Food versus no food on the pre-delay trial of delayed response.

1941 ◽  
Vol 32 (1) ◽  
pp. 153-164 ◽  
Author(s):  
John T. Cowles
Keyword(s):  
Delayed Response ◽  
Delay Trial

Analysis of changes in indirect delayed response performance in monkeys with prefrontal lesions.

1969 ◽  
Vol 68 (1, Pt.1) ◽  
pp. 147-154 ◽  
Author(s):  
Roger W. Buddington ◽  
Frederick A. King ◽  
Lamar Roberts
Keyword(s):  
Delayed Response ◽  
Response Performance

Effects of Ethyl Alcohol on Retention in a Delayed-Response Test

1965 ◽  
Vol 26 (3) ◽  
pp. 384-392 ◽  
Author(s):  
Elizabeth K. Van Laer ◽  
Murray E. Jarvik ◽  
John Van Laer
Keyword(s):  
Ethyl Alcohol ◽  
Delayed Response ◽  
Response Test

scholarly journals Time Course of ECG Depolarization and Repolarization Changes during Ischemia in PTCA Recordings

2004 ◽  
Vol 43 (01) ◽  
pp. 43-46 ◽  
Author(s):  
J. García ◽  
G. Wagner ◽  
R. Bailón ◽  
L. Sörnmo ◽  
P. Laguna ◽  
...  
Keyword(s):  
Time Course ◽  
Wave Amplitude ◽  
Temporal Evolution ◽  
Delayed Response ◽  
S Wave ◽  
Ecg Changes ◽  
T Complex ◽  
Karhunen Loeve Transform ◽  
Low Amplitude ◽  
Electrocardiogram Ecg

Summary Objectives: In this work we studied the temporal evolution of changes in the electrocardiogram (ECG) as a consequence of the induced ischemia during prolonged coronary angioplasty, comparing the time course of indexes reflecting depolarization and those reflecting repolarization. Methods: We considered both local (measured at specific points of the ECG) and global (obtained from the Karhunen-Loève transform) indexes. In particular, the evolution of Q, R and S wave amplitudes during ischemia was analyzed with respect to classical indexes such as ST level. As a measurement of sensitivity we used an Ischemic Changes Sensor (ICS), which reflects the capacity of an index to detect changes in the ECG. Results: The results showed that, in leads with low-amplitude ST-T complexes, the S wave amplitude was more sensitive in detecting ischemia than was the commonly used index ST60. It was found that in such leads the S wave amplitude initially exhibited a delayed response to ischemia when compared to ST60, but its performance was better from the second minute of occlusion. The global indexes describing the ST-T complex were, in terms of the ICS, superior to the S wave amplitude for ischemia detection. Conclusions: Ischemic ECG changes occur both at repolarization and depolarization, with alterations in the depolarization period appearing later in time. Local indexes are less sensitive to ischemia than global ones.

Successive Modulation of ENSO to the Future Greenhouse Warming

2008 ◽  
Vol 21 (1) ◽  
pp. 3-21 ◽  
Author(s):  
Soon-Il An ◽  
Jong-Seong Kug ◽  
Yoo-Geun Ham ◽  
In-Sik Kang
Keyword(s):  
General Circulation ◽  
Southern Oscillation ◽  
General Circulation Models ◽  
Tropical Pacific ◽  
Greenhouse Warming ◽  
Delayed Response ◽  
Subsurface Temperature ◽  
Climate System Model ◽  
The Mean ◽  
The Tropical Pacific

Abstract The multidecadal modulation of the El Niño–Southern Oscillation (ENSO) due to greenhouse warming has been analyzed herein by means of diagnostics of Intergovernmental Panel on Climate Change (IPCC) Fourth Assessment Report (AR4) coupled general circulation models (CGCMs) and the eigenanalysis of a simplified version of an intermediate ENSO model. The response of the global-mean troposphere temperature to increasing greenhouse gases is more likely linear, while the amplitude and period of ENSO fluctuates in a multidecadal time scale. The climate system model outputs suggest that the multidecadal modulation of ENSO is related to the delayed response of the subsurface temperature in the tropical Pacific compared to the response time of the sea surface temperature (SST), which would lead a modulation of the vertical temperature gradient. Furthermore, an eigenanalysis considering only two parameters, the changes in the zonal contrast of the mean background SST and the changes in the vertical contrast between the mean surface and subsurface temperatures in the tropical Pacific, exhibits a good agreement with the CGCM outputs in terms of the multidecadal modulations of the ENSO amplitude and period. In particular, the change in the vertical contrast, that is, change in difference between the subsurface temperature and SST, turns out to be more influential on the ENSO modulation than changes in the mean SST itself.

scholarly journals Activation of the p11/SMARCA3/Neurensin-2 pathway in parvalbumin interneurons mediates the response to chronic antidepressants

2021 ◽  
Author(s):  
Gali Umschweif ◽  
Lucian Medrihan ◽  
Kathryn A. McCabe ◽  
Yotam Sagi ◽  
Paul Greengard
Keyword(s):  
Chromatin Remodeling ◽  
Behavioral Response ◽  
Hippocampal Neurons ◽  
Selective Serotonin Reuptake Inhibitors ◽  
Chronic Treatment ◽  
Target Genes ◽  
Delayed Response ◽  
Dynamic Changes ◽  
Parvalbumin Interneurons ◽  
Transcriptional Changes

AbstractThe delayed behavioral response to chronic antidepressants depends on dynamic changes in the hippocampus. It was suggested that the antidepressant protein p11 and the chromatin remodeling factor SMARCA3 mediate this delayed response by inducing transcriptional changes in hippocampal neurons. However, what target genes are regulated by the p11/SMARCA3 complex to mediate the behavioral response to antidepressants, and what cell type mediates these molecular changes remain unknown. Here we report that the p11/SMARCA3 complex represses Neurensin-2 transcription in hippocampal parvalbumin-expressing interneurons after chronic treatment with Selective Serotonin Reuptake Inhibitors (SSRI). The behavioral response to antidepressants requires upregulation of p11, accumulation of SMARCA3 in the cell nucleus, and a consequent repression of Neurensin-2 transcription in these interneurons. We elucidate a functional role for p11/SMARCA3/Neurensin-2 pathway in regulating AMPA-receptor signaling in parvalbumin-expressing interneurons, a function that is enhanced by chronic treatment with SSRIs. These results link SSRIs to dynamic glutamatergic changes and implicate p11/SMARCA3/Neurensin-2 pathway in the development of more specific and efficient therapeutic strategies for neuropsychiatric disorders.

Delayed response to chest pain

1989 ◽  
Vol 150 (7) ◽  
pp. 412-412
Author(s):  
John Coleridge ◽  
Peter Cameron
Keyword(s):  
Chest Pain ◽  
Delayed Response
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