Enhanced riverine carbon flux from carbonate catchment to the ocean: A comparative hydrogeochemical study on Ishigaki and Iriomote islands, southwestern Japan

2010 ◽  
Vol 115 (G2) ◽  
pp. n/a-n/a ◽  
Author(s):  
Hiroyuki Ushie ◽  
Hodaka Kawahata ◽  
Atsushi Suzuki ◽  
Shohei Murayama ◽  
Mayuri Inoue
Keyword(s):  
Carbon Flux ◽  
Carbonate Catchment ◽  
Riverine Carbon Flux

scholarly journals Riverine carbon flux from the Red River system (Viet Nam and China): a modelling approach

2017 ◽  
Vol 7 (1) ◽  
Author(s):  
Thi Phuong Quynh Le ◽  
Josette Garnier ◽  
Gilles Billen ◽  
Thi Mai Huong Nguyen ◽  
Emma Rochelle-Newall ◽  
...  
Keyword(s):  
Carbon Flux ◽  
River System ◽  
Red River ◽  
Viet Nam ◽  
Riverine Carbon Flux ◽  
Modelling Approach

An assessment of the riverine carbon flux of the Xijiang River during the past 50 years

2010 ◽  
Vol 226 (1-2) ◽  
pp. 38-43 ◽  
Author(s):  
H.G. Sun ◽  
J. Han ◽  
X.X. Lu ◽  
S.R. Zhang ◽  
D. Li
Keyword(s):  
Carbon Flux ◽  
The Past ◽  
Xijiang River ◽  
The Xijiang River ◽  
Riverine Carbon Flux

scholarly journals Kog1/Raptor mediates metabolic rewiring during nutrient limitation by controlling SNF1/AMPK activity

2021 ◽  
Vol 7 (16) ◽  
pp. eabe5544
Author(s):  
Zeenat Rashida ◽  
Rajalakshmi Srinivasan ◽  
Meghana Cyanam ◽  
Sunil Laxman
Keyword(s):  
Amino Acid ◽  
Nutrient Limitation ◽  
Carbon Flux ◽  
Metabolic Flux ◽  
Carbon Allocation ◽  
Carbon Utilization ◽  
Acid Biosynthesis ◽  
Control Growth ◽  
Ampk Activity ◽  
Delayed Growth

In changing environments, cells modulate resource budgeting through distinct metabolic routes to control growth. Accordingly, the TORC1 and SNF1/AMPK pathways operate contrastingly in nutrient replete or limited environments to maintain homeostasis. The functions of TORC1 under glucose and amino acid limitation are relatively unknown. We identified a modified form of the yeast TORC1 component Kog1/Raptor, which exhibits delayed growth exclusively during glucose and amino acid limitations. Using this, we found a necessary function for Kog1 in these conditions where TORC1 kinase activity is undetectable. Metabolic flux and transcriptome analysis revealed that Kog1 controls SNF1-dependent carbon flux apportioning between glutamate/amino acid biosynthesis and gluconeogenesis. Kog1 regulates SNF1/AMPK activity and outputs and mediates a rapamycin-independent activation of the SNF1 targets Mig1 and Cat8. This enables effective glucose derepression, gluconeogenesis activation, and carbon allocation through different pathways. Therefore, Kog1 centrally regulates metabolic homeostasis and carbon utilization during nutrient limitation by managing SNF1 activity.

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