstrapping the other. In tandem, these public manifestations perpetuate the racialization of Indian languages and of Indianness, most ubiquitously in and through a discourse of “blood.” Several ideologies configure the racial logic that determines Indianness: purism (percentage of “Indian blood”), visibility (racialized—and cultural—manifestations of “blood”), continuity (maintenance of a pre-contact “bloodline”), and primitivism (expression of indigenous “blood” in and through language). I argue that this “ideological assemblage” (Kroskrity 2018) undergirds the processes of “racing Indian language(s)” and “languaging an Indian race” (H. Samy Alim 2016) that has resulted in propagating conflicts over and denials of Native American heritage.

Phenotype frequencies of blood group systems (Rh, Kell, Kidd, Duffy, MNS, P, Lewis, and Lutheran) in north Indian blood donors

2010 ◽  
Vol 43 (1) ◽  
pp. 17-22 ◽  
Author(s):  
Beenu Thakral ◽  
Karan Saluja ◽  
Ratti Ram Sharma ◽  
Neelam Marwaha
Keyword(s):  
Blood Group ◽  
Blood Donors ◽  
Indian Blood

The Indian Economy and the British Empire in the Company Period: Some additional reflections around an essay by David Washbrook

2017 ◽  
Vol 51 (2) ◽  
pp. 375-398
Author(s):  
CLAUDE MARKOVITS
Keyword(s):  
British Empire ◽  
Economic Activity ◽  
Balance Of Power ◽  
British India ◽  
Indian Economy ◽  
History Of ◽  
Actual Contribution ◽  
Point Of Departure ◽  
Indian Blood

AbstractTaking as its point of departure David Washbrook's essay ‘The Indian Economy and the British Empire’, this article takes a more detailed look at some episodes in the history of British India in the era of the Company Raj, with a view to placing them within a broader imperial framework, as advocated by Washbrook. The first part of the article examines, through an array of case studies, the actual contribution made by the Company to ‘global’ British expansion, concluding that it invested a lot of (Indian) blood and money in ventures from which it derived little benefit, as in the case of the expeditions to Manila (1762), Ceylon (1795), and Java (1811). It is shown that the Company's interests were ultimately sacrificed to the necessity of maintaining the European balance of power through consideration of the colonial interests of minor European powers such as Portugal or the Netherlands. While the Company saw its interests thus overlooked in the ‘global’ imperial arena, it could not find compensation in increased economic activity in India itself. Although the compulsions of ‘military-fiscalism’ largely explain such an outcome, we should not lose sight of the role of Indian agency in limiting the Company's options, as is shown by a rapid look at the history of both labour and capital markets, which the Company did not succeed in bending completely to its needs.

scholarly journals Weak D prevalence among Indian blood donors

2010 ◽  
Vol 4 (2) ◽  
pp. 137 ◽  
Author(s):  
RN Makroo ◽  
Vimarsh Raina ◽  
Mohit Chowdhry ◽  
Aakanksha Bhatia ◽  
Richa Gupta ◽  
...  
Keyword(s):  
Blood Donors ◽  
Indian Blood

Molecular genotyping of Indian blood group system antigens in Indian blood donors

2018 ◽  
Vol 57 (3) ◽  
pp. 388-390
Author(s):  
Harita Gogri ◽  
Pranali Pitale ◽  
Manisha Madkaikar ◽  
Swati Kulkarni
Keyword(s):  
Blood Group ◽  
Blood Donors ◽  
Blood Group System ◽  
Molecular Genotyping ◽  
Group System ◽  
Indian Blood

Ireland's blood shortage reaches crisis

The Lancet ◽  
1998 ◽  
Vol 351 (9115) ◽  
pp. 1567 ◽  
Author(s):  
Karen Birchard
Keyword(s):  
Blood Shortage

Julio C. Tello, 1880–1947

1948 ◽  
Vol 14 (1) ◽  
pp. 50-56 ◽  
Author(s):  
S. K. Lothrop
Keyword(s):  
Fishing Villages ◽  
The Family ◽  
Indian Village ◽  
The City ◽  
Indian Blood

Julio César Tello, outstanding Peruvian archaeologist, was born on April 11, 1880, under humble circumstances in the Indian village of Huarochirí, Department of Lima, in the high Andes. He died on June 3, 1947, in the city of Lima and was buried with the pomp and pageantry accorded to a Minister of State. Tello was of almost pure Indian blood, of which he was proud. His father, Julian Tello, evidently was a man of local importance as he became alcalde, but the family was poor and Julio Tello's first memory was of playing on an adobe floor. His mother was María Asunción Rojas de Tello, a descerident of the last Inca curaca who governed Huarochirí. During his childhood, Julio's father made periodic journeys to the coast where he visited relatives in the small fishing villages south of Lima. He often took his son with him on these trips.

scholarly journals Novel variants in Krueppel Like Factor 1 that cause Persistence of Fetal Hemoglobin in In(Lu) individuals

2021 ◽  
Author(s):  
Jesse Eernstman ◽  
Barbera Veldhuisen ◽  
Peter Ligthart ◽  
Marieke von Lindern ◽  
Ellen van der Schoot ◽  
...  
Keyword(s):  
Flow Cytometry ◽  
Transcription Factor ◽  
Blood Group ◽  
High Frequency ◽  
Fetal Hemoglobin ◽  
Donor Group ◽  
Wild Type ◽  
Novel Variants ◽  
Klf1 Gene ◽  
Indian Blood

Abstract Beta-hemoglobinopathies become prominent after birth due to a switch from γ-globin to the mutated β-globin. Haploinsufficiency for the erythroid specific indispensable transcription factor Krueppel-like factor 1 (KLF1) is associated with high persistence of fetal hemoglobin (HPFH). The In(Lu) phenotype, characterized by low to undetectable Lutheran blood group expression is caused by mutations within KLF1 gene. These KLF1 variants often lead to KLF1 haploinsufficiency. We screened a donor cohort of 55 Lutheran weak or negative donors for KLF1 variants. To discriminate between weak and negative Lutheran expression, a flow cytometry (FCM) assay was developed to detect Lu polymorphisms. The Lu(a-b-) (negative) donor group, showing a significant decreased CD44 (Indian blood group) expression, also showed increased HbF and HbA2 levels, with outliers expressing >5% HbF. KLF1 exons and promoter sequencing revealed variants in 80% of the Lutheran negative donors. Thirteen different variants plus one high frequency SNP (c.304T>C) were identified of which 6 were novel. In primary erythroblasts, knockdown of endogenous KLF1 resulted in decreased CD44, Lu and increased HbF expression, while KLF1 over-expressing cells were comparable to wild type (WT). In line with the pleiotropic effects of KLF1 during erythropoiesis, distinct KLF1 mutants expressed in erythroblasts display different abilities to rescue CD44 and Lu expression and/or to affect fetal (HbF) or adult (HbA) hemoglobin expression.

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