Inter-trial responding as a factor in rate and asymptote of reversal learning

1963 ◽  
Author(s):  
Robert M. Wageman ◽  
Harold W. Coppock
Keyword(s):  
Reversal Learning

Reversal learning in youth with generalized anxiety disorder and bipolar disorder

2014 ◽  
Author(s):  
Grace K. Cushman ◽  
Megan E. Puzia ◽  
Alexandra B. Weissman ◽  
Ezra Wegbreit ◽  
Kerri L. Kim ◽  
...  
Keyword(s):  
Bipolar Disorder ◽  
Anxiety Disorder ◽  
Generalized Anxiety Disorder ◽  
Reversal Learning ◽  
Generalized Anxiety

Midsession reversal learning: Pigeons learn what stimulus to avoid.

2020 ◽  
Vol 46 (2) ◽  
pp. 101-106
Author(s):  
Thomas R. Zentall ◽  
Megan Halloran ◽  
Daniel Peng
Keyword(s):  
Reversal Learning ◽  
Midsession Reversal

scholarly journals Impaired probabilistic reversal learning in anxiety: Evidence from behavioral and ERP findings

2021 ◽  
pp. 102751
Author(s):  
Lisheng Xia ◽  
Pengfei Xu ◽  
Ziyan Yang ◽  
Ruolei Gu ◽  
Dandan Zhang
Keyword(s):  
Reversal Learning ◽  
Probabilistic Reversal Learning

scholarly journals Dissecting neural pathways for forgetting in Drosophila olfactory aversive memory

2015 ◽  
Vol 112 (48) ◽  
pp. E6663-E6672 ◽  
Author(s):  
Yichun Shuai ◽  
Areekul Hirokawa ◽  
Yulian Ai ◽  
Min Zhang ◽  
Wanhe Li ◽  
...  
Keyword(s):  
Reversal Learning ◽  
Dopaminergic Neurons ◽  
Mushroom Body ◽  
Biologically Active ◽  
Molecular Pathways ◽  
Neural Pathways ◽  
Active Process ◽  
Glutamatergic Neurons ◽  
Memory Decay ◽  
Over Time

Recent studies have identified molecular pathways driving forgetting and supported the notion that forgetting is a biologically active process. The circuit mechanisms of forgetting, however, remain largely unknown. Here we report two sets of Drosophila neurons that account for the rapid forgetting of early olfactory aversive memory. We show that inactivating these neurons inhibits memory decay without altering learning, whereas activating them promotes forgetting. These neurons, including a cluster of dopaminergic neurons (PAM-β′1) and a pair of glutamatergic neurons (MBON-γ4>γ1γ2), terminate in distinct subdomains in the mushroom body and represent parallel neural pathways for regulating forgetting. Interestingly, although activity of these neurons is required for memory decay over time, they are not required for acute forgetting during reversal learning. Our results thus not only establish the presence of multiple neural pathways for forgetting in Drosophila but also suggest the existence of diverse circuit mechanisms of forgetting in different contexts.

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