scholarly journals Age differences in prenatal testosterone’s protective effects on disordered eating symptoms: Developmental windows of expression?

2015 ◽  
Vol 129 (1) ◽  
pp. 18-36 ◽  
Author(s):  
Kristen M. Culbert ◽  
S. Marc Breedlove ◽  
Cheryl L. Sisk ◽  
Pamela K. Keel ◽  
Michael C. Neale ◽  
...  
Keyword(s):  
Disordered Eating ◽  
Age Differences ◽  
Protective Effects ◽  
Developmental Windows ◽  
Disordered Eating Symptoms

Etiologic and treatment conceptualizations of disordered eating symptoms among mainland Chinese therapists

2019 ◽  
Vol 53 (3) ◽  
pp. 391-403
Author(s):  
Ruofan Ma ◽  
Kevin P. Capobianco ◽  
NiCole T. Buchanan ◽  
Zhiyuan Hu ◽  
Jonathan M. Oakman
Keyword(s):  
Disordered Eating ◽  
Mainland Chinese ◽  
Disordered Eating Symptoms

scholarly journals Advanced paternal age at birth: phenotypic and etiologic associations with eating pathology in offspring

2013 ◽  
Vol 44 (5) ◽  
pp. 1029-1041 ◽  
Author(s):  
S. E. Racine ◽  
K. M. Culbert ◽  
S. A. Burt ◽  
K. L. Klump
Keyword(s):  
Eating Disorder ◽  
Disordered Eating ◽  
Eating Pathology ◽  
Paternal Age ◽  
Advanced Paternal Age ◽  
Eating Disorder Risk ◽  
Twin Models ◽  
Age At Birth ◽  
Disorder Risk ◽  
Disordered Eating Symptoms

BackgroundAdvanced paternal age at birth has been linked to several psychiatric disorders in offspring (e.g. schizophrenia) and genetic mechanisms are thought to underlie these associations. This study is the first to investigate whether advanced paternal age at birth is associated with eating disorder risk using a twin study design capable of examining both phenotypic and genetic associations.MethodIn a large, population-based sample of female twins aged 8–17 years in mid-puberty or beyond (n = 1722), we investigated whether advanced paternal age was positively associated with disordered eating symptoms and an eating disorder history [i.e. anorexia nervosa (AN), bulimia nervosa (BN) or binge eating disorder (BED)] in offspring. Biometric twin models examined whether genetic and/or environmental factors underlie paternal age effects for disordered eating symptoms.ResultsAdvanced paternal age was positively associated with disordered eating symptoms and an eating disorder history, where the highest level of pathology was observed in offspring born to fathers ⩾40 years old. The results were not accounted for by maternal age at birth, body mass index (BMI), socio-economic status (SES), fertility treatment or parental psychiatric history. Twin models indicated decreased genetic, and increased environmental, effects on disordered eating with advanced paternal age.ConclusionsAdvanced paternal age increased risk for the full spectrum of eating pathology, independent of several important covariates. However, contrary to leading hypotheses, environmental rather than genetic factors accounted for paternal age–disordered eating associations. These data highlight the need to explore novel (potentially environmental) mechanisms underlying the effects of advanced paternal age on offspring eating disorder risk.

scholarly journals Mother–Child and Father–Child Connectedness in Adolescence and Disordered Eating Symptoms in Young Adulthood

2020 ◽  
Vol 66 (3) ◽  
pp. 366-371
Author(s):  
Vivienne M. Hazzard ◽  
Alison L. Miller ◽  
Katherine W. Bauer ◽  
Bhramar Mukherjee ◽  
Kendrin R. Sonneville
Keyword(s):  
Disordered Eating ◽  
Young Adulthood ◽  
Disordered Eating Symptoms

Body dissatisfaction and disordered eating symptoms in Finnish preadolescents

2018 ◽  
Vol 27 (1) ◽  
pp. 34-51 ◽  
Author(s):  
Rejane Augusta de Oliveira Figueiredo ◽  
Sabina Simola-Ström ◽  
Rasmus Isomaa ◽  
Elisabete Weiderpass
Keyword(s):  
Body Dissatisfaction ◽  
Disordered Eating ◽  
Disordered Eating Symptoms

Disordered Eating Symptoms Scale

2015 ◽  
Author(s):  
Annie Shearer ◽  
Jody Russon ◽  
Joanna Herres ◽  
Tita Atte ◽  
Tamar Kodish ◽  
...  
Keyword(s):  
Disordered Eating ◽  
Disordered Eating Symptoms

scholarly journals The effects of circulating testosterone and pubertal maturation on risk for disordered eating symptoms in adolescent males

2014 ◽  
Vol 44 (11) ◽  
pp. 2271-2286 ◽  
Author(s):  
K. M. Culbert ◽  
S. A. Burt ◽  
C. L. Sisk ◽  
J. T. Nigg ◽  
K. L. Klump
Keyword(s):  
Eating Disorders ◽  
Disordered Eating ◽  
Pubertal Development ◽  
Biological Factor ◽  
Adolescent Boys ◽  
Pubertal Maturation ◽  
Eating Disorder Examination ◽  
Animal Data ◽  
Circulating Levels ◽  
Disordered Eating Symptoms

BackgroundTestosterone may be a biological factor that protects males against eating disorders. Elevated prenatal testosterone exposure is linked to lower levels of disordered eating symptoms, but effects emerge only after mid-puberty. Whether circulating levels of testosterone account for decreased risk for disordered eating in boys after mid-puberty is currently unknown; however, animal data support this possibility. In rodents, prenatal testosterone's masculinizing effects on sex-differentiated behaviors emerge during puberty when circulating levels of testosterone increase and ‘activate’ the expression of masculinized phenotypes. This study investigated whether higher levels of circulating testosterone predict lower levels of disordered eating symptoms in adolescent boys, and in particular whether effects are associated with advancing pubertal maturation.MethodParticipants were 213 male twins from the Michigan State University Twin Registry. The Minnesota Eating Behavior Survey and Eating Disorder Examination Questionnaire assessed several disordered eating symptoms. The Pubertal Development Scale assessed pubertal status. Afternoon saliva samples were assayed for testosterone using enzyme immunoassays.ResultsConsistent with animal data, higher levels of circulating testosterone predicted lower levels of disordered eating symptoms in adolescent boys and effects emerged with advancing puberty. Results were not accounted for by several important covariates, including age, adiposity, or mood/anxiety symptoms.ConclusionsFindings suggest that elevated circulating testosterone may be protective and underlie decreased risk for eating pathology in males during/after puberty, whereas lower levels of testosterone may increase risk and explain why some, albeit relatively few, males develop eating disorders.

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