Central progesterone induces female sexual behavior in estrogen-primed intact male rats.

1977 ◽  
Vol 91 (6) ◽  
pp. 1417-1423 ◽  
Author(s):  
Ingeborg L. Ward ◽  
JoAnn E. Franck ◽  
William R. Crowley
Keyword(s):  
Sexual Behavior ◽  
Male Rats ◽  
Intact Male ◽  
Female Sexual Behavior

scholarly journals Effects of vasopressin on female sexual behavior in male rats

1986 ◽  
Vol 69 (2) ◽  
pp. 188-191 ◽  
Author(s):  
P. Södersten ◽  
G.J. Boer ◽  
G.J. De Vries ◽  
R.M. Buijs ◽  
P. Melin
Keyword(s):  
Sexual Behavior ◽  
Male Rats ◽  
Female Sexual Behavior

Anabolic–Androgenic Steroid Effects on the Sexual Behavior of Intact Male Rats

1997 ◽  
Vol 31 (1) ◽  
pp. 35-46 ◽  
Author(s):  
Ann S. Clark ◽  
Elizabeth V. Harrold ◽  
Alison S. Fast
Keyword(s):  
Sexual Behavior ◽  
Male Rats ◽  
Anabolic Androgenic Steroid ◽  
Intact Male ◽  
Androgenic Steroid

Female sexual behavior in male rats: Effect of hour of castration at birth

1983 ◽  
Vol 30 (4) ◽  
pp. 613-616 ◽  
Author(s):  
P. Corbier ◽  
J. Roffi ◽  
J. Rhoda
Keyword(s):  
Sexual Behavior ◽  
Male Rats ◽  
Female Sexual Behavior

Effects of oestradiol benzoate (OeB) and human chorionic gonadotrophin (hCG) on the testes and accessory sex glands of the rat

1981 ◽  
Vol 96 (2) ◽  
pp. 273-280 ◽  
Author(s):  
Mridula Chowdhury ◽  
Robert Tcholakian ◽  
Emil Steinberger
Keyword(s):  
Treated Group ◽  
Inhibitory Effect ◽  
Male Rats ◽  
Plasma Testosterone ◽  
Control Group ◽  
Intact Male ◽  
Intact Control ◽  
Testosterone Levels ◽  
Oestradiol Benzoate ◽  
Direct Inhibitory Effect

Abstract. It has been suggested that treatment of intact male rats with oestradiol benzoate (OeB) causes an interference with testosterone (T) production by the testes by a direct inhibitory effect on steroidogenesis. To test this hypothesis, different doses (5, 10 or 25 IU) of hCG were administered concomitantly with 50 μg of OeB to adult intact or hypophysectomized male rats. The testicular and plasma testosterone, and serum hCG levels were determined. The sex accessory weights were recorded. In the intact OeB-treated group of animals, hCG stimulated both the secondary sex organs and plasma testosterone levels above the intact control group. However, in hypophysectomized animals, although plasma testosterone levels increased above that of intact controls, their secondary sex organ weights did not. Moreover, inspite of high circulating hCG levels, the testicular testosterone content and concentration remained suppressed in OeB-treated animals. The reason for such dichotomy of hCG action on OeB-treated animals is not clear at present.

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