Simultaneous absorption of dilute nitric oxide and sulfur dioxide into aqueous slurries of magnesium hydroxide with added iron(II)-EDTA chelate

1982 ◽  
Vol 21 (4) ◽  
pp. 771-774 ◽  
Author(s):  
Eizo Sada ◽  
Hidehiro Kumazawa ◽  
Yasushi Sawada ◽  
Takashi Kondo
Keyword(s):  
Nitric Oxide ◽  
Sulfur Dioxide ◽  
Magnesium Hydroxide ◽  
Simultaneous Absorption

Kinetics for the simultaneous absorption of nitric oxide and sulfur dioxide with the hexamminecobalt solution

2008 ◽  
Vol 62 (1) ◽  
pp. 183-191 ◽  
Author(s):  
Yan-Peng Mao ◽  
Wei Bi ◽  
Xiang-Li Long ◽  
Wen-De Xiao ◽  
Wei Li ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Sulfur Dioxide ◽  
Simultaneous Absorption

Sulfur dioxide induces vascular relaxation through PI3K/Akt/eNOS and NO/cGMP signaling pathways in rats

2020 ◽  
Vol 39 (8) ◽  
pp. 1108-1117
Author(s):  
Q Zhang ◽  
W Lyu ◽  
M Yu ◽  
Y Niu
Keyword(s):  
Nitric Oxide ◽  
Sulfur Dioxide ◽  
Signaling Pathways ◽  
Underlying Mechanism ◽  
Cyclic Guanosine Monophosphate ◽  
Rat Aorta ◽  
Guanosine Monophosphate ◽  
Signal Pathways ◽  
Atmospheric Pollutant ◽  
Protein Levels

Sulfur dioxide (SO2) is a common exogenous atmospheric pollutant. Studies have shown that SO2 can cause vasodilation as a gas signaling molecule, but the specific signaling pathways are not well understood. This study aimed to explore the underlying mechanism behind the effects of SO2 on vasodilation of isolated rat aorta. The results showed that when the dose of SO2 was 30 μM, the vasodilation of endothelium-intact rings was partially suppressed by LY294002 and NG-nitro-l-arginine methyl ester, and the protein levels of phosphoinositide 3-kinase (PI3K), p-Akt, and p-endothelial nitric oxide synthase ( p-eNOS) were significantly increased. When the dose of SO2 was 300 μM or 1500 μM, the vasodilation of endothelium-denuded rings did not change after application of the inhibitor, but the protein levels of PI3K, p-Akt, and p-eNOS were significantly decreased, and the activity of NOS and the level of nitric oxide (NO) and cyclic guanosine monophosphate (cGMP) were significantly increased. We speculate that the mechanism of SO2-induced vasodilatation likely involved the endothelial PI3K/Akt/eNOS and NO/cGMP signal pathways. In addition, at the concentration of 1500 μM, SO2 markedly increased the level of caspase-3 and caspase-9. The results suggest that high concentrations of SO2 may cause damage to blood vessels. This study will help to further inform the etiologies of SO2-related cardiovascular disease.

Adsorption equilibrium for sulfur dioxide, nitric oxide, carbon dioxide, nitrogen on 13X and 5A zeolites

2012 ◽  
Vol 188 ◽  
pp. 77-85 ◽  
Author(s):  
Hua Deng ◽  
Honghong Yi ◽  
Xiaolong Tang ◽  
Qiongfen Yu ◽  
Ping Ning ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Carbon Dioxide ◽  
Sulfur Dioxide ◽  
Adsorption Equilibrium

Modulatory Effects Of Sulfur Dioxide On The Production Of Nitric Oxide In Murine Airway Inflammation

2011 ◽  
Author(s):  
Anita L. Reno ◽  
Lance M. Hallberg ◽  
Jesse L. Parks, IV ◽  
Ed G. Brooks ◽  
William J. Calhoun ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Sulfur Dioxide ◽  
Airway Inflammation
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