Healthy Eating Opinion Survey for individuals at risk for cardiovascular disease.

2014 ◽  
Vol 33 (8) ◽  
pp. 904-911 ◽  
Author(s):  
Amy E. Mark ◽  
Dana L. Riley ◽  
Lisa A. McDonnell ◽  
Andrew L. Pipe ◽  
Robert D. Reid
2018 ◽  
Vol 32 ◽  
pp. 43
Author(s):  
Luke Hamilton ◽  
Alejandro de la Torre ◽  
Lisa Guerra ◽  
Amelia Vinson ◽  
Lauren Williams ◽  
...  

2021 ◽  
Vol 11 (4) ◽  
pp. 261
Author(s):  
Flavia Diggelmann ◽  
Susan Bengs ◽  
Ahmed Haider ◽  
Gioia Epprecht ◽  
Anna Luisa Beeler ◽  
...  

Background: Recent studies indicate that enhanced neuronal stress responses are associated with adverse cardiovascular outcomes. A chronic inflammatory state seems to mediate this detrimental neuro-cardiac communication. Statins are among the most widely prescribed medications in primary and secondary cardiovascular disease (CVD) prevention and not only lower lipid levels but also exhibit strong anti-inflammatory and neuroprotective effects. We therefore sought to investigate the influence of statins on neuronal stress responses in a patient cohort at risk for CVD. Methods: 563 patients (61.5 ± 14.0 years) who underwent echocardiography and 18F-fluorodeoxyglucose (18F-FDG) positron emission tomography (PET) were retrospectively identified. Metabolic activity of the amygdala, a part of the brain’s salience network, was quantified by 18F-FDG uptake, while normal cardiac morphology and function were assured by echocardiography. Vertebral bone marrow metabolism, a marker of inflammatory activity, was measured by 18F-FDG PET. Results: Increased neuronal stress responses were associated with an increased inflammatory activity in the bone marrow (r = 0.152, p = 0.015) as well as with a subclinical reduction in left ventricular ejection fraction (LVEF, r = −0.138, p = 0.025). In a fully-adjusted linear regression model, statin treatment was identified as an independent, negative predictor of amygdalar metabolic activity (B-coefficient −0.171, p = 0.043). Conclusions: Our hypothesis-generating investigation suggests a potential link between the anti-inflammatory actions of statins and reduced neuronal stress responses which could lead to improved cardiovascular outcomes. The latter warrants further studies in a larger and prospective population.


2018 ◽  
Vol 48 (12) ◽  
pp. 2785-2795 ◽  
Author(s):  
William Evans ◽  
Quentin Willey ◽  
Erik D. Hanson ◽  
Lee Stoner

Nutrients ◽  
2018 ◽  
Vol 10 (11) ◽  
pp. 1725 ◽  
Author(s):  
Lauren O'Connor ◽  
Jia Li ◽  
R. Drew Sayer ◽  
Jane Hennessy ◽  
Wayne Campbell

Adherence to healthy eating patterns (HEPs) is often short-lived and can lead to repetitive attempts of adopting—but not maintaining—HEPs. We assessed effects of adopting, abandoning, and readopting HEPs (HEP cycling) on cardiovascular disease risk factors (CVD-RF). We hypothesized that HEP cycling would improve, worsen, and again improve CVD-RF. Data were retrospectively pooled for secondary analyses from two randomized, crossover, controlled feeding trials (n = 60, 52 ± 2 years, 30.6 ± 0.6 kg/m2) which included two 5–6 week HEP interventions (Dietary Approaches to Stop Hypertension-style or Mediterranean-style) separated by a four-week unrestricted eating period. Ambulatory and fasting blood pressures (BP), fasting serum lipids, lipoproteins, glucose, and insulin were measured before and during the last week of HEP interventions. Fasting systolic BP and total cholesterol decreased (−6 ± 1 mm Hg and −19 ± 3 mg/dL, respectively, p < 0.05), returned to baseline, then decreased again (−5 ± 1 mm Hg and −13 ± 3 mg/dL, respectively, p < 0.05) when adopting, abandoning, and readopting a HEP; magnitude of changes did not differ. Ambulatory and fasting diastolic BP and high-density lipoprotein cholesterol concentrations followed similar patterns; glucose and insulin remained unchanged. Low-density lipoprotein cholesterol concentrations decreased with initial adoption but not readoption (−13 ± 3 and −6 ± 3, respectively, interaction p = 0.020). Healthcare professionals should encourage individuals to consistently consume a HEP for cardiovascular health but also encourage them to try again if a first attempt is unsuccessful or short-lived.


2015 ◽  
Vol 2 (2) ◽  
pp. 56-63 ◽  
Author(s):  
Jennifer T Fink ◽  
Kathryn K Havens ◽  
Julia A Schumacher ◽  
Renee E Walker ◽  
George L Morris ◽  
...  

2011 ◽  
Vol 11 (1) ◽  
Author(s):  
Simon L Bacon ◽  
Kim L Lavoie ◽  
André Arsenault ◽  
Jocelyn Dupuis ◽  
Louise Pilote ◽  
...  

2010 ◽  
Vol 10 (2) ◽  
pp. 85-94 ◽  
Author(s):  
Zanfina Ademi ◽  
Danny Liew ◽  
Bruce Hollingsworth ◽  
Ph. Gabriel Steg ◽  
Deepak L. Bhatt ◽  
...  

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