Delayed growth of ferropericlase and bridgmanite controls slab residence at the 660‐km discontinuity

2021 ◽  
Author(s):  
Timothy Chapman ◽  
Geoffrey L. Clarke
Keyword(s):  
Delayed Growth

Delayed Growth – A Case of Hamlet Without the Prince?

1997 ◽  
Vol 7 (6) ◽  
pp. 14-15
Author(s):  
Robert M. Bernstein ◽  
B.L. Limmer ◽  
William R. Rassman
Keyword(s):  
Delayed Growth

What Is Delayed Growth?

1997 ◽  
Vol 7 (2) ◽  
pp. 22-22
Author(s):  
Robert M. Bernstein ◽  
William R. Rassman
Keyword(s):  
Delayed Growth

Delayed Growth

1997 ◽  
Vol 7 (6) ◽  
pp. 13.3-13
Author(s):  
Walter P. Unger
Keyword(s):  
Delayed Growth

Herbicide Tolerance Of Transgenic `Stevens' Cranberry Plants Depends on the Test Environment

HortScience ◽  
1998 ◽  
Vol 33 (3) ◽  
pp. 517a-517
Author(s):  
Eric L. Zeldin ◽  
Rodney A. Serres ◽  
Brent H. McCown
Keyword(s):  
Growth Form ◽  
Herbicide Tolerance ◽  
Genetically Engineered ◽  
Shoot Tip ◽  
Test Environment ◽  
Growth Environment ◽  
Commercial Formulation ◽  
Tolerance Level ◽  
Delayed Growth ◽  
Significant Injury

`Stevens' cranberry was genetically engineered to confer tolerance to the broad spectrum herbicide glufosinate. Initially, herbicide tolerance was verified by spraying greenhouse plants with the commercial formulation Liberty. Although one transformant showed significant tolerance, the tolerance level was below that required to kill goldenrod, a common weed of cranberry beds. This transformant was propagated and the plants established outdoors in a coldframe, yielding a growth form more typical of field-grown plants than that of greenhouse-grown plants. These plants, as well as untransformed cranberry and goldenrod plants, were sprayed with various levels of the herbicide. The transformed plants were not killed at glufosinate concentrations up to 1000 ppm, although delayed growth did occur. Some runner tip injury was observed at 500 ppm as well as widespread shoot tip death at higher levels. The above-ground parts of goldenrod plants were killed at 400 ppm with significant injury at 200 ppm. Untransformed cranberry plants were killed at 300 ppm and had extensive tip death even at 100 ppm. Transformed cranberry plants with confirmed “field” tolerance were re-established in the greenhouse and new vegetative growth was forced. When these plants were sprayed with glufosinate, significant shoot tip injury was observed at levels as low as 100 ppm. The degree of herbicide tolerance of transformed cranberry appears to be modulated by the growth environment, which may affect the expression of the inserted genes or the physiological sensitivity of the impacted tissues.

scholarly journals Kog1/Raptor mediates metabolic rewiring during nutrient limitation by controlling SNF1/AMPK activity

2021 ◽  
Vol 7 (16) ◽  
pp. eabe5544
Author(s):  
Zeenat Rashida ◽  
Rajalakshmi Srinivasan ◽  
Meghana Cyanam ◽  
Sunil Laxman
Keyword(s):  
Amino Acid ◽  
Nutrient Limitation ◽  
Carbon Flux ◽  
Metabolic Flux ◽  
Carbon Allocation ◽  
Carbon Utilization ◽  
Acid Biosynthesis ◽  
Control Growth ◽  
Ampk Activity ◽  
Delayed Growth

In changing environments, cells modulate resource budgeting through distinct metabolic routes to control growth. Accordingly, the TORC1 and SNF1/AMPK pathways operate contrastingly in nutrient replete or limited environments to maintain homeostasis. The functions of TORC1 under glucose and amino acid limitation are relatively unknown. We identified a modified form of the yeast TORC1 component Kog1/Raptor, which exhibits delayed growth exclusively during glucose and amino acid limitations. Using this, we found a necessary function for Kog1 in these conditions where TORC1 kinase activity is undetectable. Metabolic flux and transcriptome analysis revealed that Kog1 controls SNF1-dependent carbon flux apportioning between glutamate/amino acid biosynthesis and gluconeogenesis. Kog1 regulates SNF1/AMPK activity and outputs and mediates a rapamycin-independent activation of the SNF1 targets Mig1 and Cat8. This enables effective glucose derepression, gluconeogenesis activation, and carbon allocation through different pathways. Therefore, Kog1 centrally regulates metabolic homeostasis and carbon utilization during nutrient limitation by managing SNF1 activity.

scholarly journals Bone metabolism compensates for the delayed growth in small for gestational age neonates

Organogenesis ◽  
2013 ◽  
Vol 9 (1) ◽  
pp. 55-59 ◽  
Author(s):  
Roxane Tenta ◽  
Ifigeneia Bourgiezi ◽  
Evangelos Aliferis ◽  
Magdalini Papadopoulou ◽  
Antonis Gounaris ◽  
...  
Keyword(s):  
Bone Metabolism ◽  
Gestational Age ◽  
Small For Gestational Age ◽  
Delayed Growth

scholarly journals Delayed growth of large-scale instabilities on the surface of double-layer (Cu + Ti) conductors in strong magnetic fields

2018 ◽  
Vol 1115 ◽  
pp. 022011
Author(s):  
N A Labetskaya ◽  
I M Datsko ◽  
V I Oreshkin ◽  
S A Chaikovsky ◽  
D V Rybka ◽  
...  
Keyword(s):  
Magnetic Fields ◽  
Double Layer ◽  
Large Scale ◽  
Strong Magnetic Fields ◽  
Delayed Growth

Sabellaria Colonies at Duckpool, North Cornwall, 1971–1972, With a Note for May 1973

1974 ◽  
Vol 54 (2) ◽  
pp. 393-436 ◽  
Author(s):  
Douglas P. Wilson
Keyword(s):  
Surface Area ◽  
Close Packing ◽  
Wave Action ◽  
Physical Damage ◽  
Fast Growing ◽  
New Knowledge ◽  
Rapid Elongation ◽  
Delayed Growth

The exceptionally heavy settlement of larvae in the autumn of 1970 made it imperative to continue the observations of the previous ten years. This two-year extension has yielded much new knowledge about the growth of Sabellaria colonies at Duckpool, and about other aspects of their life. It revealed that the more densely packed the young were the faster their colony grew, provided conditions were favourable. In unfavourable circumstances close packing of young delayed growth. The rapid elongation of young tubes surrounding older tubes sometimes resulted in holes and fissures forming when the older worms failed to maintain all-round contact with surrounding young as the surface area of a colony increased. Such holes and fissures gave a false appearance of physical damage, and in colonies where they formed weakened the whole structure, becoming a factor in its ultimate destruction by wave action. Such fast-growing colonies have shorter lives than others growing more slowly and remaining more compact.

scholarly journals NOD mice, susceptible to pancreatic autoimmunity, demonstrate delayed growth of pancreatic cancer

Oncotarget ◽  
2017 ◽  
Vol 8 (46) ◽  
pp. 80167-80174
Author(s):  
James Dooley ◽  
Emanuela Pasciuto ◽  
Vasiliki Lagou ◽  
Yulia Lampi ◽  
Tom Dresselaers ◽  
...  
Keyword(s):  
Pancreatic Cancer ◽  
Nod Mice ◽  
Delayed Growth
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