scholarly journals Effects of a low-fat high-carbohydrate diet on plasma sex hormones in premenopausal women: results from a randomized controlled trial

1997 ◽  
Vol 76 (1) ◽  
pp. 127-135 ◽  
Author(s):  
NF Boyd ◽  
◽  
GA Lockwood ◽  
CV Greenberg ◽  
LJ Martin ◽  
...  
Keyword(s):  
Randomized Controlled Trial ◽  
Sex Hormones ◽  
Controlled Trial ◽  
Premenopausal Women ◽  
High Carbohydrate Diet ◽  
Low Fat ◽  
Carbohydrate Diet ◽  
High Carbohydrate ◽  
Randomized Controlled

Effects of Diet and Diet-Plus-Exercise Programs on Resting Metabolic Rate: A Meta-Analysis

1996 ◽  
Vol 6 (1) ◽  
pp. 41-61 ◽  
Author(s):  
Janice L. Thompson ◽  
Melinda M. Manore ◽  
Jerry R. Thomas
Keyword(s):  
Metabolic Rate ◽  
Dietary Restriction ◽  
Meta Analysis ◽  
Resting Metabolic Rate ◽  
Premenopausal Women ◽  
High Carbohydrate Diet ◽  
Moderate Intensity ◽  
Low Fat ◽  
Carbohydrate Diet ◽  
High Carbohydrate

Studies examining the effects of diet (D) and diet-plus-exercise (DE) programs on resting metabolic rate (RMR) report equivocal results. The purpose of this study was to use meta-analysis to determine if exercise prevents the decrease in RMR observed with dieting. Results from the 22 studies included in this analysis revealed that the majority of studies used female subjects ages 31-45 years, who were fed a relatively low-fat, high-carbohydrate diet of less than 5,023 kJ · day1. The predominant prescribed exercise was aerobic in nature, 31-60 min in duration, performed 4-5 days per week, and of moderate intensity (51-70% of). Contrary to what is reported in narrative reviews, RMR decreased significantly with both D and DE programs, and the drop with D was significantly greater than that with DE. In conclusion, the addition of exercise to dietary restriction appears to prevent some of the decrease in RMR observed in premenopausal women.

scholarly journals Premeal Low-Fat Yogurt Consumption Reduces Postprandial Inflammation and Markers of Endotoxin Exposure in Healthy Premenopausal Women in a Randomized Controlled Trial

2018 ◽  
Vol 148 (6) ◽  
pp. 910-916 ◽  
Author(s):  
Ruisong Pei ◽  
Diana M DiMarco ◽  
Kelley K Putt ◽  
Derek A Martin ◽  
Chureeporn Chitchumroonchokchai ◽  
...  
Keyword(s):  
Randomized Controlled Trial ◽  
Controlled Trial ◽  
Premenopausal Women ◽  
Low Fat ◽  
Randomized Controlled ◽  
Endotoxin Exposure ◽  
Postprandial Inflammation

scholarly journals An Almond-Based Low Carbohydrate Diet Improves Depression and Glycometabolism in Patients with Type 2 Diabetes through Modulating Gut Microbiota and GLP-1: A Randomized Controlled Trial

Nutrients ◽  
2020 ◽  
Vol 12 (10) ◽  
pp. 3036
Author(s):  
Mengxiao Ren ◽  
Huaiyu Zhang ◽  
Jindan Qi ◽  
Anni Hu ◽  
Qing Jiang ◽  
...  
Keyword(s):  
Type 2 Diabetes ◽  
Randomized Controlled Trial ◽  
Gut Microbiota ◽  
Controlled Trial ◽  
Glycosylated Hemoglobin ◽  
Depression Symptoms ◽  
Carbohydrate Diet ◽  
Low Carbohydrate Diet ◽  
Randomized Controlled

Background: Alow carbohydrate diet (LCD) is more beneficial for the glycometabolism in type 2 diabetes (T2DM) and may be effective in reducing depression. Almond, which is a common nut, has been shown to effectively improve hyperglycemia and depression symptoms. This study aimed to determine the effect of an almond-based LCD (a-LCD) on depression and glycometabolism, as well as gut microbiota and fasting glucagon-like peptide 1 (GLP-1) in patients with T2DM. Methods: This was a randomized controlled trial which compared an a-LCD with a low-fat diet (LFD). Forty-five participants with T2DM at a diabetes club and the Endocrine Division of the First and Second Affiliated Hospital of Soochow University between December 2018 to December 2019 completed each dietary intervention for 3 months, including 22 in the a-LCD group and 23 in the LFD group. The indicators for depression and biochemical indicators including glycosylated hemoglobin (HbA1c), gut microbiota, and GLP-1 concentration were assessed at the baseline and third month and compared between the two groups. Results: A-LCD significantly improved depression and HbA1c (p < 0.01). Meanwhile, a-LCD significantly increased the short chain fatty acid (SCFAs)-producing bacteria Roseburia, Ruminococcus and Eubacterium. The GLP-1 concentration in the a-LCD group was higher than that in the LFD group (p < 0.05). Conclusions: A-LCD could exert a beneficial effect on depression and glycometabolism in patients with T2DM. We speculate that the role of a-LCD in improving depression in patients with T2DM may be associated with it stimulating the growth of SCFAs-producing bacteria, increasing SCFAs production and GPR43 activation, and further maintaining GLP-1 secretion. In future studies, the SCFAs and GPR43 activation should be further examined.

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